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Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis
Abstract
Previous studies have revealed that autoantibodies, complement components, and Fc receptors each participate in the pathogenesis of erosive arthritis in K/BxN mice. However, it is not known which cellular populations are responsive to these inflammatory signals. We find that two strains of mice deficient in mast cells, W/W v and Sl/Sl d , were resistant to development of joint inflammation and that susceptibility was restored in the W/W v strain by mast cell engraftment. Thus, mast cells may function as a cellular link between autoantibodies, soluble mediators, and other effector populations in inflammatory arthritis.
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Metadata
- Authors
- Lee, D. M., Friend, D. S., Gurish, M. F., Benoist, C., Mathis, D., & Brenner, M. B.
- Venue
- Science
- Published
- 2002-09-06
- Volume
- 297
- Issue
- 5587
- Pages
- 1689-1692
- ISSN
- 0036-8075 (ISSN-L)
- 1095-9203
- OpenAlex
- W1982709334
- Cited by
- 750
- References
- 2
- FWCI
- 17.53
- Type
- journal article
- Language
- en
- Open access
- closed
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