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Cited by 260

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Role of Type I Myosins in Receptor-Mediated Endocytosis in Yeast

Geli, M. Isabel, Riezman, Howard·1996·Science

Abstract

Type I myosins are thought to drive actin-dependent membrane motility, but the direct demonstration in vivo of their involvement in specific cellular processes has been difficult. Deletion of the genes MYO3 and MYO5 , which encode the yeast type I myosins, almost abolished growth. A double-deleted mutant complemented with a MYO5 temperature-sensitive allele ( myo5-1 ) showed a strong defect in the internalization step of receptor-mediated endocytosis, whereas the secretory pathway remained apparently unaffected. Thus, myosin I activity is required for a budding event in endocytosis but not for several other aspects of membrane traffic.

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Crossref · 10.1126/science.272.5261.533

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Metadata

Authors
Geli, M. I., & Riezman, H.
Venue
Science
Published
1996-04-26
Volume
272
Issue
5261
Pages
533-535
ISSN
  • 0036-8075 (ISSN-L)
  • 1095-9203
OpenAlex
W2022975161
Cited by
260
References
2
FWCI
10.73
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Role of Type I Myosins in Receptor-Mediated Endocytosis in Yeast. https://app.cochat.ai/papers/10.1126/science.272.5261.533
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Geli, M. I., & Riezman, H. (1996). Role of Type I Myosins in Receptor-Mediated Endocytosis in Yeast. Science. https://doi.org/10.1126/science.272.5261.533

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.