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Cited by 750

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis

Lee, David M., Friend, Daniel S., Gurish, Michael F., Benoist, Christophe, Mathis, Diane, Brenner, Michael B.·2002·Science

Abstract

Previous studies have revealed that autoantibodies, complement components, and Fc receptors each participate in the pathogenesis of erosive arthritis in K/BxN mice. However, it is not known which cellular populations are responsive to these inflammatory signals. We find that two strains of mice deficient in mast cells, W/W v and Sl/Sl d , were resistant to development of joint inflammation and that susceptibility was restored in the W/W v strain by mast cell engraftment. Thus, mast cells may function as a cellular link between autoantibodies, soluble mediators, and other effector populations in inflammatory arthritis.

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Registry

Crossref · 10.1126/science.1073176

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Metadata

Authors
Lee, D. M., Friend, D. S., Gurish, M. F., Benoist, C., Mathis, D., & Brenner, M. B.
Venue
Science
Published
2002-09-06
Volume
297
Issue
5587
Pages
1689-1692
ISSN
  • 0036-8075 (ISSN-L)
  • 1095-9203
OpenAlex
W1982709334
Cited by
750
References
2
FWCI
17.53
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis. https://app.cochat.ai/papers/10.1126/science.1073176
The paper
Lee, D. M., Friend, D. S., Gurish, M. F., Benoist, C., Mathis, D., & Brenner, M. B. (2002). Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis. Science. https://doi.org/10.1126/science.1073176

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.