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Cited by 1,484

The DOI matches its Crossref record; 4 of the 4 references it cites were found in a registry or an index.

Muscle fatigue: what, why and how it influences muscle function

Open access
Enoka, Roger M., Duchateau, Jacques·2008·The Journal of Physiology

Abstract

Much is known about the physiological impairments that can cause muscle fatigue. It is known that fatigue can be caused by many different mechanisms, ranging from the accumulation of metabolites within muscle fibres to the generation of an inadequate motor command in the motor cortex, and that there is no global mechanism responsible for muscle fatigue. Rather, the mechanisms that cause fatigue are specific to the task being performed. The development of muscle fatigue is typically quantified as a decline in the maximal force or power capacity of muscle, which means that submaximal contractions can be sustained after the onset of muscle fatigue. There is even evidence that the duration of some sustained tasks is not limited by fatigue of the principal muscles. Here we review experimental approaches that focus on identifying the mechanisms that limit task failure rather than those that cause muscle fatigue. Selected comparisons of tasks, groups of individuals and interventions with the task‐failure approach can provide insight into the rate‐limiting adjustments that constrain muscle function during fatiguing contractions.

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Crossref · 10.1113/jphysiol.2007.139477

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Metadata

Authors
Enoka, R. M., & Duchateau, J.
Venue
The Journal of Physiology
Publisher
Wiley
Published
2008-01
Volume
586
Issue
1
Pages
11-23
ISSN
  • 0022-3751 (ISSN-L)
  • 1469-7793
OpenAlex
W2107387209
Cited by
1,484
References
4
FWCI
20.80
Type
journal article
Language
en
Open access
green

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.