The DOI matches its Crossref record; 115 of the 115 references it cites were found in a registry or an index.
RHO Family GTPases in the Biology of Lymphoma
Abstract
RHO GTPases are a class of small molecules involved in the regulation of several cellular processes that belong to the RAS GTPase superfamily. The RHO family of GTPases includes several members that are further divided into two different groups: typical and atypical. Both typical and atypical RHO GTPases are critical transducers of intracellular signaling and have been linked to human cancer. Significantly, both gain-of-function and loss-of-function mutations have been described in human tumors with contradicting roles depending on the cell context. The RAS family of GTPases that also belong to the RAS GTPase superfamily like the RHO GTPases, includes arguably the most frequently mutated genes in human cancers (K-RAS, N-RAS, and H-RAS) but has been extensively described elsewhere. This review focuses on the role of RHO family GTPases in human lymphoma initiation and progression.
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“However, 46% of cases are observed in diffuse large B cell lymphoma (DLBCL) and are found to be related to somatic hypermutations in the noncoding region, rather than chromosomal rearrangements [41].”
From The Role of RhoH in TCR Signalling and Its Involvement in Diseases (2021) · cites this paper as [41]
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Cites
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Cited by
RHO GTPases: from new partners to complex immune syndromes
El Masri, R., & Delon, J. (2021) · Nature Reviews Immunology · doi:10.1038/s41577-021-00500-7 · Cited by 131
The Role of RhoH in TCR Signalling and Its Involvement in Diseases
Ahmad Mokhtar, A. M., Hashim, I. F., Mohd Zaini Makhtar, M., Salikin, N. H., & Amin-Nordin, S. (2021) · Cells · doi:10.3390/cells10040950 · Cited by 25
Metadata
- Authors
- Voena, C., & Chiarle, R.
- Affiliations
- Department of Molecular Biotechnology and Health Sciences, University of Torino, 10126 Torino, Italy
- ORCID
- Voena, Claudia
- Venue
- Cells
- Publisher
- MDPI AG
- Published
- 2019-06-26
- Volume
- 8
- Issue
- 7
- Pages
- 646
- ISSN
- 2073-4409
- PMC
- PMC6678807
- Cited by
- 35
- References
- 115
- Language
- en
- Funders
- National Cancer Institute (R01 CA196703-01)
- H2020 Marie Skłodowska-Curie Actions (675712)
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