Signaling to NF-κB

Hayden, Matthew S., Ghosh, Sankar·2004·Genes & Development

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Items 201–367 of 367

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  26. IKKβ Is Essential for Protecting T Cells from TNFα-Induced Apoptosis
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  35. RelA/p65 is a molecular target for the immunosuppressive action of protein kinase A.
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  37. Targeted disruption of Traf5 gene causes defects in CD40- and CD27-mediated lymphocyte activation
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  61. Cytoplasmic Sequestration of Rel Proteins by IκBα Requires CRM1-Dependent Nuclear Export
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  62. The α and β Subunits of IκB Kinase (IKK) Mediate TRAF2-Dependent IKK Recruitment to Tumor Necrosis Factor (TNF) Receptor 1 in Response to TNF
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  64. Direct Phosphorylation of NF-κB1 p105 by the IκB Kinase Complex on Serine 927 Is Essential for Signal-induced p105 Proteolysis*
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  65. Akt-Dependent Phosphorylation Specifically Regulates Cot Induction of NF-κB-Dependent Transcription
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  73. βTrCP-Mediated Proteolysis of NF-κB1 p105 Requires Phosphorylation of p105 Serines 927 and 932
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  91. The IκB Kinase (IKK) Complex Is Tripartite and Contains IKKγ but Not IKAP as a Regular Component
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  92. p50–NF-κB Complexes Partially Compensate for the Absence of RelB: Severely Increased Pathology in p50−/−relB−/−Double-knockout Mice
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  94. The generation of nfkb2 p52: mechanism and efficiency
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    Amir Orian, Alan L. Schwartz, Alain Israël, Simon T. Whiteside, Chaim Kahana, Aaron J. Ciechanover · 1999 · Molecular and Cellular Biology
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  97. Human T-cell Lymphotropic Virus Type 1 Tax Induction of Biologically Active NF-κB Requires IκB Kinase-1-mediated Phosphorylation of RelA/p65
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