Cited by 55

The DOI matches its Crossref record; 1 of the 1 references it cites were found in a registry or an index.

Mobilization of cytoplasmic CE droplets by overexpression of human macrophage cholesteryl ester hydrolase

Ghosh, Shobha, St. Clair, Richard W., Rudel, Lawrence L.·2003·Journal of Lipid Research

Overview

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Integrity signals

Registry

Crossref · 10.1194/jlr.m300162-jlr200

Cites

1 of 1 references verified0 citation errors · 0 not checkable

Statistics

Verification

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

  • Much of the free cholesterol within the endocytic pathway resides in the ERC.

    “Recent studies have determined that much of the free cholesterol within the endocytic pathway resides in the tubulo-vesicular endocytic recycling compartment [9,10]”

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Citations

Cites

Flagged first, then by citations

Research works printed but not linked: 40

  1. Bastiaanse, 1997, The effect of membrane cholesterol content on ion transport processes in plasma membranes, Cardiovasc. Res., 33, 272, 10.1016/S0008-6363(96)00193-9 · DOI
  2. Rothblat, 2002, Cellular cholesterol flux studies: methodological considerations, Atherosclerosis., 163, 1, 10.1016/S0021-9150(01)00713-4 · DOI
  3. Morel, 1996, Comparison of the intracellular metabolism and trafficking of 25-hydroxycholesterol and cholesterol in macrophages, J. Lipid Res., 37, 2041, 10.1016/S0022-2275(20)37568-4 · DOI
  4. Ishii, 1992, Beta-VLDL-induced cholesterol ester deposition in macrophages may be regulated by neutral cholesterol esterase activity, Arterioscler. Thromb., 12, 1139, 10.1161/01.ATV.12.10.1139 · DOI
  5. Mathur, 1985, A defect in mobilization of cholesteryl esters in rabbit macrophages, Biochim. Biophys. Acta., 834, 48, 10.1016/0005-2760(85)90175-4 · DOI
  6. Yancey, 1994, Mechanism of the defect in cholesteryl ester clearance from macrophages of atherosclerosis-susceptible White Carneau pigeons, J. Lipid Res., 35, 2114, 10.1016/S0022-2275(20)39918-1 · DOI
  7. Goldberg, 1990, Stimulation of neutral cholesteryl ester hydrolase by cAMP in P388D1 macrophages, Biochim. Biophys. Acta., 1042, 132, 10.1016/0005-2760(90)90067-8 · DOI
  8. Small, 1989, Hormone sensitive lipase is responsible for the neutral cholesteryl ester hydrolase activity in macrophages, FEBS Lett., 247, 205, 10.1016/0014-5793(89)81335-3 · DOI
  9. Khoo, 1993, Expression of hormone-sensitive lipase mRNA in macrophages, J. Lipid Res., 34, 1969, 10.1016/S0022-2275(20)35114-2 · DOI
  10. Escary, 1998, Hormone-sensitive lipase overexpression increases cholesteryl ester hydrolysis in macrophage foam cells, Arterioscler. Thromb. Vasc. Biol., 18, 991, 10.1161/01.ATV.18.6.991 · DOI
  11. Osuga, 2000, Targeted disruption of hormone-sensitive lipase results in male sterility and adipocyte hypertrophy, but not in obesity, Proc. Natl. Acad. Sci. USA., 97, 787, 10.1073/pnas.97.2.787 · DOI
  12. Contreras, 2002, Hormone-sensitive lipase is not required for cholesteryl ester hydrolysis in macrophages, Biochem. Biophys. Res. Commun., 292, 900, 10.1006/bbrc.2002.6757 · DOI
  13. Ishii, 1995, Genetic differences of lipid metabolism in macrophages from C57BL/6J and C3H/HeN mice, Arterioscler. Thromb. Vasc. Biol., 15, 1189, 10.1161/01.ATV.15.8.1189 · DOI
  14. Graham, 1996, Impaired mobilization of cholesterol from stored cholesteryl esters in human (THP-1) macrophages, Atherosclerosis., 120, 135, 10.1016/0021-9150(95)05695-5 · DOI
  15. Hakamata, 1994, Species difference in cholesteryl ester cycle and HDL-induced cholesterol efflux from macrophage foam cells, Arterioscler. Thromb., 14, 1860, 10.1161/01.ATV.14.11.1860 · DOI
  16. Reue, 1997, Evidence for hormone-sensitive lipase mRNA in human monocyte/macrophages, Arterioscler. Thromb. Vasc. Biol., 17, 3428, 10.1161/01.ATV.17.12.3428 · DOI
  17. Johnson, 2000, Hormone sensitive lipase mRNA in both monocyte and macrophage forms of human THP-1 cell line, Comp. Biochem. Physiol., 126, 543, 10.1016/S0305-0491(00)00220-0 · DOI
  18. Contreras, 1994, Essential differences in cholesterol ester metabolism between human monocyte-derived and J774 macrophages: evidence against the presence of hormone sensitive lipase in human macrophage, Arterioscler. Thromb., 14, 443, 10.1161/01.ATV.14.3.443 · DOI
  19. Li, 1997, Modified low-density lipoprotein enhances the secretion of bile salt-stimulated cholesterol esterase by human monocyte-macrophages. Species-specific difference in macrophage cholesteryl ester hydrolase, J. Biol. Chem., 272, 28666, 10.1074/jbc.272.45.28666 · DOI
  20. Ghosh, 2000, Cholesteryl ester hydrolase in human monocyte/macrophage: cloning, sequencing and expression of full-length cDNA, Physiol. Genomics., 2, 1, 10.1152/physiolgenomics.2000.2.1.1 · DOI
  21. Lee, 2000, Differential expression of ACAT1 and ACAT2 among cells within liver, intestine, kidney, and adrenal of nonhuman primates, J. Lipid Res., 41, 1991, 10.1016/S0022-2275(20)32360-9 · DOI
  22. Reid, 2001, Hydrogen peroxide induced stress in human keratinocytes and its effect on biothionol toxicity, Toxicol. In Vitro., 15, 441, 10.1016/S0887-2333(01)00049-2 · DOI
  23. Ishikawa, 1974, Quantitative analysis of cholesterol in 5 to 20 microliter of plasma, J. Lipid Res., 15, 286, 10.1016/S0022-2275(20)36809-7 · DOI
  24. Klansek, 1995, Cholesterol quantitation by GLC: artifactual formation of short-chain steryl esters, J. Lipid Res., 36, 2261, 10.1016/S0022-2275(20)39210-5 · DOI
  25. Mahlberg, 1990, Potential problems in the use of commercial preparations of radiolabeled cholesterol, Atherosclerosis., 84, 95, 10.1016/0021-9150(90)90078-W · DOI
  26. Christian, 1999, Comparison of the capacity of beta-cyclodextrin derivatives and cyclophanes to shuttle cholesterol between cells and serum lipoproteins, J. Lipid Res., 40, 1475, 10.1016/S0022-2275(20)33390-3 · DOI
  27. Haynes, 2000, Efflux of cholesterol from different cellular pools, Biochemistry., 39, 4508, 10.1021/bi992125q · DOI
  28. Yamauchi, 2002, Differential regulation of apolipoprotein A-I/ATP binding cassette transporter A1-mediated cholesterol and phospholipid release, Biochim. Biophys. Acta., 1585, 1, 10.1016/S1388-1981(02)00304-9 · DOI
  29. Sliskovic, 1991, Therapeutic potential of ACAT inhibitors as lipid lowering and anti-atherosclerotic agents, Trends Pharmacol. Sci., 12, 194, 10.1016/0165-6147(91)90546-5 · DOI
  30. Matsuda, 1994, ACAT inhibitors as anti-atherosclerotic agents, Med. Res. Rev., 14, 271, 10.1002/med.2610140302 · DOI
  31. Matsuo, 1995, Effect of FR145237, a novel ACAT inhibitor on atherogenesis in cholesterol fed and WHHl rabbits. Evidence for a direct effect on the arterial wall, Biochim. Biophys. Acta., 1259, 254, 10.1016/0005-2760(95)00178-6 · DOI
  32. Nicolosi, 1998, The ACAT inhibitor, CI 1011 is effective in the prevention and regression of aortic fatty streak in hamsters, Atherosclerosis., 137, 77, 10.1016/S0021-9150(97)00279-7 · DOI
  33. Perrey, 2001, Preferential pharmacological inhibition of macrophage ACAT increases plaque formation in mouse and rabbit models of atherogenesis, Atherosclerosis., 155, 359, 10.1016/S0021-9150(00)00599-2 · DOI
  34. Accad, 2000, Massive xanthomatosis and altered composition of atherosclerotic lesions in hyperlipidemic mice lacking acyl CoA:cholesterol acyltransferase-1, J. Clin. Invest., 105, 711, 10.1172/JCI9021 · DOI
  35. Yagu, 2000, Absence of ACAT-1 attenuates atherosclerosis but causes dry eye and cutaneous xanthomatosis in mice with congenital hyperlipidemia, J. Biol. Chem., 275, 21324, 10.1074/jbc.M002541200 · DOI
  36. Fazio, 2001, Increased atherosclerosis in LDL receptor-null mice lacking ACAT1 in macrophages, J. Clin. Invest., 107, 163, 10.1172/JCI10310 · DOI
  37. Kellner-Weibel, 1999, Cytotoxic cholesterol is generated by the hydrolysis of cytoplasmic cholesteryl ester and transported to the plasma membrane, Atherosclerosis., 146, 309, 10.1016/S0021-9150(99)00155-0 · DOI
  38. Heinonen, 2002, Inhibition of acyl coenzyme A-cholesterol acyltransferase: a possible treatment of atherosclerosis?, Curr. Atheroscler. Rep., 4, 65, 10.1007/s11883-002-0064-9 · DOI
  39. Escary, 1999, Paradoxical effect on atherosclerosis of hormone-sensitive lipase overexpression in macrophages, J. Lipid Res., 40, 397, 10.1016/S0022-2275(20)32443-3 · DOI
  40. Ghosh, 2001, Cloning of the human cholesteryl ester hydrolase promoter: identification of functional peroxisomal proliferator-activated receptor responsive elements, Biochem. Biophys. Res. Commun., 284, 1065, 10.1006/bbrc.2001.5078 · DOI

Cited by

Sorted by citations

Metadata

Authors
Ghosh, S., St. Clair, R. W., & Rudel, L. L.
Venue
Journal of Lipid Research
Publisher
Elsevier BV
Published
2003-10
Volume
44
Issue
10
Pages
1833-1840
ISSN
0022-2275
Cited by
55
References
1
Language
en

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review