Defining structural heart disease in the adult patient: current scope, inherent challenges and future directions
Abstract
Structural heart disease refers to non-coronary cardiovascular disease processes and related interventions. The last 10 years has seen tremendous advances in both the understanding of these processes and the therapeutic modalities aimed to treat them. Technology continues to evolve and clinical trials are ongoing to help delineate the appropriate role these therapies will play. Inherent challenges exist in training physicians in structural heart disease. Ultimately, credentialing societies and training programmes will emerge to help assure expertise within the cardiovascular community. This review focuses on the scope of structural heart disease, summarizes some of the current and future technologies available, highlights inherent challenges pertaining to structural heart disease and outlines future directions the field may take.
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Structural heart diseases are a spectrum of prevalent cardiac disorders with a long presymptomatic course and substantially elevated risk of heart failure and premature death.
“Structural heart diseases (SHDs) represent a spectrum of prevalent cardiac disorders with a long presymptomatic course and with substantially elevated risk of heart failure (HF) and premature death.1”
From Ensemble Deep Learning Algorithm for Structural Heart Disease Screening Using Electrocardiographic Images (2025) · cites this paper as [1]
Cites
Appendage obliteration to reduce stroke in cardiac surgical patients with atrial fibrillation
Blackshear, J. L., & Odell, J. A. (1996) · The Annals of Thoracic Surgery · doi:10.1016/0003-4975(95)00887-x · Cited by 1,686 · cited 1× · see citing works
“The left atrial appendage (LAA) is the site of thrombus formation and the source of embolism in more than 90% of cases based on autopsy studies and echocardiography.(21) Occlusion of the LAA may prevent thromboembolism by removing the site of thrombus formation.”
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Dhingra, L. S., Aminorroaya, A., Sangha, V., Pedroso, A. F., Shankar, S. V., Coppi, A., Foppa, M., Brant, L. C. C., Barreto, S. M., Ribeiro, A. L. P., et al. (2025) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2025.01.030 · Cited by 53 · see citing works
- Authors
- Steinberg, D. H., Staubach, S., Franke, J., & Sievert, H.
- Affiliations
- Medical University of South Carolina (US)
- Cardiovascular Center Frankfurt (DE)
- Corresponding author
- Sievert, H.
- Venue
- European Heart Journal Supplements
- Publisher
- Oxford University Press (OUP)
- Published
- 2010-09-01
- Volume
- 12
- Issue
- Suppl E
- Pages
- E2-E9
- ISSN
- 1520-765X (ISSN-L)
- OpenAlex
- W2048023421
- Cited by
- 51
- References
- 1
- Citations by year
- 2026: 4 · 2025: 7 · 2024: 6 · 2023: 7 · 2022: 1 · 2021: 4 · 2020: 1 · 2019: 3 · 2018: 1 · 2017: 2 · 2016: 2 · 2015: 3 · 2014: 6 · 2012: 2
- FWCI
- 0.66
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- European Heart Journal Supplements (publishedVersion)
- Topics
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Cardiac Valve Diseases and Treatments
- Health Sciences › Medicine › Epidemiology › Congenital Heart Disease Studies
- Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Cardiac Arrhythmias and Treatments
- Keywords
- structural heart disease, adult congenital heart disease, transcatheter interventions, physician credentialing, cardiovascular technology, clinical trials
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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