The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
Small heterodimer partner SHP mediates liver X receptor (LXR)-dependent suppression of inflammatory signaling by promoting LXR SUMOylation specifically in astrocytes
Abstract
The transcriptional regulator SHP promotes SUMOylation of the LXR transcriptional factors specifically in astrocytes to limit inflammatory signaling.
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LXRs become SUMOylated in the presence of ligands, repressing inflammatory target genes.
“LXRs as the oxysterol-sensitive nuclear receptors become SUMOylated in the presence of endogenous and/or synthetic ligands, thereby repressing the transcription of inflammatory target genes in multiple cell types (33, 34, 35).”
From SUMOylation of the ubiquitin ligase IDOL decreases LDL receptor levels and is reversed by SENP1 (2021) · cites this paper as [35]
Citations
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Sorted by citations
SUMOylation of the ubiquitin ligase IDOL decreases LDL receptor levels and is reversed by SENP1
Wang, J.-Q., Lin, Z.-C., Li, L.-L., Zhang, S.-F., Li, W.-H., Liu, W., Song, B.-L., & Luo, J. (2021) · Journal of Biological Chemistry · doi:10.1074/jbc.ra120.015420 · Cited by 24
Metadata
- Authors
- Lee, J. H., Kim, H., Park, S. J., Woo, J. H., Joe, E.-H., & Jou, I.
- Affiliations
- Department of Pharmacology, Chronic Inflammatory Disease Research Center, Ajou University School of Medicine, Suwon 442-721, Korea.
- Venue
- Science Signaling
- Published
- 2016-08-02
- Volume
- 9
- Issue
- 439
- ISSN
- 1945-0877
- 1937-9145
- Cited by
- 24
- References
- 6
- Language
- en
- Funders
- National Research Foundation of Korea
- Ministry of Science, ICT and Future Planning (NRF-2012R1A5A2048183, NRF-2013R1A1A3008531)
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