The DOI matches its Crossref record.
Excess Weight Risk Factor for Alcoholic Liver Disease
Abstract
Though the hepatotoxicity of ethanol has been established, only 8% to 20% of chronic alcoholics develop cirrhosis. The aim of this study was to assess whether being overweight is a risk factor for alcoholic liver disease. One thousand six hundred four alcoholic patients were studied. According to the liver biopsies, 194 patients had a normal liver; 402 had steatosis without fibrosis; 281 presented with fibrosis, with or without steatosis; 119 presented with acute alcoholic hepatitis (AAH) without cirrhosis; 232 indicated cirrhosis without AAH; and 179 presented with cirrhosis with AAH. One hundred ninety-seven patients had clinically obvious cirrhosis. In the study, five variables were studied as risk factors: age, sex, daily consumption of alcohol during the previous 5 years, the total duration of alcohol abuse, and tendency to be overweight (body mass index [BMI] > or = 25 in women and > or = 27 in men). The BMI was calculated according to the minimum weight over the 10 previous years. In the first stepwise logistic regression analysis, age, being overweight for at least 10 years, being of the female sex, and the total duration of alcohol abuse were independently correlated with the presence of cirrhosis. In the second analysis, female sex being overweight were the two independent risk factors of AAH. In the third analysis, being overweight for at least 10 years was the only independent risk factor of steatosis. These results show that the presence of excess weight for at least 10 years is a risk factor for cirrhosis, AAH, and steatosis. Our results suggest that there is a possible potential for the metabolic effects of ethanol ingestion caused by excess weight in patients with alcoholic liver disease.
Overview
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
In the citing paper
“This corroborates older fi ndings of BMI as an independent risk factor for fi brosis in individuals with ALD.”
From EASL-EASD-EASO Clinical Practice Guidelines on the management of metabolic dysfunction-associated steatotic liver disease (MASLD). (2024) · cites this paper as [85]
Citations
Cited by
Sorted by citations
Items 401–500 of 669
Day, C. (2006) · Medicine · doi:10.1053/j.mpmed.2006.10.003 · Cited by 13
Adams, L. A., Kemp, W. W., Muller, K., Powell, E. E., Roberts, S. K., Bertot, L. C., Best, S., Deed, G., Emery, J. D., HOCKING, S. L., et al. (2025) · The Medical Journal of Australia · doi:10.5694/mja2.70008 · Cited by 12
Ali, H. (2022) · Annals of Gastroenterology · doi:10.20524/aog.2022.0718 · Cited by 12
“While the exact incidence remains unknown, a prevalence of 20% has been reported previously in patients with a history of alcoholism [20].”
BackgroundLi, F., Chen, J., Liu, Y., Gu, Z., Jiang, M., Zhang, L., Chen, S., Deng, Z., McClain, C. J., & Feng, W. (2021) · Cells · doi:10.3390/cells10123333 · Cited by 12
“A combination of binge alcohol intake and a high fat-containing diet has been shown to synergistically promote the development and progression of alcoholrelated liver diseases in patients [7,44] and animal models [45–50].”
BackgroundInterleukin 10 promoter haplotype is associated with alcoholic liver cirrhosis in Taiwanese patients
Yang, A., Wen, L., Yang, C., Wang, S., Chen, C., & Bair, M. (2014) · The Kaohsiung Journal of Medical Sciences · doi:10.1016/j.kjms.2014.02.016 · Cited by 12
“Factors such as female gender, obesity, and drinking patterns have been well documented [7,8].”
BackgroundNolin, T. (2003) · Clinical Pharmacology & Therapeutics · doi:10.1016/j.clpt.2003.09.003 · Cited by 12
“Indeed, obesity is a risk factor for alcohol-induced hepatotoxicity”
BackgroundModular effects of estradiol on ethanol-induced apoptosis in human intestinal epithelial cells
Asai, K., Buurman, W. A., Reutelingsperger, C. P. M., et al. (2005) · Scandinavian Journal of Gastroenterology · doi:10.1080/00365520510011605 · Cited by 12
Hard clinical outcomes in patients with NAFLD
Kumar, R. K. (2013) · Hepatology International · doi:10.1007/s12072-013-9455-y · Cited by 12
“In subjects with heavy alcohol consumption, excess body weight markedly increases the risk of acute alcoholic hepatitis and cirrhosis [57, 58].”
BackgroundCots, M. V., Ndugga, N., & Bataller, R. A. (2019) · Wiley eBooks · doi:10.1002/9781119211419.ch34 · Cited by 13
Is There a Diagnostic Miracle on the Horizon? Emerging Biomarkers in MASLD
Iwaszko-Sochal, K., Kasztelan‐Szczerbińska, B., & Cichoż‐Lach, H. (2025) · Journal of Clinical Medicine · doi:10.3390/jcm14176148 · Cited by 13
Lifestyle Interventions Beyond Diet and Exercise for Patients With Nonalcoholic Fatty Liver Disease.
Esteban, J. P., & Dinani, A. M. (2020) · PubMed Central · Cited by 10
Genetic and Environmental Susceptibility to Alcoholic Hepatitis
Morgan, M. Y., Sharma, M., & Atkinson, S. R. (2021) · Clinics in Liver Disease · doi:10.1016/j.cld.2021.04.001 · Cited by 11
“Excess weight is a risk factor for the development of alcohol-related liver disease per se and has also been shown to be an independent risk factor for the development of steatohepatitis on liver biopsy.(43) Thus, people drinking excessively who have been overweight for at least 10 years, defined by a BMI of 25 or gre…”
BackgroundInfluentialXiao, Y., Gao, C., Wu, J., Li, J., Wang, L., You, Y., Peng, T., Zhang, K., Cao, M., & Hong, J. (2022) · Frontiers in Pharmacology · doi:10.3389/fphar.2022.995523 · Cited by 10
“Excess alcohol consumption and obesity promote the development of steatohepatitis, cirrhosis, and hepatocellular carcinoma (Hart et al., 2010; Naveau et al., 1997; Ruhl and Everhart, 2005; Diehl, 2004).”
BackgroundSong, Q., Chen, Y., Ding, Q., Griffiths, A., Liu, L., Park, J., Liew, C. W., Nieto, N., Li, S., Dou, X., et al. (2023) · Hepatology Communications · doi:10.1097/hc9.0000000000000059 · Cited by 10
Alomari, M., Rashid, M. U., Chadalavada, P. N., Ragheb, J. G., Zafar, H., Suarez, Z. K., Khazaaleh, S., Gonzalez, A. J., & Castro, F. (2023) · World Journal of Hepatology · doi:10.4254/wjh.v15.i4.477 · Cited by 11
Predictive Factors for Pure Steatosis in Alcoholic Patients
Naveau, S., Thaury, J., Barri‐Ova, N., et al. (2009) · Alcoholism Clinical and Experimental Research · doi:10.1111/j.1530-0277.2009.00932.x · Cited by 10
“We have reported previously that obesity is an independent risk factor of steatosis in alcoholic patients (Naveau et al., 1997).”
BackgroundInfluentialSelf-citationSuzuki, M., Kon, K., Ikejima, K., Arai, K., Uchiyama, A., Aoyama, T., Yamashina, S., Ueno, T., & Watanabe, S. (2019) · Alcoholism Clinical and Experimental Research · doi:10.1111/acer.13982 · Cited by 10
“Metabolic syndrome increases the risk of liver injury as well as morbidity and mortality related to liver injury due to chronic alcohol consumption (Almeda-Valdes et al., 2016; Hellerbrand, 2010; Naveau et al., 1997; Raff et al., 2015; Tsukamoto, 2007).”
BackgroundTakahashi, A., Ohira, T., Uemura, M. Y., Hosoya, M., Yasumura, S., Hashimoto, S., Ohira, H., Sakai, A., Ohtsuru, A., Satoh, H., et al. (2017) · Scientific Reports · doi:10.1038/s41598-017-00776-7 · Cited by 10
The Relationship of Overweight and Obesity to High Mortality Rates from Liver Cirrhosis in Mexico
Méndez‐Sánchez, N., Sánchez-Castillo, C. P., Villa, A. R., et al. (2004) · Annals of Hepatology · doi:10.1016/s1665-2681(19)32111-8 · Cited by 10
“Naveau et al.(21) showed that the presence of excess weight for at least 10 years is a risk factor for cirrhosis, acute alcohol hepatitis, and steatosis.”
BackgroundGlobal Burden of Alcohol‐Associated Liver Disease Across 204 Countries, 1990–2021: GBD 2021 Findings
Wang, Y., Han, X., Zhang, X., Yuan, X., Wei, Q., Sang, L.-X., Chang, B., & Quan-sheng, L. (2025) · Liver International · doi:10.1111/liv.70283 · Cited by 11
“The research results show that excess body weight for at least 10 years is a risk factor for liver cirrhosis, acute alcoholic hepatitis, and steatosis [38].”
Daswani, R., Kumar, A., Anikhindi, S. A., et al. (2018) · Indian Journal of Gastroenterology · doi:10.1007/s12664-018-0842-0 · Cited by 9
Clinical Practical Guidelines: Management of Alcoholic Liver Disease
Klyarytskaya, I. L., & Стилиди, Е. И. (2014) · Cited by 9
Emerging Biomarkers in Alcohol-associated Hepatitis
Im, G. Y. (2022) · Journal of Clinical and Experimental Hepatology · doi:10.1016/j.jceh.2022.07.246 · Cited by 9
Non-alcoholic steatohepatitis: physiopathological, clinical and therapeutic implications
Pérez-Aguilar, F., Benlloch, S., Berenguer, M., Beltrán, B., & Berenguer, J. (2004) · Revista Española de Enfermedades Digestivas · doi:10.4321/s1130-01082004000900006 · Cited by 9
Lin, C., & Wen, T. (2012) · BMC Public Health · doi:10.1186/1471-2458-12-889 · Cited by 9
“Although it could be argued that other underlying factors, such as obesity or other chronic diseases may influence the relationship between drinking and alcohol-related mortality [33,34], those factors may be not associated with alcohol taxation.”
BackgroundBang, H.-A., Kwon, Y.-H., Lee, M.-J., & Lee, W. (2014) · Journal of Clinical Medicine Research · doi:10.14740/jocmr1975e · Cited by 9
“However, in a practice, most patients with alcoholic hepatitis in ALD drink more than 100 g/day (which corresponds to 6 - 7 drinks per day where one drink contains 13 - 15 g of alcohol), with 150 200 g per day being common [18-20].”
BackgroundBody mass index and risk of liver cirrhosis in middle aged UK women: Prospective study
Naveau, S. (2010) · Gastroentérologie Clinique et Biologique · doi:10.1016/j.gcb.2010.05.003 · Cited by 9
Self-citationLi, W., Xia, Y., Yang, J., Sanyal, A. J., Shah, V. H., Chalasani, N., & Yu, Q. (2024) · Frontiers in Immunology · doi:10.3389/fimmu.2024.1377236 · Cited by 8
“Overweight, de fi ned as BMI ≥ 25 in women and ≥ 27 in men, is a recognized risk factor for more severe histological liver damage and the progression of alcoholic liver disease (82, 83).”
Dai, X., Feng, S., & Li, T. X. (2025) · Pharmacological Research · doi:10.1016/j.phrs.2025.107720 · Cited by 9
Synergistic Effect of Alcohol Consumption and Body Mass on Serum Concentrations of Cytokeratin‐18
González‐Quintela, A., Tomé, S., Fernández‐Merino, C., Rey, J., Meijide, L. M., & Gudé, F. (2011) · Alcoholism Clinical and Experimental Research · doi:10.1111/j.1530-0277.2011.01570.x · Cited by 8
“Finally, obesity is a risk factor for ALD (Naveau et al., 1997; Raynard et al., 2002).”
BackgroundAlcoholic liver disease and its relationship with metabolic syndrome
Ishii, H., Horie, Y., Yamagishi, Y., et al. (2010) · Cited by 8
Hepatoprotective Effect of Trehalose: Insight into Its Mechanisms of Action
Forouzanfar, F., Guest, P. C., Jamialahmadi, T., & Sahebkar, A. (2021) · Advances in experimental medicine and biology · doi:10.1007/978-3-030-73234-9_34 · Cited by 8
Alcohol-related liver disease. Clinical practice guidelines. Consensus document sponsored by AEEH
Bataller, R. A., Cabezas, J., Aller, R., Cots, M. V., Abad, J., Albillos, A., Altamirano, J., Arias, M., Bañares, R., Caballería, J., et al. (2019) · Gastroenterología y Hepatología (English Edition) · doi:10.1016/j.gastre.2019.09.003 · Cited by 8
Études Myrmécologiques En 1878 (Primière Partie) Avec L'Anatomie Du Gésier Des Fourmis.
Forel, A.-H. (2010) · INFM-OAR (INFN Catania) · doi:10.5281/zenodo.25567 · Cited by 8
Potts, J. R., Farahi, N., Howard, M. R., Taylor, M., Heard, S., Shankar, A., Alexander, G., Chilvers, E. R., Verma, S., & Peters, A. M. (2018) · Bioscience Reports · doi:10.1042/bsr20180466 · Cited by 8
Alcohol consumption as a cofactor for other liver diseases
Altamirano, J., & Michelena, J. (2013) · Clinical Liver Disease · doi:10.1002/cld.197 · Cited by 8
Naveau, S. (2006) · Gastroentérologie Clinique et Biologique · doi:10.1016/s0399-8320(06)73224-9 · Cited by 8
Self-citationTINIAKOS, D. G., Anstee, Q. M., Brunt, E. M., & Burt, A. D. (2023) · Elsevier eBooks · doi:10.1016/b978-0-7020-8228-3.00005-3 · Cited by 7
MetALD: Genetic Factors and Clinical Outcomes
Israelsen, M. E., Trépo, E., Krag, A. A., & Stender, S. (2025) · Seminars in Liver Disease · doi:10.1055/a-2630-0952 · Cited by 7
“79 – 81 Such interaction is associated with increased risk of developing decompensated cirrhosis and HCC.”
Corticosteroids Versus Pentoxifylline for Severe Alcoholic Hepatitis
Njei, B. M., Do, A., McCarty, T. R., & Fortune, B. E. (2016) · Journal of Clinical Gastroenterology · doi:10.1097/mcg.0000000000000585 · Cited by 7
“PTX 59 — 48 (9) 68/32 26* — 65 (26) 18.”
Cho, W., Park, S. Y., Oh, H., El‐Aty, A. M. A., Hacımüftüoğlu, A., Kim, D. S., Jung, T. W., & Jeong, J. H. (2023) · Journal of Medicinal Food · doi:10.1089/jmf.2022.k.0093 · Cited by 7
Insulin resistance is associated with esophageal varices in alcoholic liver disease patients
Degré, D., Gustot, T., Gerkens, A., et al. (2016) · European Journal of Gastroenterology & Hepatology · doi:10.1097/meg.0000000000000627 · Cited by 7
Nutritional Assessment and Management of the Malnourished Patient
Mason, J. B. (2010) · Elsevier eBooks · doi:10.1016/b978-1-4160-6189-2.00004-4 · Cited by 7
Toxic AGEs (TAGE) Theory in the Pathogenesis of NAFLD and ALD
Takeuchi, M. (2015) · International Journal of Diabetes and Clinical Research · doi:10.23937/2377-3634/1410036 · Cited by 8
“The risks of developing and accelerating the progression of ALD are also increased by the intake of alcoholic beverages [9-11].”
BackgroundBhadoria, A. S., Kedarisetty, C. K., Bihari, C., Kumar, G., Jindal, A., Bhardwaj, A., Shasthry, V., Benjamin, J., Kumar, M., & Sarin, S. K. (2018) · Liver International · doi:10.1111/liv.13958 · Cited by 7
Nonalcoholic Fatty Liver Disease
Argo, C. K., Henry, Z. H., & Caldwell, S. H. (2017) · Wiley eBooks · doi:10.1002/9781119251316.ch32 · Cited by 7
Nonalcoholic fatty liver disease. New nomenclature. Review
Манжалій, Е. (2023) · Modern Gastroenterology · doi:10.30978/mg-2023-4-91 · Cited by 10
Patel, S. A., Kim, R. G., Shui, A. M., Magee, C., Lu, M., Chen, J., Tana, M. M.-S., Huang, C., & Khalili, M. (2023) · Gastro Hep Advances · doi:10.1016/j.gastha.2023.09.012 · Cited by 6
“3 Moreover, obese patients with a history of heavy alcohol use have a higher propensity for developing steatosis and cirrhosis compared to their normal-weight counterparts.”
Bergheim, I., Eagon, P. K., Dooley, S., & Breitkopf‐Heinlein, K. (2010) · Annals of the New York Academy of Sciences · doi:10.1111/j.1749-6632.2010.05833.x · Cited by 6
Risk Communication for Viral Hepatitis Management Among Migrants
Adesina, E., Oyero, O. S., Okorie, N., Ben-Enukora, C., & Adeyeye, B. (2021) · IGI Global eBooks · doi:10.4018/978-1-6684-2414-8.ch002 · Cited by 6
Kitajima, T., Moonka, D., Yeddula, S., et al. (2020) · Clinical Transplantation · doi:10.1111/ctr.13837 · Cited by 6
Stéatohépatite métabolique : 30 ans de recherches qui ont changé la NASH
Ratziu, V., & Poynard, T. (2009) · Gastroentérologie Clinique et Biologique · doi:10.1016/j.gcb.2009.05.001 · Cited by 6
Severe Alcoholic Hepatitis Effectively Treated with Vitamin E as an Add-on to Corticosteroids
Miyashima, Y., Shibata, M., Honma, Y., et al. (2017) · Internal Medicine · doi:10.2169/internalmedicine.8767-16 · Cited by 6
“It is characterized by the acute onset of a fever, jaundice, hepatomegaly, leukophilia and liver failure in heavy drinkers of alcohol (2).”
BackgroundHagström, H., Höijer, J., Andréasson, A., Bottai, M., Johansson, K., Ludvigsson, J. F., & Stephansson, O. (2019) · Alimentary Pharmacology & Therapeutics · doi:10.1111/apt.15162 · Cited by 6
Alcohol, Diet and Drug Use Preceding Alcoholic Hepatitis
Parker, R. A., & Neuberger, J. (2018) · Digestive Diseases · doi:10.1159/000487392 · Cited by 6
Nutrition, Nutritional Habits, Obesity, and Prevalence of Chronic Diseases in Workers
Koleva, M. (1999) · Reviews on Environmental Health · doi:10.1515/reveh.1999.14.1.21 · Cited by 6
Non‐Alcoholic Fatty Liver Disease
Day, C. P. (2010) · doi:10.1002/9781444314403.ch29 · Cited by 6
Nonalcoholic Fatty Liver Disease
Caldwell, S. H., Argo, C. K., & Al‐Osaimi, A. M. S. (2011) · Wiley eBooks · doi:10.1002/9781119950509.ch32 · Cited by 6
Herms, Q., Gratacós‐Ginès, J., & Pose, E. (2025) · Gastroenterology report · doi:10.1093/gastro/goaf088 · Cited by 5
Therapeutic perspectives on PDE4B inhibition in adipose tissue dysfunction and chronic liver injury
Staller, D. W., Bennett, R. G., & Mahato, R. I. (2024) · Expert Opinion on Therapeutic Targets · doi:10.1080/14728222.2024.2369590 · Cited by 6
“Naveau et al. conducted a study of one thousand and sixty-four biopsies from alcoholic patients in 1997, and determined that being obese for a decade or longer was a significant risk factor for the development of alcohol-associated cirrhosis [8].”
Current status of alcoholic liver cirrhosis and factors involved in the progression
Horie, Y., Ishii, H., Yamagishi, Y., et al. (2009) · Kanzo · doi:10.2957/kanzo.50.507 · Cited by 5
Impact of estrogen monotherapy on survival in women with stage III-IV non-small cell lung cancer
Heilbroner, S. P., Xanthopoulos, E. P., Buono, D. L., et al. (2018) · Lung Cancer · doi:10.1016/j.lungcan.2018.12.021 · Cited by 5
Risk Communication for Viral Hepatitis Management Among Migrants
Adesina, E., Oyero, O. S., Okorie, N., Ben-Enukora, C., & Adeyeye, B. (2019) · Advances in media, entertainment and the arts (AMEA) book series · doi:10.4018/978-1-7998-0210-5.ch014 · Cited by 5
Enfermedad hepática inducida por el alcohol
Caballería, J., Altamirano, J., Michelena, J., & Parés, A. R. (2012) · Medicine - Programa de Formación Médica Continuada Acreditado · doi:10.1016/s0304-5412(12)70352-9 · Cited by 5
Immunization against Intestinal Bacterial Endotoxin Prevents Alcoholic Fatty Liver in Rats.
NISHIMURA, G., Nakahara, K., Misawa, N., et al. (2001) · Journal of Veterinary Medical Science · doi:10.1292/jvms.63.275 · Cited by 5
Daniels, S. J., Leeming, D. J., Detlefsen, S., Bruun, M. F., Hjuler, S. T., Henriksen, K., Hein, P., Krag, A. A., Karsdal, M. A., Nielsen, M. J., et al. (2020) · American Journal of Physiology-Gastrointestinal and Liver Physiology · doi:10.1152/ajpgi.00066.2019 · Cited by 7
Nonalcoholic Fatty Liver Disease
Lackner, C. (2018) · Elsevier eBooks · doi:10.1016/b978-0-323-42873-6.00012-3 · Cited by 7
Current and Future Biomarkers in Alcoholic Hepatitis
Rutledge, S. M., & Im, G. Y. (2021) · Clinics in Liver Disease · doi:10.1016/j.cld.2021.03.010 · Cited by 5
Norden‐Krichmar, T. M., Rotroff, D. M., Schwantes-An, T.-H. L., Bataller, R. A., Goldman, D. A., Nagy, L. E., & Liangpunsakul, S. (2023) · Hepatology · doi:10.1097/hep.0000000000000617 · Cited by 5
Jerome, R. N., Zahn, L. A., Abner, J. J., Joly, M. M., Shirey-Rice, J. K., Wallis, R. S., Bernard, G. R., & Pulley, J. M. (2024) · Translational Gastroenterology and Hepatology · doi:10.21037/tgh-23-40 · Cited by 6
“Its exact incidence is unknown, but some have estimated that its incidence in alcoholics may be up to 20% (59).”
Mathurin, P. (2009) · Gastroentérologie Clinique et Biologique · doi:10.1016/j.gcb.2009.06.004 · Cited by 5
“[16] Naveau S, Giraud V, Borotto E, Aubert A, Capron F, Chaput JC.”
BackgroundInfluentialStewart, S. F., & Day, C. P. (2006) · Elsevier eBooks · doi:10.1016/b978-1-4160-3258-8.50034-6 · Cited by 5
Dasarathy, S., & McCullough, A. J. (2011) · doi:10.1002/9781119950509.ch26 · Cited by 10
Singh, K. R., Muktesh, G., Gunjan, D., Kochhar, R. K., Singh, V., Das, A., Siddappa, P. K., & Singh, K. (2019) · JGH Open · doi:10.1002/jgh3.12165 · Cited by 4
Marot, A., Henrion, J., Knebel, J., Trépo, E., Moreno, C., & Deltenre, P. (2023) · Hepatology Communications · doi:10.1097/hc9.0000000000000229 · Cited by 4
Diagnosis and Management of Early Stages of ALD
Gratacós‐Ginès, J., Alvarado‐Tápias, E., Aguado, D. M., López‐Pelayo, H., Bataller, R. A., & Pose, E. (2025) · Seminars in Liver Disease · doi:10.1055/a-2541-2892 · Cited by 4
“33 Other environmental factors include tobacco use, 34 certain diets, 35 and metabolic comorbidities, 36,37 the latter one being discussed in detail in a separate section.”
Ye, R.-Q., Chen, Y., Ma, C., Cheng, X., Guo, W., & Li, S. (2025) · Biomedicine & Pharmacotherapy · doi:10.1016/j.biopha.2025.118191 · Cited by 4
Strong, M., Pearson, T., MacNab, Y. C., & Maheswaran, R. (2012) · Spatial and Spatio-temporal Epidemiology · doi:10.1016/j.sste.2012.04.007 · Cited by 4
Natural history and factors influencing the course of alcohol-related liver disease
Kendrick, S. F. W., & Day, C. (2013) · Clinical Liver Disease · doi:10.1002/cld.145 · Cited by 4
“High-fat/low-carbohydrate diets have been shown to associate with more advanced ALD,(14) and all stages of alcohol-related liver injury are more common in patients with obesity.(15) As with other fibrotic liver diseases, epidemiological evidence suggests that the risk of cirrhosis is increased by smoking and is reduce…”
BackgroundManaging excessive alcohol consumption at a population level: The earlier the better
Louvet, A., & Krag, A. A. (2018) · Journal of Hepatology · doi:10.1016/j.jhep.2017.12.015 · Cited by 4
Chalin, A., Turlin, B., Ousmen, A., Michalak, S., Mueller, J. H., Mueller, S., Legros, L., Bardou‐Jacquet, E., Viel, J., Samson, M., et al. (2023) · Alimentary Pharmacology & Therapeutics · doi:10.1111/apt.17515 · Cited by 4
Epidemiology and Risk Factors for Alcoholic Liver Disease
Lazo, M., & Mitchell, M. C. (2015) · Springer eBooks · doi:10.1007/978-3-319-20538-0_1 · Cited by 6
Number of hospital contacts with alcohol problems predicts later risk of alcoholic liver cirrhosis
Askgaard, G., Tolstrup, J. S., Kjær, M. S., & Leon, D. A. (2018) · Scandinavian Journal of Public Health · doi:10.1177/1403494818763436 · Cited by 4
Of liver, whisky and plants: a requiem for colchicine in alcoholic cirrhosis?
Lonardo, A., & Loria, P. (2002) · European Journal of Gastroenterology & Hepatology · doi:10.1097/00042737-200204000-00003 · Cited by 4
Clinical Features, Disease Modifiers, and Natural History of Alcoholic Liver Disease
Marsano, L. S., Vatsalya, V., Hassan, A., & McClain, C. J. (2015) · Springer eBooks · doi:10.1007/978-3-319-20538-0_8 · Cited by 5
Spahr, L., & Hadengue, A. (2007) · doi:10.1002/9780470691861.ch12d · Cited by 4
Epidemiological Aspects of Alcoholic Liver Disease
Caballería, J. (2007) · Wiley eBooks · doi:10.1002/9780470691861.ch12a · Cited by 4
Cell Biology of NASH: Fibrosis and Cell Proliferation
Leclercq, I. A., & Horsmans, Y. (2004) · Wiley eBooks · doi:10.1002/9780470987438.ch12 · Cited by 4
Chronic alcohol and liver health: The gut-liver axis as a therapeutic target
Nair, D. M., Vajravelu, L. K., Vimala, P. B., Lathakumari, R. H., Panneerselvam, V. P., & Thulukanam, J. (2025) · Gastroenterology & Endoscopy · doi:10.1016/j.gande.2025.12.002 · Cited by 3
Enfermedad hepática por infiltración grasa no alcohólica. La nueva pandemia del milenio
Santos, L., Hernández, G., Varón, A., Beltrán, Ó., Botero, R. C., & Mejía, G. (2010) · Redalyc (Universidad Autónoma del Estado de México) · Cited by 3
Koteish, A. A., & Diehl, A. M. (2001) · Current Treatment Options in Gastroenterology · doi:10.1007/s11938-001-0020-8 · Cited by 3
“Obesity appears to be a comorbidity factor because the prevalence of both early stage ALD and cirrhosis is increased among obese patients compared with nonobese patients with alcohol dependency [ 5 ,14,15].”
BackgroundHepatite alcoólica aguda - Artigo de revisão
Matos, L. C., Batista, P., Monteiro, N., et al. (2013) · GE Portuguese Journal of Gastroenterology · doi:10.1016/j.jpg.2013.03.001 · Cited by 3
Henry, Z. H., Argo, C. K., & Caldwell, S. H. (2018) · Springer eBooks · doi:10.1007/978-3-319-94355-8_18 · Cited by 3
Habib, S., Murakami, T. T., Takyar, V. K., et al. (2023) · Gastroenterology Research · doi:10.14740/gr1556 · Cited by 3
“Most patients with AH have a history of alcohol use, usually more than 100 g/day for two or more decades [1, 2].”
BackgroundDefining recompensation of alcohol‐related liver disease: A step beyond decompensated cirrhosis
Gratacós‐Ginès, J., & Pose, E. (2023) · Liver International · doi:10.1111/liv.15690 · Cited by 3
Alcohol and Nutrition as Risk Factors for Chronic Liver Disease
Bellentani, S., Tiribelli, C., & Bedogni, G. (2012) · Humana Press eBooks · doi:10.1007/978-1-62703-047-2_38 · Cited by 3
O’Shea, R., Dasarathy, S., & McCullough, A. J. (2008) · Cambridge University Press eBooks · doi:10.1017/cbo9780511547416.007 · Cited by 3
The Liver in Type 2 Diabetes Mellitus
Diehl, A., & Choi, S. S. (2008) · Humana Press eBooks · doi:10.1007/978-1-60327-043-4_21 · Cited by 2
“For example, obesity is an independent risk factor for cirrhosis in alcoholic fatty liver disease ( 41 ).”
BackgroundGenetically obese (ob/ob) mice are resistant to the lethal effects of thioacetamide hepatotoxicity
Won, Y., Song, J., Lim, J.-H., et al. (2015) · Toxicology and Applied Pharmacology · doi:10.1016/j.taap.2015.12.001 · Cited by 3
Diagnostic Approaches and Clinical End Points of Treatment in Alcoholic Liver Disease
Cheong, J. Y., Stein, E. J., & Bataller, R. A. (2015) · Springer eBooks · doi:10.1007/978-3-319-20538-0_10 · Cited by 3
Metadata
- Authors
- Naveau, S., Giraud, V., Borotto, E., Aubert, A., Capron, F., & Chaput, J.
- Affiliations
- Hôpital Antoine-Béclère (FR)
- Venue
- Hepatology
- Published
- 1997-01
- Volume
- 25
- Issue
- 1
- Pages
- 108-111
- ISSN
- 0270-9139 (ISSN-L)
- OpenAlex
- W1987296974
- Cited by
- 666
- References
- 0
- Citations by year
- 2026: 4 · 2025: 19 · 2024: 27 · 2023: 40 · 2022: 19 · 2021: 21 · 2020: 27 · 2019: 22 · 2018: 41 · 2017: 29 · 2016: 36 · 2015: 45 · 2014: 37 · 2013: 21 · 2012: 27
- FWCI
- 6.31
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Hepatology (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Pathology and Forensic Medicine › Alcohol Consumption and Health Effects
- Health Sciences › Medicine › Hepatology › Liver Disease Diagnosis and Treatment
- Health Sciences › Medicine › Hepatology › Liver Disease and Transplantation
- Keywords
- alcoholic liver disease, overweight, body mass index, alcoholic cirrhosis, alcoholic hepatitis, steatosis, risk factors, sex differences, ethanol-induced hepatotoxicity
- MeSH terms
- Adult, Aged, Female, Humans, Liver Cirrhosis, Alcoholic, Male, Middle Aged, Obesity, Risk Factors, Multivariate Analysis
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
