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Hypermutation of HIV-1 DNA in the Absence of the Vif Protein
Abstract
The Vif (viral infectivity factor) protein of human immunodeficiency virus–1 (HIV-1) is essential for the production of infectious virus by T lymphocytes and macrophages, the natural targets for HIV-1 infection. Vif is dispensable for HIV infection in certain transformed cell lines (“permissive
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Cited by
Mehle, A., Strack, B., Ancuta, P., Zhang, C., McPike, M., & Gabuzda, D. (2004) · Journal of Biological Chemistry · doi:10.1074/jbc.m313093200 · Cited by 474
Hiener, B., Horsburgh, B. A., Eden, J.-S., Barton, K., Schlub, T. E., Lee, E., von Stockenstrom, S., Odevall, L., Milush, J. M., Liegler, T., et al. (2017) · Cell Reports · doi:10.1016/j.celrep.2017.09.081 · Cited by 370
Keckesova, Z., Ylinen, L. M. J., & Towers, G. J. (2004) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.0402474101 · Cited by 344
Reconstitution of an Infectious Human Endogenous Retrovirus
Lee, Y. N., & Bieniasz, P. D. (2007) · PLoS Pathogens · doi:10.1371/journal.ppat.0030010 · Cited by 311
Complementary function of the two catalytic domains of APOBEC3G
Navarro, F., Bollman, B., Chen, H., König, R., Yu, Q., Chiles, K., & Landau, N. R. (2005) · Virology · doi:10.1016/j.virol.2005.01.011 · Cited by 305
Metadata
- Authors
- Lecossier, D., Bouchonnet, F., Clavel, F., & Hance, A. J.
- Venue
- Science
- Published
- 2003-05-16
- Volume
- 300
- Issue
- 5622
- Pages
- 1112-1112
- ISSN
- 0036-8075 (ISSN-L)
- 1095-9203
- OpenAlex
- W2012564250
- Cited by
- 651
- References
- 0
- FWCI
- 20.36
- Type
- journal article
- Language
- en
- Open access
- closed
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- Lecossier, D., Bouchonnet, F., Clavel, F., & Hance, A. J. (2003). Hypermutation of HIV-1 DNA in the Absence of the Vif Protein. Science. https://doi.org/10.1126/science.1083338
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.