The DOI matches its Crossref record; 50 of the 50 references it cites were found in a registry or an index.
Role of neurotrophins in mouse neuronal development
Abstract
During vertebrate development, naturally occurring neuronal cell death is regulated by target‐derived peptide factors, called neurotrophins. A recent series of papers describe the phenotypes of germline‐targeted mutant mice deficient in neurotrophins and their receptors. Histological analysis of these mice for the first time has provided knowledge about the specific neuron populations that are dependent on neurotrophin action for development. Mice deficient for nerve growth factor (NGF) and its high‐affinity receptor, encoded by the trk A proto‐oncogene, suffer from complete loss of sympathetic neurons and sensory neurons responsive to temperature and pain. Mice deficient for brain‐derived neurotrophic factor (BDNF) and its receptor, encoded by the trk B gene, display loss of sensory neurons responsive to tactile stimuli. In addition, trk B mutant mice experience loss of motor neurons indicating a possible specific function of the second TrkB ligand, neurotrophin‐4 (NT‐4), in motor neuron development. Mice deficient for neurotrophin‐3 (NT‐3) and its receptor, encoded by the trkC gene, show abnormal movements caused by the loss of sensory proprioceptive neurons.— Klein, R. Role of neurotrophins in mouse neuronal development. FASEB J. 8: 738‐744; 1994.
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An NGF-TrkA-Mediated Retrograde Signal to Transcription Factor CREB in Sympathetic Neurons
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Metadata
- Authors
- Klein, R.
- Venue
- The FASEB Journal
- Publisher
- Wiley
- Published
- 1994-07
- Volume
- 8
- Issue
- 10
- Pages
- 738-744
- ISSN
- 0892-6638 (ISSN-L)
- 1530-6860
- OpenAlex
- W1832821166
- Cited by
- 259
- References
- 50
- FWCI
- 7.98
- Type
- journal article
- Language
- en
- Open access
- closed
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