Segal, Benjamin M.

Circulating Ly-6C+ myeloid precursors migrate to the CNS and play a pathogenic role during autoimmune demyelinating disease

King, I. L., Dickendesher, T. L., & Segal, B. M. (2009) · Blood

Multiple Sclerosis Research StudiesNeuroinflammation and Neurodegeneration Mechanisms

Cited by 444

Ageing and multiple sclerosis

Graves, J. S., Krysko, K. M., Hua, L. H., Absinta, M., Franklin, R. J. M., & Segal, B. M. (2023) · The Lancet Neurology

Multiple Sclerosis Research StudiesPsoriasis: Treatment and Pathogenesis

Cited by 260

An emerging role for eotaxins in neurodegenerative disease

Huber, A. K., Giles, D. A., Segal, B. M., & Irani, D. N. (2018) · Clinical Immunology

Neuroinflammation and Neurodegeneration MechanismsChemokine receptors and signaling

Cited by 133

CNS-resident classical DCs play a critical role in CNS autoimmune disease

Giles, D. A., Duncker, P. C., Wilkinson, N. M., Washnock-Schmid, J. M., & Segal, B. M. (2018) · Journal of Clinical Investigation

Multiple Sclerosis Research StudiesImmunotherapy and Immune Responses

Cited by 99

A self-limiting, TREM2-dependent anabolic program confers microglial resilience in Alzheimer’s disease

Lin, D., Atkinson, J. R., Wu, W., Chen, M., Jayasekara, S., Segal, B. M., Ma, A., & Gao, J. (2026) · bioRxiv (Cold Spring Harbor Laboratory)

Neuroinflammation and Neurodegeneration MechanismsAlzheimer's disease research and treatments

Cited by 0