Arnett, Heather A.
NuMedii, Inc., San Mateo, CA, USA.
10 papers on CoChatORCID 0000-0002-8442-3312
Most cited papers
- Verified
Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis
Adams, T. S., Schupp, J. C., Poli, S., Ayaub, E. A., Neumark, N., Ahangari, F., Chu, S. G., Raby, B. A., DeIuliis, G., Januszyk, M., et al. (2020) · Science Advances
Interstitial Lung Diseases and Idiopathic Pulmonary FibrosisSingle-cell and spatial transcriptomics
Cited by 1,420
- Verified
TNFα promotes proliferation of oligodendrocyte progenitors and remyelination
Arnett, H. A., Mason, J., Marino, M., Suzuki, K., Matsushima, G. K., & Ting, J. P.-Y. (2001) · Nature Neuroscience
Neurogenesis and neuroplasticity mechanismsMultiple Sclerosis Research Studies
Cited by 1,012
- Verified
bHLH Transcription Factor Olig1 Is Required to Repair Demyelinated Lesions in the CNS
Arnett, H. A., Fancy, S. P. J., Alberta, J. A., Zhao, C., Plant, S. R., Kaing, S., Raine, C. S., Rowitch, D. H., Franklin, R. J. M., & Stiles, C. D. (2004) · Science
Multiple Sclerosis Research StudiesNeurogenesis and neuroplasticity mechanisms
Cited by 416
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The oligodendrocyte-specific G protein–coupled receptor GPR17 is a cell-intrinsic timer of myelination
Chen, Y., Wu, H., Wang, S., Koito, H., Li, J., Ye, F., Hoang, J., Escobar, S. S., Gow, A., Arnett, H. A., et al. (2009) · Nature Neuroscience
Neurogenesis and neuroplasticity mechanismsMultiple Sclerosis Research Studies
Cited by 346
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Functional Genomic Analysis of Remyelination Reveals Importance of Inflammation in Oligodendrocyte Regeneration
Arnett, H. A., Wang, Y., Matsushima, G. K., Suzuki, K., & Ting, J. P.-Y. (2003) · The Journal of Neuroscience
Multiple Sclerosis Research StudiesNeurogenesis and neuroplasticity mechanisms
Cited by 190
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Mechanisms of Oncostatin M-Induced Pulmonary Inflammation and Fibrosis
Mozaffarian, A., Brewer, A. W., Trueblood, E. S., Luzina, I. G., Todd, N. W., Atamas, S. P., & Arnett, H. A. (2008) · The Journal of Immunology
Interstitial Lung Diseases and Idiopathic Pulmonary FibrosisSystemic Sclerosis and Related Diseases
Cited by 153
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Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitis
Weinger, J. G., Brosnan, C. F., Loudig, O., Goldberg, M. F., Macian, F., Arnett, H. A., Prieto, A. L., Tsiperson, V., & Shafit-Zagardo, B. (2011) · Journal of Neuroinflammation
Multiple Sclerosis Research StudiesPhagocytosis and Immune Regulation
Cited by 134
- Verified
The protective role of nitric oxide in a neurotoxicant-induced demyelinating model
Arnett, H. A., Hellendall, R. P., Matsushima, G. K., Suzuki, K., Laubach, V. E., Sherman, P., & Ting, J. P.-Y. (2002) · The Journal of Immunology
Multiple Sclerosis Research StudiesNeuroinflammation and Neurodegeneration Mechanisms
Cited by 71
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Astroglial‐derived lymphotoxin‐α exacerbates inflammation and demyelination, but not remyelination
Plant, S. R., Arnett, H. A., & Ting, J. P. (2005) · Glia
Multiple Sclerosis Research StudiesNeuroinflammation and Neurodegeneration Mechanisms
Cited by 60
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Lymphotoxin β Receptor (LtβR): Dual Roles in Demyelination and Remyelination and Successful Therapeutic Intervention Using LtβR–Ig Protein
Plant, S. R., Iocca, H. A., Wang, Y., Thrash, J. C., O'Connor, B. P., Arnett, H. A., Fu, Y.-X., Carson, M. J., & Ting, J. P.-Y. (2007) · The Journal of Neuroscience
Multiple Sclerosis Research StudiesNeuroinflammation and Neurodegeneration Mechanisms
Cited by 51
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