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Cited by 3,736

The DOI matches its Crossref record; 15 of the 15 references it cites were found in a registry or an index.

Inflammation in Atherosclerosis

Open access
Libby, Peter·2012·Arteriosclerosis, Thrombosis, and Vascular Biology

Abstract

Experimental work has elucidated molecular and cellular pathways of inflammation that promote atherosclerosis. Unraveling the roles of cytokines as inflammatory messengers provided a mechanism whereby risk factors for atherosclerosis can alter arterial biology, and produce a systemic milieu that favors atherothrombotic events. The discovery of the immune basis of allograft arteriosclerosis demonstrated that inflammation per se can drive arterial hyperplasia, even in the absence of traditional risk factors. Inflammation regulates aspects of plaque biology that trigger the thrombotic complications of atherosclerosis. Translation of these discoveries to humans has enabled both novel mechanistic insights and practical clinical advances.

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Crossref · 10.1161/atvbaha.108.179705

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Metadata

Authors
Libby, P.
Venue
Arteriosclerosis, Thrombosis, and Vascular Biology
Published
2012-09
Volume
32
Issue
9
Pages
2045-2051
ISSN
  • 1079-5642 (ISSN-L)
  • 1524-4636
OpenAlex
W2156593520
Cited by
3,736
References
15
FWCI
110.81
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Inflammation in Atherosclerosis. https://app.cochat.ai/papers/10.1161/atvbaha.108.179705
The paper
Libby, P. (2012). Inflammation in Atherosclerosis. Arteriosclerosis, Thrombosis, and Vascular Biology. https://doi.org/10.1161/atvbaha.108.179705

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.