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Cited by 270

The DOI matches its Crossref record.

Further Studies on Leydig Cell Function in Old Age

RUBENS, R., DHONT, M., VERMEULEN, A.·1974·The Journal of Clinical Endocrinology & Metabolism
DOI 10.1210/jcem-39-1-40
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Abstract

Pathophysiological mechanisms leading to a decreased Leydig cell function in old males were studied. Increased basal plasma LH and FSH levels, an increased pituitary reaction to LHRH 200 μg iv, and a decreased responsiveness of the Leydig cells to hCG stimulation, lead to the conclusion that the decreased Leydig cell function in old age has a primary testicular origin. The plasma estrone and estradiol levels are slightly but significantly increased in elderly males, the free estradiol concentration is however unchanged. There exists a highly significant correlation between the (log transformed) apparent free testosterone concentration and LH and FSH levels suggesting that the feedback of the gonadotropin secretion is regulated via the free rather than via total testosterone levels.

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Authors
RUBENS, R., DHONT, M., & VERMEULEN, A.
Venue
The Journal of Clinical Endocrinology & Metabolism
Publisher
The Endocrine Society
Published
1974-07
Volume
39
Issue
1
Pages
40-45
ISSN
  • 0021-972X (ISSN-L)
  • 1945-7197
DOI
10.1210/jcem-39-1-40
OpenAlex
W2092790183
Cited by
270
References
0
FWCI
8.28
Type
journal article
Language
en
Open access
closed

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.