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Cited by 738

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Role of Reactive Oxygen Intermediates and Cognate Redox Signaling in Disease Resistance

Grant, John J., Loake, Gary J.·2000·Plant Physiology

Abstract

Plants have evolved a plethora of sophisticated defense mechanisms to resist their potential colonization by microbial pathogens and parasites. Preformed physical and chemical barriers constitute the first line of defense. Superimposed upon these is a battery of inducible responses, their engagement being dependent upon successful recognition of the invading pathogen (Hammond-Kosack and Jones, 1996). This exquisitely specific recognition event is thought to be mediated via the direct or indirect interaction between the product of a microbial avirulence (avr) gene and the corresponding plant disease resistance (R) gene product. This often results in the programmed execution of challenged host cells, producing a visible area of cell death, the hypersensitive response (HR), surrounding the site of attempted pathogen ingress (Dangl et al., 1996). Following HR formation is the establishment of immunity in systemic tissues to secondary infections termed systemic acquired resistance (SAR), which conveys protection against a broad spectrum of microbial pathogens.

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Metadata

Authors
Grant, J. J., & Loake, G. J.
Venue
Plant Physiology
Published
2000-09-01
Volume
124
Issue
1
Pages
21-30
ISSN
  • 0032-0889 (ISSN-L)
  • 1532-2548
OpenAlex
W2095305581
Cited by
738
References
2
FWCI
52.85
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Role of Reactive Oxygen Intermediates and Cognate Redox Signaling in Disease Resistance. https://app.cochat.ai/papers/10.1104/pp.124.1.21
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Grant, J. J., & Loake, G. J. (2000). Role of Reactive Oxygen Intermediates and Cognate Redox Signaling in Disease Resistance. Plant Physiology. https://doi.org/10.1104/pp.124.1.21

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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