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The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

Resveratrol Protects against Oxidized LDL-Induced Breakage of the Blood-Brain Barrier by Lessening Disruption of Tight Junctions and Apoptotic Insults to Mouse Cerebrovascular Endothelial Cells ,

Lin, Yi-Ling, Chang, Huai-Chia, Chen, Ta-Liang, Chang, Jin-Han, Chiu, Wen-Ta, Lin, Jia-Wei, Chen, Ruei-Ming·2010·The Journal of Nutrition
DOI 10.3945/jn.110.123505
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  • Disruption of BBB integrity can lead to an increased permeability of the endothelium for cholesterol-carrying lipoproteins and facilitate the exposure of blood-circulating components to neurons.

    “In addition, disruption of BBB integrity can lead to an increased permeability of the endothelium for cholesterol-carrying lipoproteins and facilitate the exposure of bloodcirculating components to neurons.[23][24][25]”

    From Canonical Wnt pathway and the LDL receptor superfamily in neuronal cholesterol homeostasis and function (2024) · cites this paper as [24]

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Research works printed but not linked: 39

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  4. Huber, 2001, Molecular physiology and pathophysiology of tight junctions in the blood-brain barrier, Trends Neurosci., 24, 719, 10.1016/S0166-2236(00)02004-X · DOI
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  8. van der Goes, 2001, Reactive oxygen species enhance the migration of monocytes across the blood-brain barrier in vitro, FASEB J., 15, 1852, 10.1096/fj.00-0881fje · DOI
  9. Matsuura, 2008, Oxidation of LDL and its clinical implication, Autoimmun Rev., 7, 558, 10.1016/j.autrev.2008.04.018 · DOI · Paper page
  10. Zorn-Pauly, 2005, Oxidized LDL induces ventricular myocyte damage and abnormal electrical activity–role of lipid hydroperoxides, Cardiovasc Res., 66, 74, 10.1016/j.cardiores.2004.12.009 · DOI
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  12. Schroeter, 2001, Flavonoids protect neurons from oxidized low-density-lipoprotein-induced apoptosis involving c-Jun N-terminal kinase (JNK), c-Jun and caspase-3, Biochem J., 358, 547, 10.1042/bj3580547 · DOI
  13. Chen, 2007, Oxidized low-density lipoprotein induces apoptotic insults to mouse cerebral endothelial cells via a Bax-mitochondria-caspase protease pathway, Toxicol Appl Pharmacol., 219, 42, 10.1016/j.taap.2006.11.031 · DOI
  14. Salvayre, 2002, Oxidized low-density lipoprotein-induced apoptosis, Biochim Biophys Acta., 1585, 213, 10.1016/S1388-1981(02)00343-8 · DOI
  15. Pervaiz, 2003, Resveratrol: from grapevines to mammalian biology, FASEB J., 17, 1975, 10.1096/fj.03-0168rev · DOI
  16. Baur, 2006, Therapeutic potential of resveratrol: the in vivo evidence, Natl Rev Drug Discov., 5, 493, 10.1038/nrd2060 · DOI · Paper page
  17. Gusman, 2001, A reappraisal of the potential chemopreventive and chemotherapeutic properties of resveratrol, Carcinogenesis., 22, 1111, 10.1093/carcin/22.8.1111 · DOI
  18. Yoshida, 2007, Resveratrol ameliorates experimental autoimmune myocarditis, Circ J., 71, 397, 10.1253/circj.71.397 · DOI
  19. Danz, 2009, Resveratrol prevents doxorubicin cardiotoxicity through mitochondrial stabilization and the Sirt1 pathway, Free Radic Biol Med., 46, 1589, 10.1016/j.freeradbiomed.2009.03.011 · DOI
  20. Xu, 1998, Involvement of de novo ceramide biosynthesis in tumor necrosis factor-alpha/cycloheximide-induced cerebral endothelial cell death, J Biol Chem., 273, 16521, 10.1074/jbc.273.26.16521 · DOI
  21. Li, 2003, LOX-1 mediates oxidized low-density lipoprotein-induced expression of matrix metalloproteinases in human coronary artery endothelial cells, Circulation., 107, 612, 10.1161/01.CIR.0000047276.52039.FB · DOI
  22. Liu, 1996, Lipoperoxidative injury to macrophages by oxidatively modified low density lipoprotein may play an important role in foam cell formation, Atherosclerosis., 121, 55, 10.1016/0021-9150(95)05683-1 · DOI
  23. Wu, 2009, Lipopolysaccharide triggers macrophage activation of inflammatory cytokine expression, chemotaxis, phagocytosis, and oxidative ability via a toll-like receptor 4-dependent pathway: validated by RNA interference, Toxicol Lett., 191, 195, 10.1016/j.toxlet.2009.08.025 · DOI
  24. Chen, 2003, Propofol suppresses macrophage functions and modulates mitochondrial membrane potential and cellular adenosine triphosphate synthesis, Anesthesiology., 98, 1178, 10.1097/00000542-200305000-00021 · DOI
  25. Chang, 2009, Cytoskeleton interruption in human hepatoma HepG2 cells induced by ketamine occurs possibly through suppression of calcium mobilization and mitochondrial function, Drug Metab Dispos., 37, 24, 10.1124/dmd.108.023325 · DOI
  26. Lee, 2009, Apoptotic insults to human HepG2 cells induced by S-(+)-ketamine occurs through activation of a Bax-mitochondria-caspase protease pathway, Br J Anaesth., 102, 80, 10.1093/bja/aen322 · DOI
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  28. Chang, 2010, Lipoteichoic acid-induced TNF-alpha and IL-6 gene expressions and oxidative stress production in macrophages are suppressed by ketamine through downregulating tolllike receptor 2-mediated activation of ERK1/2 and NFkappaB, Shock., 33, 485, 10.1097/SHK.0b013e3181c3cea5 · DOI
  29. Tai, 2007, Pretreatment with low nitric oxide protects osteoblasts from high nitric oxide-induced apoptotic insults through regulation of c-Jun N-terminal kinase/c-Jun-mediated Bcl-2 gene expression and protein translocation, J Orthop Res., 25, 625, 10.1002/jor.20365 · DOI
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  33. Yap, 2001, Microtubule integrity is essential for apical polarization and epithelial morphogenesis in the thyroid, Cell Motil Cytoskeleton., 48, 201, 10.1002/1097-0169(200103)48:3<201::AID-CM1009>3.0.CO;2-C · DOI
  34. Molitoris, 1996, Cellular ATP depletion induces disruption of the spectrin cytoskeletal network, Am J Physiol., 271, F790
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  36. Ho, 2009, Runx2-mediated bcl-2 gene expression contributes to nitric oxide protection against hydrogen peroxide-induced osteoblast apoptosis, J Cell Biochem., 108, 1084, 10.1002/jcb.22338 · DOI
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Metadata

Authors
Lin, Y.-L., Chang, H.-C., Chen, T.-L., Chang, J.-H., Chiu, W.-T., Lin, J.-W., & Chen, R.-M.
Venue
The Journal of Nutrition
Publisher
Elsevier BV
Published
2010-12
Volume
140
Issue
12
Pages
2187-2192
ISSN
0022-3166
DOI
10.3945/jn.110.123505
Cited by
77
References
3
Language
en
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