Production of the Alzheimer Amyloid β Protein by Normal Proteolytic Processing

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8. The Class A Scavenger Receptor Binds to Proteoglycans and Mediates Adhesion of Macrophages to the Extracellular Matrix
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9. A Comparative Study of β-Amyloid Peptides Aβ1-42 and Aβ25-35 Toxicity in Organotypic Hippocampal Slice Cultures
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10. Styrylbenzoxazole Derivatives forIn VivoImaging of Amyloid Plaques in the Brain
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11. Glutamine synthetase, hemoglobin α-chain, and macrophage migration inhibitory factor binding to amyloid β-protein: their identification in rat brain by a novel affinity chromatography and in Alzheimer’s disease brain by immunoprecipitation
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13. Degradation of Amyloid β-Protein by a Serine Protease-α2-Macroglobulin Complex
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14. Role of neurotransmission in the regulation of amyloid β-protein precursor processing
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15. Amyloid β-Protein (Aβ) 1–40 But Not Aβ1–42 Contributes to the Experimental Formation of Alzheimer Disease Amyloid Fibrils in Rat Brain
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18. Presenilin 1 Regulates Pharmacologically Distinct γ-Secretase Activities
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19. The Solved and Unsolved Mysteries of the Genetics of Early-Onset Alzheimer 's Disease
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26. Effect of endothelial cell polarity on β-amyloid-induced migration of monocytes across normal and AD endothelium
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31. AßT Amyloidogenesis: Unique, or Variation on a Systemic Theme
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33. The release of Alzheimer's disease beta amyloid peptide is reduced by phorbol treatment.
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34. The length of Amyloid-β in Hereditary Cerebral Hemorrhage with Amyloidosis, Dutch Type
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38. Autoantibodies to Redox-modified Oligomeric Aβ Are Attenuated in the Plasma of Alzheimer's Disease Patients
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39. Brain Clearance of Alzheimer's Amyloid-β40 in the Squirrel Monkey: A SPECT Study in a Primate Model of Cerebral Amyloid Angiopathy
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41. Effect of energy shortage and oxidative stress on amyloid precursor protein metabolism in COS cells
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42. Low‐n oligomers as therapeutic targets of Alzheimer’s disease
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43. BACE1 inhibition more effectively suppresses initiation than progression of β-amyloid pathology
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44. Nerve Growth Factor Pathobiology During the Progression of Alzheimer’s Disease
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