The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension
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Cites
Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage
Nishikawa, T., Edelstein, D., Du, X. L., Yamagishi, S.-I., Matsumura, T., Kaneda, Y., Yorek, M. A., Beebe, D., Oates, P. J., Hammes, H.-P., et al. (2000) · Nature · doi:10.1038/35008121 · Cited by 4,277
Role of oxidative stress in diabetic complications: a new perspective on an old paradigm.
Baynes, J. W., & Thorpe, S. R. (1999) · Diabetes · doi:10.2337/diabetes.48.1.1 · Cited by 2,441
Diabetes mellitus and free radicals
Wolff, S. P. (1993) · British Medical Bulletin · doi:10.1093/oxfordjournals.bmb.a072637 · Cited by 660
Metabolic and Vascular Factors in the Pathogenesis of Diabetic Neuropathy
Cameron, N. E., & Cotter, M. A. (1997) · Diabetes · doi:10.2337/diab.46.2.s31 · Cited by 312 · cited 1×
“The significance of the diabetes-induced decrease in renal oxygen tension for renal function is unknown, but prevalence of tissue hypoxia has been postulated to be associated with both development of diabetic neuropathy and retinopathy [22, 23].”
BackgroundOn the mechanism by which vascular endothelial cells regulate their oxygen consumption
Clementi, E., Brown, G. C., Foxwell, N., & Moncada, S. (1999) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.96.4.1559 · Cited by 296
Increased renal metabolism in diabetes. Mechanism and functional implications
Körner, A., Eklöf, A.-C., Celsi, G., & Aperia, A. (1994) · Diabetes · doi:10.2337/diab.43.5.629 · Cited by 185
Cited by
Vallon, V., & Thomson, S. C. (2012) · Annual Review of Physiology · doi:10.1146/annurev-physiol-020911-153333 · Cited by 376
Intrarenal oxygenation: unique challenges and the biophysical basis of homeostasis
Evans, R. G., Gardiner, B. S., Smith, D. W., & O'Connor, P. M. (2008) · American Journal of Physiology-Renal Physiology · doi:10.1152/ajprenal.90230.2008 · Cited by 274
Hansell, P., Welch, W. J., Blantz, R. C., & Palm, F. (2013) · Clinical and Experimental Pharmacology and Physiology · doi:10.1111/1440-1681.12034 · Cited by 269
Self-citationPathophysiology of the Diabetic Kidney
Vallon, V., & Komers, R. (2011) · Comprehensive Physiology · doi:10.1002/cphy.c100049 · Cited by 259
Hesp, A. C., Schaub, J. A., Prasad, P. V., Vallon, V., Laverman, G. D., Bjornstad, P., & van Raalte, D. H. (2020) · Kidney International · doi:10.1016/j.kint.2020.02.041 · Cited by 202
Metadata
- Authors
- Palm, F., Cederberg, J., Hansell, P., Liss, P., & Carlsson, P.-O.
- Venue
- Diabetologia
- Published
- 2003-08-01
- Volume
- 46
- Issue
- 8
- Pages
- 1153-1160
- ISSN
- 0012-186X (ISSN-L)
- 1432-0428
- OpenAlex
- W2035637304
- Cited by
- 296
- References
- 6
- FWCI
- 5.64
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Palm, F., Cederberg, J., Hansell, P., Liss, P., & Carlsson, P.-O. (2003). Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension. Diabetologia. https://doi.org/10.1007/s00125-003-1155-z
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
