The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.
Mobilization of cytoplasmic CE droplets by overexpression of human macrophage cholesteryl ester hydrolase
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
Much of the free cholesterol within the endocytic pathway resides in the ERC.
“Recent studies have determined that much of the free cholesterol within the endocytic pathway resides in the tubulo-vesicular endocytic recycling compartment [9,10]”
From EHD1 regulates cholesterol homeostasis and lipid droplet storage (2007) · cites this paper as [10]
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Brown, M. S., Ho, Y. K., & Goldstein, J. L. (1980) · Journal of Biological Chemistry · doi:10.1016/s0021-9258(19)70568-7 · Cited by 722
Flagged first, then by citations
Research works printed but not linked: 40
- Bastiaanse, 1997, The effect of membrane cholesterol content on ion transport processes in plasma membranes, Cardiovasc. Res., 33, 272, 10.1016/S0008-6363(96)00193-9 · DOI
- Rothblat, 2002, Cellular cholesterol flux studies: methodological considerations, Atherosclerosis., 163, 1, 10.1016/S0021-9150(01)00713-4 · DOI
- Morel, 1996, Comparison of the intracellular metabolism and trafficking of 25-hydroxycholesterol and cholesterol in macrophages, J. Lipid Res., 37, 2041, 10.1016/S0022-2275(20)37568-4 · DOI
- Ishii, 1992, Beta-VLDL-induced cholesterol ester deposition in macrophages may be regulated by neutral cholesterol esterase activity, Arterioscler. Thromb., 12, 1139, 10.1161/01.ATV.12.10.1139 · DOI
- Mathur, 1985, A defect in mobilization of cholesteryl esters in rabbit macrophages, Biochim. Biophys. Acta., 834, 48, 10.1016/0005-2760(85)90175-4 · DOI
- Yancey, 1994, Mechanism of the defect in cholesteryl ester clearance from macrophages of atherosclerosis-susceptible White Carneau pigeons, J. Lipid Res., 35, 2114, 10.1016/S0022-2275(20)39918-1 · DOI
- Goldberg, 1990, Stimulation of neutral cholesteryl ester hydrolase by cAMP in P388D1 macrophages, Biochim. Biophys. Acta., 1042, 132, 10.1016/0005-2760(90)90067-8 · DOI
- Small, 1989, Hormone sensitive lipase is responsible for the neutral cholesteryl ester hydrolase activity in macrophages, FEBS Lett., 247, 205, 10.1016/0014-5793(89)81335-3 · DOI
- Khoo, 1993, Expression of hormone-sensitive lipase mRNA in macrophages, J. Lipid Res., 34, 1969, 10.1016/S0022-2275(20)35114-2 · DOI
- Escary, 1998, Hormone-sensitive lipase overexpression increases cholesteryl ester hydrolysis in macrophage foam cells, Arterioscler. Thromb. Vasc. Biol., 18, 991, 10.1161/01.ATV.18.6.991 · DOI
- Osuga, 2000, Targeted disruption of hormone-sensitive lipase results in male sterility and adipocyte hypertrophy, but not in obesity, Proc. Natl. Acad. Sci. USA., 97, 787, 10.1073/pnas.97.2.787 · DOI
- Contreras, 2002, Hormone-sensitive lipase is not required for cholesteryl ester hydrolysis in macrophages, Biochem. Biophys. Res. Commun., 292, 900, 10.1006/bbrc.2002.6757 · DOI
- Ishii, 1995, Genetic differences of lipid metabolism in macrophages from C57BL/6J and C3H/HeN mice, Arterioscler. Thromb. Vasc. Biol., 15, 1189, 10.1161/01.ATV.15.8.1189 · DOI
- Graham, 1996, Impaired mobilization of cholesterol from stored cholesteryl esters in human (THP-1) macrophages, Atherosclerosis., 120, 135, 10.1016/0021-9150(95)05695-5 · DOI
- Hakamata, 1994, Species difference in cholesteryl ester cycle and HDL-induced cholesterol efflux from macrophage foam cells, Arterioscler. Thromb., 14, 1860, 10.1161/01.ATV.14.11.1860 · DOI
- Reue, 1997, Evidence for hormone-sensitive lipase mRNA in human monocyte/macrophages, Arterioscler. Thromb. Vasc. Biol., 17, 3428, 10.1161/01.ATV.17.12.3428 · DOI
- Johnson, 2000, Hormone sensitive lipase mRNA in both monocyte and macrophage forms of human THP-1 cell line, Comp. Biochem. Physiol., 126, 543, 10.1016/S0305-0491(00)00220-0 · DOI
- Contreras, 1994, Essential differences in cholesterol ester metabolism between human monocyte-derived and J774 macrophages: evidence against the presence of hormone sensitive lipase in human macrophage, Arterioscler. Thromb., 14, 443, 10.1161/01.ATV.14.3.443 · DOI
- Li, 1997, Modified low-density lipoprotein enhances the secretion of bile salt-stimulated cholesterol esterase by human monocyte-macrophages. Species-specific difference in macrophage cholesteryl ester hydrolase, J. Biol. Chem., 272, 28666, 10.1074/jbc.272.45.28666 · DOI
- Ghosh, 2000, Cholesteryl ester hydrolase in human monocyte/macrophage: cloning, sequencing and expression of full-length cDNA, Physiol. Genomics., 2, 1, 10.1152/physiolgenomics.2000.2.1.1 · DOI
- Lee, 2000, Differential expression of ACAT1 and ACAT2 among cells within liver, intestine, kidney, and adrenal of nonhuman primates, J. Lipid Res., 41, 1991, 10.1016/S0022-2275(20)32360-9 · DOI
- Reid, 2001, Hydrogen peroxide induced stress in human keratinocytes and its effect on biothionol toxicity, Toxicol. In Vitro., 15, 441, 10.1016/S0887-2333(01)00049-2 · DOI
- Ishikawa, 1974, Quantitative analysis of cholesterol in 5 to 20 microliter of plasma, J. Lipid Res., 15, 286, 10.1016/S0022-2275(20)36809-7 · DOI
- Klansek, 1995, Cholesterol quantitation by GLC: artifactual formation of short-chain steryl esters, J. Lipid Res., 36, 2261, 10.1016/S0022-2275(20)39210-5 · DOI
- Mahlberg, 1990, Potential problems in the use of commercial preparations of radiolabeled cholesterol, Atherosclerosis., 84, 95, 10.1016/0021-9150(90)90078-W · DOI
- Christian, 1999, Comparison of the capacity of beta-cyclodextrin derivatives and cyclophanes to shuttle cholesterol between cells and serum lipoproteins, J. Lipid Res., 40, 1475, 10.1016/S0022-2275(20)33390-3 · DOI
- Haynes, 2000, Efflux of cholesterol from different cellular pools, Biochemistry., 39, 4508, 10.1021/bi992125q · DOI
- Yamauchi, 2002, Differential regulation of apolipoprotein A-I/ATP binding cassette transporter A1-mediated cholesterol and phospholipid release, Biochim. Biophys. Acta., 1585, 1, 10.1016/S1388-1981(02)00304-9 · DOI
- Sliskovic, 1991, Therapeutic potential of ACAT inhibitors as lipid lowering and anti-atherosclerotic agents, Trends Pharmacol. Sci., 12, 194, 10.1016/0165-6147(91)90546-5 · DOI
- Matsuda, 1994, ACAT inhibitors as anti-atherosclerotic agents, Med. Res. Rev., 14, 271, 10.1002/med.2610140302 · DOI
- Matsuo, 1995, Effect of FR145237, a novel ACAT inhibitor on atherogenesis in cholesterol fed and WHHl rabbits. Evidence for a direct effect on the arterial wall, Biochim. Biophys. Acta., 1259, 254, 10.1016/0005-2760(95)00178-6 · DOI
- Nicolosi, 1998, The ACAT inhibitor, CI 1011 is effective in the prevention and regression of aortic fatty streak in hamsters, Atherosclerosis., 137, 77, 10.1016/S0021-9150(97)00279-7 · DOI
- Perrey, 2001, Preferential pharmacological inhibition of macrophage ACAT increases plaque formation in mouse and rabbit models of atherogenesis, Atherosclerosis., 155, 359, 10.1016/S0021-9150(00)00599-2 · DOI
- Accad, 2000, Massive xanthomatosis and altered composition of atherosclerotic lesions in hyperlipidemic mice lacking acyl CoA:cholesterol acyltransferase-1, J. Clin. Invest., 105, 711, 10.1172/JCI9021 · DOI
- Yagu, 2000, Absence of ACAT-1 attenuates atherosclerosis but causes dry eye and cutaneous xanthomatosis in mice with congenital hyperlipidemia, J. Biol. Chem., 275, 21324, 10.1074/jbc.M002541200 · DOI
- Fazio, 2001, Increased atherosclerosis in LDL receptor-null mice lacking ACAT1 in macrophages, J. Clin. Invest., 107, 163, 10.1172/JCI10310 · DOI
- Kellner-Weibel, 1999, Cytotoxic cholesterol is generated by the hydrolysis of cytoplasmic cholesteryl ester and transported to the plasma membrane, Atherosclerosis., 146, 309, 10.1016/S0021-9150(99)00155-0 · DOI
- Heinonen, 2002, Inhibition of acyl coenzyme A-cholesterol acyltransferase: a possible treatment of atherosclerosis?, Curr. Atheroscler. Rep., 4, 65, 10.1007/s11883-002-0064-9 · DOI
- Escary, 1999, Paradoxical effect on atherosclerosis of hormone-sensitive lipase overexpression in macrophages, J. Lipid Res., 40, 397, 10.1016/S0022-2275(20)32443-3 · DOI
- Ghosh, 2001, Cloning of the human cholesteryl ester hydrolase promoter: identification of functional peroxisomal proliferator-activated receptor responsive elements, Biochem. Biophys. Res. Commun., 284, 1065, 10.1006/bbrc.2001.5078 · DOI
Cited by
Sorted by citations
Intracellular cholesterol transport
Soccio, R. E., & Breslow, J. L. (2004) · Arteriosclerosis, Thrombosis, and Vascular Biology · doi:10.1161/01.atv.0000131264.66417.d5 · Cited by 297
EHD1 regulates cholesterol homeostasis and lipid droplet storage
Naslavsky, N., Rahajeng, J., Rapaport, D., Horowitz, M., & Caplan, S. (2007) · Biochemical and Biophysical Research Communications · doi:10.1016/j.bbrc.2007.04.022 · Cited by 53
Metadata
- Authors
- Ghosh, S., St. Clair, R. W., & Rudel, L. L.
- Venue
- Journal of Lipid Research
- Publisher
- Elsevier BV
- Published
- 2003-10
- Volume
- 44
- Issue
- 10
- Pages
- 1833-1840
- ISSN
- 0022-2275
- Cited by
- 55
- References
- 1
- Language
- en
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review