The DOI matches its Crossref record; 130 of the 130 references it cites were found in a registry or an index.
The development of psychopathy
Abstract
The current review focuses on the construct of psychopathy, conceptualized as a clinical entity that is fundamentally distinct from a heterogeneous collection of syndromes encompassed by the term ‘conduct disorder’. We will provide an account of the development of psychopathy at multiple levels: ultimate causal (the genetic or social primary cause), molecular, neural, cognitive and behavioral. The following main claims will be made: (1) that there is a stronger genetic as opposed to social ultimate cause to this disorder. The types of social causes proposed (e.g., childhood sexual/physical abuse) should elevate emotional responsiveness, not lead to the specific form of reduced responsiveness seen in psychopathy; (2) The genetic influence leads to the emotional dysfunction that is the core of psychopathy; (3) The genetic influence at the molecular level remains unknown. However, it appears to impact the functional integrity of the amygdala and orbital/ventrolateral frontal cortex (and possibly additional systems); (4) Disruption within these two neural systems leads to impairment in the ability to form stimulus–reinforcement associations and to alter stimulus–response associations as a function of contingency change. These impairments disrupt the impact of standard socialization techniques and increase the risk for frustration‐induced reactive aggression respectively.
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Cites
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Cited by
Frick, P. J., & White, S. F. (2008) · Journal of Child Psychology and Psychiatry · doi:10.1111/j.1469-7610.2007.01862.x · Cited by 1,290
Marsh, A. A., Finger, E. C., Mitchell, D. G. V., Reid, M. E., Sims, C., Kosson, D. S., Towbin, K. E., Leibenluft, E., Pine, D. S., & Blair, R. J. R. (2008) · American Journal of Psychiatry · doi:10.1176/appi.ajp.2007.07071145 · Cited by 852
Self-citationAmygdala Hypoactivity to Fearful Faces in Boys With Conduct Problems and Callous-Unemotional Traits
Jones, A. P., Laurens, K. R., Herba, C. M., Barker, G. J., & Viding, E. (2009) · American Journal of Psychiatry · doi:10.1176/appi.ajp.2008.07071050 · Cited by 564
Rubia, K. (2011) · Biological Psychiatry · doi:10.1016/j.biopsych.2010.09.023 · Cited by 440
Shamay-Tsoory, S. G., Harari, H., Aharon-Peretz, J., & Levkovitz, Y. (2010) · Cortex · doi:10.1016/j.cortex.2009.04.008 · Cited by 432
Metadata
- Authors
- Blair, R. J. R., Peschardt, K. S., Budhani, S., Mitchell, D. G. V., & Pine, D. S.
- Venue
- Journal of Child Psychology and Psychiatry
- Publisher
- Wiley
- Published
- 2006-02-22
- Volume
- 47
- Issue
- 3-4
- Pages
- 262-276
- ISSN
- 0021-9630 (ISSN-L)
- 1469-7610
- OpenAlex
- W2139361844
- Cited by
- 486
- References
- 130
- FWCI
- 32.20
- Type
- journal article
- Language
- en
- Open access
- closed
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- Blair, R. J. R., Peschardt, K. S., Budhani, S., Mitchell, D. G. V., & Pine, D. S. (2006). The development of psychopathy. Journal of Child Psychology and Psychiatry. https://doi.org/10.1111/j.1469-7610.2006.01596.x
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
