The DOI matches its Crossref record; 85 of the 85 references it cites were found in a registry or an index.
Idiopathic pulmonary fibrosis: an epithelial/fibroblastic cross-talk disorder
Open accessAbstract
Idiopathic pulmonary fibrosis is a chronic and usually progressive lung disorder of unknown etiology. A growing body of evidence suggests that, in contrast to other interstitial lung diseases, IPF is a distinct entity in which inflammation is a secondary and non-relevant pathogenic partner. Evidence includes the presence of similar mild/moderate inflammation either in early or late disease, and the lack of response to potent anti-inflammatory therapy. Additionally, it is clear from experimental models and some human diseases that it is possible to have fibrosis without inflammation. An evolving hypothesis proposes that IPF may result from epithelial micro-injuries and abnormal wound healing.
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Cites
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“A variety of inhibitors of the IL-4/IL-13 pathway have been reported, such as SOCS-1 (also known as SSI-1) [21]; however, this inhibitor was suggested to prevent activation of STAT and, for example, act on Jak proteins [37].”
BackgroundInfluentialIdiopathic Pulmonary Fibrosis: Clinical Relevance of Pathologic Classification
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Cited by
The biology and function of fibroblasts in cancer
Kalluri, R. (2016) · Nature Reviews Cancer · doi:10.1038/nrc.2016.73 · Cited by 4,366
TGF-β1 induces human alveolar epithelial to mesenchymal cell transition (EMT)
Kasai, H., Allen, J. T., Mason, R. M., Kamimura, T., & Zhang, Z. (2005) · Respiratory Research · doi:10.1186/1465-9921-6-56 · Cited by 744
Inhibition of Wnt/β-catenin/CREB binding protein (CBP) signaling reverses pulmonary fibrosis
Henderson, W. R., Chi, E. Y., Ye, X., Nguyen, C., Tien, Y.-T., Zhou, B., Borok, Z., Knight, D. A., & Kahn, M. (2010) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.1001520107 · Cited by 462
Caveolin-1: a critical regulator of lung fibrosis in idiopathic pulmonary fibrosis
Wang, X. M., Zhang, Y., Kim, H. P., Zhou, Z., Feghali-Bostwick, C. A., Liu, F., Ifedigbo, E., Xu, X., Oury, T. D., Kaminski, N., et al. (2006) · The Journal of Experimental Medicine · doi:10.1084/jem.20061536 · Cited by 397
Severe lung fibrosis requires an invasive fibroblast phenotype regulated by hyaluronan and CD44
Li, Y., Jiang, D., Liang, J., Meltzer, E. B., Gray, A., Miura, R., Wogensen, L., Yamaguchi, Y., & Noble, P. W. (2011) · Journal of Experimental Medicine · doi:10.1084/jem.20102510 · Cited by 382
Metadata
- Authors
- Selman, M., & Pardo, A.
- Venue
- Respiratory Research
- Published
- 2001-10-11
- Volume
- 3
- Issue
- 1
- Pages
- 3-3
- ISSN
- 1465-9921 (ISSN-L)
- 1465-993X
- OpenAlex
- W1585502126
- Cited by
- 379
- References
- 85
- FWCI
- 7.72
- Type
- journal article
- Language
- en
- Open access
- gold
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
