The DOI matches its Crossref record; 37 of the 37 references it cites were found in a registry or an index.
Parental transmission of type 2 diabetes: the Framingham Offspring Study
Open accessAbstract
Study of parental transmission of diabetes provides insight into the relative contributions of underlying maternal and paternal influences. We estimated risk for type 2 diabetes and milder degrees of glucose intolerance associated with parental diabetes among subjects of the population-based Framingham Offspring Study, in which participants are primarily Caucasian and at relatively low risk for diabetes and for which both parental and offspring phenotypes were ascertained by direct examination. Parental diabetes, assessed over 40 years of biennial follow-up, was defined by use of hypoglycemic drug therapy or a casual plasma glucose level > or = 11.1 mmol/l at any examination. Offspring glucose tolerance, assessed over 20 years of quadrennial follow-up, was defined by fasting plasma glucose levels > or = 7.8 mmol/l at any two examinations, use of hypoglycemic drug therapy at any examination, or with a 75-g oral glucose tolerance test (1980 World Health Organization criteria) at the most recent examination. We calculated odds ratios (ORs) and 95% CIs for offspring glucose tolerance status using generalized estimating equations to account for differential correlations within and between families. The 2,527 offspring came from 1,303 nuclear families, of which 77.6% had two or more siblings per family and in which the prevalence of parental diabetes was 24.6%. The mean offspring age was 54 years (range 26-82), 53% were women, 8.6% had diabetes, 11.4% had impaired glucose tolerance, 76.3% had no parental diabetes, 10.5% had maternal diabetes, 11.5% had paternal diabetes, and 1.7% had bilineal diabetes. Relative to individuals without parental diabetes, the age-adjusted ORs (95% CI) for offspring type 2 diabetes or abnormal glucose tolerance (fasting plasma glucose > or = 6.1 mmol/l or 2-h postchallenge glucose tolerance > or = 7.8 mmol/l) among individuals with maternal diabetes were 3.4 (2.3-4.9) and 2.7 (2.0-3.7), respectively; among individuals with paternal diabetes were 3.5 (2.3-5.2) and 1.7 (1.2-2.4), respectively; and among individuals with bilineal diabetes were 6.1 (2.9-13.0) and 5.2 (2.6-10.5), respectively. Although maternal and paternal diabetes conferred equivalent risk for offspring type 2 diabetes, offspring with maternal diabetes were slightly more likely to have abnormal glucose tolerance compared with those with paternal diabetes (OR 1.6, 95% CI 1.1-2.4). Offspring with maternal diabetes and an age of onset of <50 years had marked increased risk for both type 2 diabetes (9.7, 4.3-22.0) and abnormal glucose tolerance (9.0, 4.2-19.7). We conclude that risk ratios for offspring type 2 diabetes are consistent with a simple additive risk model, where risk when both parents are affected equals the sum of risk when either parent is affected. For maternal diabetes to confer excess risk for mild but not overt glucose intolerance, offspring of diabetic fathers may transit abnormal to impaired glucose tolerance relatively quickly, or diabetic mothers may transmit risk for a mild slowly progressive form of abnormal glucose tolerance in addition to overt diabetes. Very high risk for abnormal glucose homeostasis among offspring with young age-of-onset maternal diabetes is consistent with hypotheses that perinatal exposures increase diabetes risk. Given equivalent risk ratios for type 2 diabetes, fathers may transmit unique paternal genetic factors of similar strength to maternal environmental factors.
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
The source backs part of the claim, not all of it — or backs it for a different population, condition or comparison.
In the citing paper
“Diabetes during pregnancy also predisposes offspring for the development of obesity, T2D and cardiometabolic disease risk factors [33,70,71,72,73,74,75,76,77] (Figure 1).”
From Recent Experimental Studies of Maternal Obesity, Diabetes during Pregnancy and the Developmental Origins of Cardiovascular Disease (2022) · cites this paper as [33]
Experimental — accuracy not yet measuredThe cited source says what the claim attributes to it.
In the citing paper
“Powerful evidence for a genetic component to T2D ( 1 ) has driven extensive efforts to identify genetic variants contributing to risk.”
From β-Cell Failure in Type 2 Diabetes: Postulated Mechanisms and Prospects for Prevention and Treatment (2014) · cites this paper as [1]
The cited source says what the claim attributes to it.
In the citing paper
“maternal diabetes conferred a greater risk of abnormal glucose tolerance in the offspring, when compared to paternal diabetes [33].”
From Recent Experimental Studies of Maternal Obesity, Diabetes during Pregnancy and the Developmental Origins of Cardiovascular Disease (2022) · cites this paper as [33]
The cited source says what the claim attributes to it.
In the citing paper
“maternal and paternal diabetes conferred equivalent risk for offspring T2D”
From Recent Experimental Studies of Maternal Obesity, Diabetes during Pregnancy and the Developmental Origins of Cardiovascular Disease (2022) · cites this paper as [33]
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Report of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus
The Expert Committee on the Diagnosis and Classification of Diabetes Mellitus* (2002) · Diabetes Care · doi:10.2337/diacare.25.2007.s5 · Cited by 10,325
Longitudinal Data Analysis for Discrete and Continuous Outcomes
Zeger, S. L., & Liang, K. (1986) · Biometrics · doi:10.2307/2531248 · Cited by 7,781
Extension of Multiple Range Tests to Group Means with Unequal Numbers of Replications
Kramer, C. Y. (1956) · Biometrics · doi:10.2307/3001469 · Cited by 3,146
Barker, D. J. P., Hales, C. N., Fall, C. H. D., Osmond, C., Phipps, K., & Clark, P. M. S. (1993) · Diabetologia · doi:10.1007/bf00399095 · Cited by 2,468
DeChiara, T. M., Efstratiadis, A., & Robertsen, E. J. (1990) · Nature · doi:10.1038/345078a0 · Cited by 1,663
AN INVESTIGATION OF CORONARY HEART DISEASE IN FAMILIES
Kannel, W. B., Feinleib, M., McNAMARA, P. M., Garrison, R. J., & Castelli, W. P. (1979) · American Journal of Epidemiology · doi:10.1093/oxfordjournals.aje.a112813 · Cited by 1,639
Harris, M. I., Hadden, W. C., Knowler, W. C., & Bennett, P. H. (1987) · Diabetes · doi:10.2337/diab.36.4.523 · Cited by 1,096
Barnett, A. H., Eff, C., Leslie, R. D., & Pyke, D. A. (1981) · Diabetologia · doi:10.1007/bf00262007 · Cited by 966
DIABETES INCIDENCE IN PIMA INDIANS: CONTRIBUTIONS OF OBESITY AND PARENTAL DIABETES1
KNOWLER, W. C., PETTITT, D. J., SAVAGE, P. J., & BENNETT, P. H. (1981) · American Journal of Epidemiology · doi:10.1093/oxfordjournals.aje.a113079 · Cited by 561
Congenital susceptibility to NIDDM. Role of intrauterine environment
Pettitt, D. J., Aleck, K. A., Baird, H. R., Carraher, M. J., Bennett, P. H., & Knowler, W. C. (1988) · Diabetes · doi:10.2337/diab.37.5.622 · Cited by 530
Cited by
Sorted by citations
Items 401–500 of 669
Roberts‐Thomson, K. M., Hu, D., Russell, R., Greenaway, T. M., Betik, A. C., Parker, L., Kaur, G., Richards, S. M., Premilovac, D., Wadley, G. D., et al. (2022) · American Journal of Physiology-Endocrinology and Metabolism · doi:10.1152/ajpendo.00109.2022 · Cited by 6
Murai, N., Saito, N., Nii, S., Nishikawa, Y., Suzuki, A., Kodama, E., Iida, T., Mikura, K., Imai, H., Hashizume, M., et al. (2022) · Metabolism Open · doi:10.1016/j.metop.2022.100196 · Cited by 6
The comparison of second phase insulin secretion in patients treated with repaglinide or gliclazide
Yang, C.-C., Lin, J.-D., Kuo, K., Wu, C., Li, J.-C., Hung, Y. J., Wang, T. F., Lee, C., Kuo, S. W., & Pei, D. (2007) · Diabetes & Metabolic Syndrome Clinical Research & Reviews · doi:10.1016/j.dsx.2006.11.006 · Cited by 6
Subclinical Cardiovascular Disease Assessment in Persons with Diabetes
Warraich, H. J., & Nasir, K. (2013) · Current Cardiology Reports · doi:10.1007/s11886-013-0358-2 · Cited by 7
Janghorbani, M., & Amini, M. (2012) · Metabolic Syndrome and Related Disorders · doi:10.1089/met.2012.0070 · Cited by 6
Risk for metabolic syndrome predisposes to alterations in the thalamic metabolism
Heikkilä, O., Lundbom, N. M. I., Timonen, M. H., Groop, P., Heikkinen, S., & Mäkimattila, S. (2008) · Metabolic Brain Disease · doi:10.1007/s11011-008-9094-5 · Cited by 6
de Visser, K. L., Landman, G. W. D., Jong, B. M., de Visser, W., Meerman, G. J. T., & Bilo, H. J. G. (2015) · PLoS ONE · doi:10.1371/journal.pone.0132549 · Cited by 6
Deng, L., Liu, S., Gong, Y., Tian, H., Liu, Y., Song, J., Ran, X., Yu, H., Zhang, X., Long, Y., et al. (2016) · Metabolic Syndrome and Related Disorders · doi:10.1089/met.2016.0002 · Cited by 6
Hermans, M. P., Ahn, S. A., & Rousseau, M. F. (2018) · Journal of Diabetes · doi:10.1111/1753-0407.12840 · Cited by 6
Family history and pedigree charting--a simple genetic tool for Indian diabetics.
Joshi, S., & Parikh, R. M. (2006) · PubMed · Cited by 6
McCance, D. R., & Cassidy, L. (2024) · Wiley eBooks · doi:10.1002/9781119697473.ch71 · Cited by 7
Peng, H., Wang, M., Guo, H., Hou, T., Li, Y., Zhang, H., Tan, Y., Qin, X., Wu, Y., Chen, D., et al. (2025) · Nutrients · doi:10.3390/nu17081361 · Cited by 5
Heianza, Y., & Qi, L. (2017) · Elsevier eBooks · doi:10.1016/b978-0-12-802928-2.00029-1 · Cited by 6
Brandão, M., Lopes, C. M. M., & Ramos, E. (2013) · Preventive Medicine · doi:10.1016/j.ypmed.2013.06.028 · Cited by 5
Early beta-cell dysfunction characterizes males with type 2 diabetes of Yemenite origin
Magnazi, M. B., Zornitzki, T., Ulman, M., Madar, Z., & Knobler, H. (2016) · Acta Diabetologica · doi:10.1007/s00592-016-0838-0 · Cited by 6
Association of RETN gene polymorphism at +299 G>A with type 2 diabetes mellitus: a meta-analysis
Kumar, V., Singh, J., Aneja, A., & Singh, J. (2019) · International Journal of Diabetes in Developing Countries · doi:10.1007/s13410-019-00746-x · Cited by 5
Yoshiuchi, I. (2021) · Biochemical Genetics · doi:10.1007/s10528-021-10045-y · Cited by 5
Laboratory diagnostics as a basis for 5P medicine
Николаевич, Щ. С., & Shcherbo, D. S. (2019) · Вестник Российского государственного медицинского университета · doi:10.24075/brsmu.2018.095 · Cited by 5
Sikjær, M. G., Klitgaard, A., Hilberg, O., & Løkke, A. (2022) · International Journal of COPD · doi:10.2147/copd.s364899 · Cited by 5
Kinship analysis of type 2 diabetes mellitus familial aggregation in Taiwan
Liao, P., Ting, M., Kuo, C., Ding, Y.-H., Lin, C.-M., & Hsu, K. (2022) · Biomedical Journal · doi:10.1016/j.bj.2022.07.003 · Cited by 5
Lapeyre, G., Cougnard‐Grégoire, A., Delyfer, M., Delcourt, C., Hadjadj, S., Blanco, L., Pupier, É., Rougier, M., Rajaobelina, K., Mohammedi, K., et al. (2017) · Diabetes & Metabolism · doi:10.1016/j.diabet.2017.02.006 · Cited by 5
Montes-Ochoa, S., Serna-Arrieta, K., Estrada-Ávila, S., Lopez, F. G., & Sánchez, I. P. (2016) · Medicina y Laboratorio · doi:10.36384/01232576.105 · Cited by 5
Appraising the Value of Genomic Association Studies
George, A. L. (2008) · Journal of the American Society of Nephrology · doi:10.1681/asn.2008050524 · Cited by 5
Circadian clock controlled glycolipid metabolism and its relevance to disease management
Lin, L., Huang, Y., Li, A., Cai, Y., Yan, Y., Huang, Y., He, L., Chen, Y., & Wang, S. (2025) · Biochemical Pharmacology · doi:10.1016/j.bcp.2025.116967 · Cited by 4
The prevention of adult cardiovascular disease must begin in childhood: evidence and imperative
Khoury, M., Ware, A. L., & McCrindle, B. W. (2025) · Nature Reviews Cardiology · doi:10.1038/s41569-025-01209-z · Cited by 10
Quaytman, J., David, N. L., Venugopal, S., Amorim, T., Beatrice, B., Toledo, F. G. S., Miller, R. G., Steinhauser, M. L., & Fazeli, P. K. (2024) · Contemporary Clinical Trials · doi:10.1016/j.cct.2024.107698 · Cited by 4
Chavey, A., Movassat, J., & Porth, B. (2011) · InTech eBooks · doi:10.5772/23027 · Cited by 4
Impaired Fasting Glucose Rate by Diabetes Family History
Kim, E.-H., Bae, S.-G., Kim, K.-Y., & Na, Y.-J. (2017) · Journal of Health Informatics and Statistics · doi:10.21032/jhis.2017.42.1.63 · Cited by 4
Amelia, R., Burhan, B., & Lelo, A. (2018) · Family Medicine & Primary Care Review · doi:10.5114/fmpcr.2018.79339 · Cited by 4
Leventer‐Roberts, M., Gofer, I., Barak‐Corren, Y., Reis, B. Y., & Balicer, R. D. (2019) · European Journal of Public Health · doi:10.1093/eurpub/ckz152 · Cited by 4
van der Meer, T. P., Wolffenbuttel, B. H. R., & Patel, C. J. (2021) · Diabetologia · doi:10.1007/s00125-021-05419-1 · Cited by 6
Pires, L. C., Marçola, L. G., SIQUEIRA, J. P. B. D., Vieira, N. A., Jorge, R. A., Barbosa, A. P., & Batista, M. J. (2022) · Revista Brasileira de Medicina de Família e Comunidade · doi:10.5712/rbmfc17(44)2986 · Cited by 4
Pattern Visual Evoked Potential in Non Diabetic offspring‘s of Type II Diabetes
Achuthan, A., & Ramesh, J. (2015) · The Journal of Medical Research · doi:10.31254/jmr.2015.1306 · Cited by 4
Goyal, Y., Verma, A. K., Kumar, S., Bhatt, D., Ahmad, F., & Dev, K. (2022) · Gene Reports · doi:10.1016/j.genrep.2022.101655 · Cited by 5
A replication study of 49 Type 2 diabetes risk variants in a Punjabi Pakistani population
Zia, A., Wang, X., Bhatti, A., Demirci, F. Y., Zhao, W., Rasheed, A., Samuel, M., Kiani, A. K., Ismail, M., Zafar, J., et al. (2015) · Diabetic Medicine · doi:10.1111/dme.13012 · Cited by 5
Murraya koenigii Leaves and Their Use in Dementia
Mani, V., Ahmad, A., Ramasamy, K., Lim, S. M., & Majeed, A. B. A. (2014) · Elsevier eBooks · doi:10.1016/b978-0-12-407824-6.00097-5 · Cited by 4
Study of Diabetes in Dharwad- an Urban Area in India
Patil, P. S., Dixit, U. R., & Hiralal, B. D. (2011) · doi:10.17485/ijst/2011/v4i11/30274 · Cited by 4
Eliraqi, G. M., Vistisen, D., Lauritzen, T., Sandbæk, A., Jørgensen, M. E., & Færch, K. (2015) · Diabetic Medicine · doi:10.1111/dme.12764 · Cited by 4
Perlmuter, L. C., Singh, S. P., Gabhart, J. M., Pungan, R., & Siedlarz, M. (2008) · Experimental Aging Research · doi:10.1080/03610730701876961 · Cited by 4
Ordered Stratification to Reduce Heterogeneity in Linkage to Diabetes‐related Quantitative Traits
Meigs, J. B., Manning, A. K., Dupuis, J., Liu, C., Florez, J. C., & Cupples, L. A. (2008) · Obesity · doi:10.1038/oby.2008.354 · Cited by 4
Self-citationThe interplay of lifestyle and genetic susceptibility in Type 2 diabetes risk
Franks, P. W., & Shungin, D. (2011) · Diabetes Management · doi:10.2217/dmt.11.3 · Cited by 4
Maternal and Paternal Transmission of Diabetes: Influence of Nutritional Factors
Rahim, M. (2015) · Journal of Diabetes & Metabolism · doi:10.4172/2155-6156.1000504 · Cited by 4
Long-term implications: Child and adult
Dabelea, D., & Pettitt, D. J. (2008) · Series in maternal-fetal medicine · doi:10.3109/9781439802007.048 · Cited by 4
Zarkoob, H., Lewinsky, S., Almgren, P., Melander, O., & Fakhrai-Rad, H. (2017) · PLoS ONE · doi:10.1371/journal.pone.0180180 · Cited by 4
Genetics of Diabetes and Diabetic Complications
Prasad, R. B., Ahlqvist, E., & Groop, L. (2018) · Endocrinology · doi:10.1007/978-3-319-45015-5_6 · Cited by 5
Latent Autoimmune Diabetes in Adults
Brooks‐Worrell, B., & Palmer, J. P. (2010) · Humana Press eBooks · doi:10.1007/978-1-60327-478-4_19 · Cited by 4
Ferreira, C. R., Gomes, P. C. F. D. L., Robison‡, K. M., Cooper, B. R., & Shannahan, J. H. (2024) · Molecular Omics · doi:10.1039/d3mo00214d · Cited by 4
Ndonwi, E. N., Matshazi, D. M., Davison, G. M., Erasmus, R. T., Kengne, A. P., & Matsha, T. E. (2025) · Biomedical Reports · doi:10.3892/br.2025.2022 · Cited by 3
Interplay of Modifiable and Non-Modifiable Risk Factors for Diabetes Mellitus in Saudi Adults
Jareebi, M. A., & Gosadi, I. M. (2025) · Diagnostics · doi:10.3390/diagnostics15192451 · Cited by 6
HEMŞİRELİK ÖĞRENCİLERİNİN DİYABET RİSKLERİNİN BELİRLENMESİ
DOĞAN, E. S., Şahin, C. K., Akan, D. D., PAKYÜZ, S. Ç., Işık, K., & MINGIR, K. (2022) · Izmir Democracy University Health Sciences Journal · doi:10.52538/iduhes.1107704 · Cited by 3
Serum Insulin and Insulin Resistance in the Off-Springs of Type 2 Diabetes Mellitus
Rahim, M., & Rahim, M. (2014) · Journal of Diabetes & Metabolism · doi:10.4172/2155-6156.1000425 · Cited by 3
Acevedo-Negrete, A. P., Porchia, L. M., González-Mejía, M. E., Torres‐Rasgado, E., Solis-Cano, D. G., Ruiz‐Vivanco, G., & Pérez-Fuentes, R. (2017) · Diabetes & Metabolic Syndrome Clinical Research & Reviews · doi:10.1016/j.dsx.2017.07.012 · Cited by 3
Role of harmonics and subharmonics in peripheral pulse analysis
Gd, J., Bhat, S. N., Sawant, M. S., & Deshpande, A. (2020) · MGM Journal of Medical Sciences · doi:10.4103/mgmj.mgmj_47_20 · Cited by 3
Fachirah, J., & Sari, M. I. (2021) · Open Access Macedonian Journal of Medical Sciences · doi:10.3889/oamjms.2021.5695 · Cited by 3
Obirikorang, C., Adu, E. A., Anto, E. O., Acheampong, E., Quaye, L., Amoah, B. Y., Annani-Akollor, M. E., Kwakye, A. S., Fokuoh, F., Appiah, M., et al. (2021) · Sci · doi:10.3390/sci3040040 · Cited by 3
Berhanie, H., Mihretie, Z., & Anandapandian, K. T. K. (2020) · Indian Journal of Medical Sciences · doi:10.25259/ijms_21_2019 · Cited by 3
Hamad, A. F., Yan, L., Leslie, W. D., Morin, S. N., Walld, R., Roos, L. L., Yang, S., & Lix, L. M. (2021) · Canadian Journal of Diabetes · doi:10.1016/j.jcjd.2021.04.005 · Cited by 3
Bauman, D. E., Beachy, B. R., & Ogbeide, S. A. (2018) · Springer eBooks · doi:10.1007/978-3-319-70539-2_15 · Cited by 3
The Infant of the Diabetic Mother: Metabolic Imprinting
Snell‐Bergeon, J. K., & Dabelea, D. (2009) · Humana Press eBooks · doi:10.1007/978-1-60327-250-6_20 · Cited by 3
Lauenborg, J., JØRGENSEN, M. K., Damm, P., Major‐Pedersen, A., Eiberg, H., Urhammer, S. A., Pedersen, O. B., & Hansen, T. F. (2011) · Acta Obstetricia Et Gynecologica Scandinavica · doi:10.1111/j.1600-0412.2011.01276.x · Cited by 3
Amini, M., & Janghorbani, M. (2009) · Diabetes & Metabolic Syndrome Clinical Research & Reviews · doi:10.1016/j.dsx.2009.02.005 · Cited by 3
TYPE 1 DIABETES IN PREGNANCY; INFLUENCES ON MOTHER AND FETUS
Nelson, S. M., & Lindsay, R. S. (2009) · Fetal and Maternal Medicine Review · doi:10.1017/s0965539509002344 · Cited by 3
Ponziani, M. C., Karamouzis, I., Mele, C., Chasseur, L., Zavattaro, M., Caputo, M., Samà, M. T., Busti, A., Pagano, L., Castello, L. M., et al. (2017) · HORMONES · doi:10.14310/horm.2002.1760 · Cited by 3
Kabisch, S., Meyer, N. M. T., Honsek, C., Kemper, M., Gerbracht, C., Arafat, A. M., Dambeck, U., Osterhoff, M. A., Weickert, M. O., & Pfeiffer, A. F. H. (2022) · Nutrients · doi:10.3390/nu14224721 · Cited by 3
Yan, Y. (2019) · Carolina Digital Repository (University of North Carolina at Chapel Hill) · doi:10.17615/kpne-0v56 · Cited by 3
Genetic Risk Factors for Type 2 Diabetes and Related Traits
Hertel, J. K. (2012) · NORA - Norwegian Open Research Archives · Cited by 3
Gillberg, L., Jacobsen, S. B., Ribel‐Madsen, R., Gjesing, A. P., Boesgaard, T. W., Ling, C., Pedersen, O. B., Hansen, T. F., & Vaag, A. A. (2013) · PLoS ONE · doi:10.1371/annotation/5c3cf392-57b5-4e80-9a66-4997d10200ae · Cited by 3
Maternal High Fiber Diet Protects Offspring Against Type 2 DiabetesPreprint
Toh, H., Thomson, J. A., & Jiang, P. (2020) · bioRxiv (Cold Spring Harbor Laboratory) · doi:10.1101/2020.09.26.315069 · Cited by 3
Albajy, M. A., & Mihailescu, D. F. (2024) · Health Problems of Civilization · doi:10.5114/hpc.2024.136467 · Cited by 2
Seth, T., Mehra, P., & Muhuri, P. K. (2024) · IEEE Transactions on Fuzzy Systems · doi:10.1109/tfuzz.2024.3456030 · Cited by 2
Al-Hajj, S., Khan, Z. U., Gul, A., Ahmed, O., Gupta, A., & Mlawa, G. (2024) · Cureus · doi:10.7759/cureus.72848 · Cited by 2
Mukherjee, A. K., Debbarman, T., Banerjee, B. D., & Siddiqi, S. S. (2024) · Indian Journal of Endocrinology and Metabolism · doi:10.4103/ijem.ijem_43_24 · Cited by 2
Functional mechanisms for type 2 diabetes-associated genetic variants
Zhu, X.-W., Deng, F., Wu, L., Tang, Z., & Lei, S. (2015) · Journal of Diabetes and its Complications · doi:10.1016/j.jdiacomp.2015.02.007 · Cited by 2
Genetics of Type 2 Diabetes: A Review
Fakruddin, M. (2019) · Journal of Current and Advance Medical Research · doi:10.3329/jcamr.v6i1.40787 · Cited by 2
Zimmermann, B. M., Starke, G., Shaw, D., Elger, B. S., & Koné, I. (2020) · Swiss Medical Weekly · doi:10.4414/smw.2020.20274 · Cited by 2
Factores de riesgo para obesidad en población femenina del Istmo de Tehuantepec, Oaxaca, México
Blas, B. H., Blas, S. R., Martínez, A. Z., Sánchez-Cruz, G., & Bernardino-Hernández, H. U. (2020) · Acta Médica Costarricense · doi:10.51481/amc.v62i1.1055 · Cited by 1
Glycaemic control and family history of diabetes mellitus: is it all in the genes?
Chetty, R. R., & Pillay, S. (2021) · Journal of Endocrinology Metabolism and Diabetes of South Africa · doi:10.1080/16089677.2021.1897229 · Cited by 2
Lotfy, A., Hemeda, M., Ali, A., Bauomy, I. M., & Shola, M. G. H. (2020) · Al-Azhar Medical Journal · doi:10.21608/amj.2020.91617 · Cited by 4
Koziatek, C. A., Bohart, I., Caldwell, R., Swartz, J. L., Rosen, P. E., Desai, S., Krol, K., Neill, D. B., & Lee, D. C. (2023) · Journal of Urban Health · doi:10.1007/s11524-023-00771-6 · Cited by 2
Type 2 Diabetes-Etiology, Epidemiology, Pathogenesis, Treatment
Burns, C., & Francis, N. (2023) · Springer eBooks · doi:10.1007/978-3-319-12125-3_34-3 · Cited by 2
Children and Adolescents With Hybrid Diabetes: A Management Conundrum
Gagnon, C. A., Schmitt, J. A., & Ashraf, A. P. (2023) · Endocrine Practice · doi:10.1016/j.eprac.2023.10.134 · Cited by 2
Wajsbrot, N. B., Leite, N. C., de França, P. H. C., Cardoso, C. R. L., Salles, G. F., & Villela‐Nogueira, C. A. (2023) · Digestive Diseases and Sciences · doi:10.1007/s10620-023-08214-7 · Cited by 2
Aljani, B., Garbe, A. I., Sedlmeier, E., Lickert, R., Rost, F., Ziegler, A., Bonifacio, E. E., & Eugster, A. (2024) · Placenta · doi:10.1016/j.placenta.2024.10.008 · Cited by 3
DASHTI, M. H., Ali, N. M., Alsaleh, H., John, S. E., Nizam, R., Thanaraj, T. A., & Al‐Mulla, F. (2024) · Frontiers in Endocrinology · doi:10.3389/fendo.2024.1443737 · Cited by 2
Mudd‐Martin, G., & Martinez, M. C. (2011) · Nursing Clinics of North America · doi:10.1016/j.cnur.2011.02.008 · Cited by 2
Cauchi, S., & Froguel, P. (2010) · International Journal of Diabetes Mellitus · doi:10.1016/j.ijdm.2010.05.013 · Cited by 2
Family history of diabetes and awareness of risk factors in Type 2 Diabetic patients
Sultana, R., Muhammad, T., & Mehmood, R. (2010) · DOAJ (DOAJ: Directory of Open Access Journals) · Cited by 2
Tyrovolas, S., Polychronopoulos, E., Mariolis, A. D., Piscopo, S., Valacchi, G., Makri, K., Zeimbekis, A., Tyrovola, D., Bountziouka, V., Gotsis, E., et al. (2016) · Angiology · doi:10.1177/0003319716642140 · Cited by 3
Alcocer, B. S., Valle, A. R. C., & Guerrero, J. G. (2016) · RIDE Revista Iberoamericana para la Investigación y el Desarrollo Educativo · doi:10.23913/ride.v6i12.216 · Cited by 2
Alcocer, B. S., Valle, A. R. C., & Guerrero, J. G. (2016) · Revista Iberoamericana para la Investigación y el Desarrollo Educativo: RIDE · Cited by 2
Oostvogels, A. J. J. M., Landstra, C. P., Britsemmer, L., Lodewijkx, R., Stronks, K., Roseboom, T. J., & Vrijkotte, T. G. M. (2017) · Diabetes & Metabolism · doi:10.1016/j.diabet.2017.01.003 · Cited by 2
Udler, M. S., & Florez, J. C. (2017) · Elsevier eBooks · doi:10.1016/b978-0-12-800685-6.00014-x · Cited by 2
Middleton, T. L., Brooks, B. A., Constantino, M., Wu, T., Wong, J., & Yue, D. K. (2019) · Journal of Diabetes and its Complications · doi:10.1016/j.jdiacomp.2019.107440 · Cited by 2
Hu, M., Hu, S., Tan, J., & Yang, Y. (2023) · Nutrition Metabolism and Cardiovascular Diseases · doi:10.1016/j.numecd.2023.01.018 · Cited by 2
Kowaleski‐Jones, L., Zick, C. D., Brown, B. B., Curtis, D. S., Meeks, H., & Smith, K. R. (2023) · Journal of Biosocial Science · doi:10.1017/s0021932023000056 · Cited by 2
Walia, G. K., Sharma, P., Agarwal, T., Lal, M., Negandhi, H., Prabhakaran, D., Khadgawat, R., SACHDEVA, M. P., & Gupta, V. (2023) · PLoS ONE · doi:10.1371/journal.pone.0291339 · Cited by 2
Preconception Care for Women with Diabetes Mellitus
Blank, H., & Wyckoff, J. (2009) · Humana Press eBooks · doi:10.1007/978-1-60327-250-6_15 · Cited by 2
Swanson, T. N., Parker, M. N., Byrne, M. E., Ramirez, E., Kwarteng, E. A., Faulkner, L. M., Djan, K. G., Zenno, A., Chivukula, K. K., LeMay‐Russell, S., et al. (2021) · Pediatric Diabetes · doi:10.1111/pedi.13286 · Cited by 3
Maternal and paternal transmission of type 2 diabetes
Hemminki, K., Li, X., Sundquist, J., & Sundquist, K. (2011) · Journal of Internal Medicine · doi:10.1111/j.1365-2796.2011.02413.x · Cited by 2
Early detection of type 2 diabetes mellitus in Chinese and Indian adult population
Gao, W. (2010) · Työväentutkimus Vuosikirja · Cited by 2
Metadata
- Authors
- Meigs, J. B., Cupples, L. A., & Wilson, P. W.
- Affiliations
- Harvard University (US)
- Venue
- Diabetes
- Publisher
- American Diabetes Association
- Published
- 2000-12-01
- Volume
- 49
- Issue
- 12
- Pages
- 2201-2207
- ISSN
- 0012-1797 (ISSN-L)
- 1939-327X
- OpenAlex
- W1989797259
- Cited by
- 666
- References
- 37
- Citations by year
- 2026: 15 · 2025: 17 · 2024: 27 · 2023: 29 · 2022: 44 · 2021: 40 · 2020: 37 · 2019: 37 · 2018: 31 · 2017: 32 · 2016: 26 · 2015: 41 · 2014: 36 · 2013: 44 · 2012: 28
- FWCI
- 2.42
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Locations
- Diabetes (publishedVersion)
- PubMed (publishedVersion)
- Funders
- American Diabetes Association
- National Institutes of Health
- National Heart, Lung, and Blood Institute (N01-HC-38083)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes, Cardiovascular Risks, and Lipoproteins
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genetic Associations and Epidemiology
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Diabetes and associated disorders
- Keywords
- type 2 diabetes, parental diabetes, familial aggregation, maternal diabetes, paternal diabetes, impaired glucose tolerance, diabetes risk, Framingham Offspring Study
- MeSH terms
- Adult, Aged, Aged, 80 and over, Blood Glucose, Diabetes Mellitus, Type 2, Fathers, Female, Glucose Tolerance Test, Humans, Hypoglycemic Agents, Insulin Resistance, Male, Middle Aged, Mothers, Cohort Studies, Prevalence, Odds Ratio, Randomized Controlled Trials as Topic, Genetic Predisposition to Disease
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
