The DOI matches its Crossref record; 451 of the 451 references it cites were found in a registry or an index.
Immunology of Mycobacterium tuberculosis Infections
Open accessAbstract
Tuberculosis (TB) is a serious global public health challenge that results in significant morbidity and mortality worldwide. TB is caused by infection with the bacilli Mycobacterium tuberculosis(M. tuberculosis), which has evolved a wide variety of strategies in order to thrive within its host. Understanding the complex interactions between M. tuberculosis and host immunity can inform the rational design of better TB vaccines and therapeutics. This chapter covers innate and adaptive immunity against M. tuberculosis infection, including insights on bacterial immune evasion and subversion garnered from animal models of infection and human studies. In addition, this chapter discusses the immunology of the TB granuloma, TB diagnostics, and TB comorbidities. Finally, this chapter provides a broad overview of the current TB vaccine pipeline.
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1 retracted
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RetractedTwo subsets of memory T lymphocytes with distinct homing potentials and effector functions
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Toll-Like Receptor Triggering of a Vitamin D-Mediated Human Antimicrobial Response
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Tuberculosis Associated with Infliximab, a Tumor Necrosis Factor α–Neutralizing Agent
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A Novel Transcription Factor, T-bet, Directs Th1 Lineage Commitment
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Antimicrobial Autophagy: A Conserved Innate Immune Response in Drosophila
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PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression
Day, C. L., Kaufmann, D. E., Kiepiela, P., Brown, J. A., Moodley, E. S., Reddy, S., Mackey, E. W., Miller, J. D., Leslie, A. J., DePierres, C., et al. (2006) · Nature · doi:10.1038/nature05115 · Cited by 2,680
An essential role for interferon gamma in resistance to Mycobacterium tuberculosis infection.
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Metadata
- Authors
- Sia, J. K., & Rengarajan, J.
- Venue
- Microbiology Spectrum
- Publisher
- American Society for Microbiology
- Published
- 2019-07-19
- Volume
- 7
- Issue
- 4
- ISSN
- 2165-0497 (ISSN-L)
- OpenAlex
- W2961618894
- Cited by
- 312
- References
- 451
- FWCI
- 18.19
- Type
- journal article
- Language
- en
- Open access
- green
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
