Rawls, John F.

The severity of nonalcoholic fatty liver disease is associated with gut dysbiosis and shift in the metabolic function of the gut microbiota

Boursier, J., Mueller, O., Barret, M., Machado, M., Fizanne, L., Araujo‐Perez, F., Guy, C. D., Seed, P. C., Rawls, J. F., David, L. A., et al. (2016) · Hepatology

Gut microbiota and healthLiver Disease Diagnosis and Treatment

Cited by 1,483

The Idea of Public Reason Revisited

Rawls, J. (1997) · The University of Chicago Law Review

Political Philosophy and EthicsComparative constitutional jurisprudence studies

Cited by 1,371

Molecular characterization of mucosal adherent bacteria and associations with colorectal adenomas

Shen, X. J., Rawls, J. F., Randall, T. A., Burcall, L., Mpande, C., Jenkins, N., Jovov, B., Abdo, Z., Sandler, R. S., & Keku, T. O. (2010) · Gut Microbes

Gut microbiota and healthColorectal Cancer Screening and Detection

Cited by 418

Mutational analysis of endothelin receptor b1 (rose) during neural crest and pigment pattern development in the zebrafish Danio rerio

Parichy, D. M., Mellgren, E. M., Rawls, J. F., Lopes, S. S., Kelsh, R. N., & Johnson, S. L. (2000) · Developmental Biology

melanin and skin pigmentationDevelopmental Biology and Gene Regulation

Cited by 230

How the zebrafish gets its stripes

Rawls, J. F., Mellgren, E. M., & Johnson, S. L. (2001) · Developmental Biology

melanin and skin pigmentationZebrafish Biomedical Research Applications

Cited by 165

Zebrafish kit mutation reveals primary and secondary regulation of melanocyte development during fin stripe regeneration

Rawls, J. F., & Johnson, S. L. (2000) · Development

melanin and skin pigmentationZebrafish Biomedical Research Applications

Cited by 106

Got worms? Perinatal exposure to helminths prevents persistent immune sensitization and cognitive dysfunction induced by early-life infection

Williamson, L. L., McKenney, E. A., Holzknecht, Z. E., Belliveau, C., Rawls, J. F., Poulton, S., Parker, W., & Bilbo, S. D. (2016) · Brain, Behavior, and Immunity

Parasites and Host InteractionsNeuroinflammation and Neurodegeneration Mechanisms

Cited by 84