Campisi, Judith

Controlled induction of DNA double-strand breaks in the mouse liver induces features of tissue ageing

White, R. R., Milholland, B., de Bruin, A., Curran, S., Laberge, R.-M., van Steeg, H., Campisi, J., Maslov, A. Y., & Vijg, J. (2015) · Nature Communications

Genetics, Aging, and Longevity in Model OrganismsTelomeres, Telomerase, and Senescence

Cited by 107

Acute myeloid leukemia induces protumoral p16INK4a-driven senescence in the bone marrow microenvironment

Abdul-Aziz, A. M., Sun, Y., Hellmich, C., Marlein, C. R., Mistry, J., Forde, E., Piddock, R. E., Shafat, M. S., Morfakis, A., Mehta, T., et al. (2019) · Blood

Telomeres, Telomerase, and SenescenceAcute Myeloid Leukemia Research

Cited by 105

Cellular Senescence Contributes to Large Elastic Artery Stiffening and Endothelial Dysfunction With Aging: Amelioration With Senolytic Treatment

Clayton, Z. S., Rossman, M. J., Mahoney, S. A., Venkatasubramanian, R., Maurer, G. S., Hutton, D. A., VanDongen, N. S., Greenberg, N. T., Longtine, A. G., Ludwig, K. R., et al. (2023) · Hypertension

Telomeres, Telomerase, and SenescenceCardiovascular Health and Disease Prevention

Cited by 84

Cdkn1a transcript variant 2 is a marker of aging and cellular senescence.

López-Domínguez, J. A., Rodríguez-López, S., Ahumada-Castro, U., Desprez, P.-Y., Konovalenko, M., Laberge, R.-M., Cárdenas, C., Villalba, J. M., & Campisi, J. (2021) · Aging

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 83

Mitochondrial DNA damage induces apoptosis in senescent cells

Laberge, R.-M., Adler, D., DeMaria, M., Mechtouf, N., Teachenor, R., Cardin, G. B., Desprez, P.-Y., Campisi, J., & Rodier, F. (2013) · Cell Death & Disease

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 82

Pleiotropic age-dependent effects of mitochondrial dysfunction on epidermal stem cells

Velarde, M. C., Demaria, M., Melov, S., & Campisi, J. (2015) · Proceedings of the National Academy of Sciences

Mitochondrial Function and PathologyTelomeres, Telomerase, and Senescence

Cited by 77

Elimination of senescent osteoclast progenitors has no effect on the age‐associated loss of bone mass in mice

Kim, H., Chang, J., Iyer, S., Han, L., Campisi, J., Manolagas, S. C., Zhou, D., & Almeida, M. (2019) · Aging Cell

Bone Metabolism and DiseasesTelomeres, Telomerase, and Senescence

Cited by 74

Kinetics of G1 transit following brief starvation for serum factors.

Campisi, J., Morreo, G., & Pardee, A. B. (1984) · Experimental Cell Research

Cancer-related Molecular PathwaysProtein Kinase Regulation and GTPase Signaling

Cited by 71

The power of proteomics to monitor senescence-associated secretory phenotypes and beyond: toward clinical applications

Basisty, N., Kale, A., Patel, S., Campisi, J., & Schilling, B. (2020) · Expert Review of Proteomics

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 69

p53: Pro-aging or pro-longevity?

de Keizer, P. L. J., Laberge, R.-M., & Campisi, J. (2010) · Aging

Cancer-related Molecular PathwaysGenetics, Aging, and Longevity in Model Organisms

Cited by 48

Quantitative Proteomic Analysis of the Senescence-Associated Secretory Phenotype by Data-Independent Acquisition.

Neri, F., Basisty, N., Desprez, P., Campisi, J., & Schilling, B. (2021) · Current Protocols

Telomeres, Telomerase, and SenescenceAdvanced Proteomics Techniques and Applications

Cited by 48

Responses of human embryonic stem cells and their differentiated progeny to ionizing radiation

Zou, Y., Zhang, N., Ellerby, L. M., Davalos, A. R., Zeng, X., Campisi, J., & Desprez, P.-Y. (2012) · Biochemical and Biophysical Research Communications

Effects of Radiation ExposurePluripotent Stem Cells Research

Cited by 41

Between Scylla and Charybdis: p53 links tumor suppression and aging

Campisi, J. (2002) · Mechanisms of Ageing and Development

Cancer-related Molecular PathwaysGenetics, Aging, and Longevity in Model Organisms

Cited by 33

A Fully-Automated Senescence Test (FAST) for the high-throughput quantification of senescence-associated markers

Neri, F., Takajjart, S. N., Lerner, C. A., Desprez, P.-Y., Schilling, B., Campisi, J., & Gerencser, A. A. (2024) · GeroScience

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 17

A natural variation-based screen in mouse cells reveals USF2 as a regulator of the DNA damage response and cellular senescence

Kang, T., Moore, E. C., Kopania, E. E. K., King, C. D., Schilling, B., Campisi, J., Good, J. M., & Brem, R. B. (2023) · G3: Genes, Genomes, Genetics

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 10

Senescent cell heterogeneity and responses to senolytic treatment are related to cell cycle status during senescence induction

Neri, F., Zheng, S., Watson, M. A., Desprez, P.-Y., Gerencser, A. A., Campisi, J., Wirtz, D., Wu, P.-H., & Schilling, B. (2025) · Aging

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 6

Inhibition of the metalloprotease ADAM19 as a novel senomorphic strategy to ameliorate gut permeability and senescence markers by modulating senescence-associated secretory phenotype (SASP)

Bar, S., Hilsabeck, T. A. U., Pattavina, B., López-Domínguez, J. A., Basisty, N., Bons, J., Watson, M., Schilling, B., Campisi, J., Kapahi, P., et al. (2025) · Aging

Telomeres, Telomerase, and SenescenceGenetics, Aging, and Longevity in Model Organisms

Cited by 3