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Cited by 515

The DOI matches its Crossref record; 13 of the 13 references it cites were found in a registry or an index.

Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity

Open access
DeNardo, David G., Andreu, Pauline, Coussens, Lisa M.·2010·Cancer and Metastasis Reviews

Abstract

Tumor-associated myeloid cells have been implicated in regulating many of the "hallmarks of cancer" and thus fostering solid tumor development and metastasis. However, the same innate leukocytes also participate in anti-tumor immunity and restraint of malignant disease. While many factors regulate the propensity of myeloid cells to promote or repress cancerous growths, polarized adaptive immune responses by B and T lymphocytes have been identified as regulators of many aspects of myeloid cell biology by specifically regulating their functional capabilities. Here, we detail the diversity of heterogeneous B and T lymphocyte populations and their impacts on solid tumor development through their abilities to regulate myeloid cell function in solid tumors.

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Crossref · 10.1007/s10555-010-9223-6

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Metadata

Authors
DeNardo, D. G., Andreu, P., & Coussens, L. M.
Venue
Cancer and Metastasis Reviews
Published
2010-04-21
Volume
29
Issue
2
Pages
309-316
ISSN
  • 0167-7659 (ISSN-L)
  • 1573-7233
OpenAlex
W2171481006
Cited by
515
References
13
FWCI
2.15
Type
journal article
Language
en
Open access
hybrid

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CoChat. (2026). Citation check: Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity. https://app.cochat.ai/papers/10.1007/s10555-010-9223-6
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DeNardo, D. G., Andreu, P., & Coussens, L. M. (2010). Interactions between lymphocytes and myeloid cells regulate pro- versus anti-tumor immunity. Cancer and Metastasis Reviews. https://doi.org/10.1007/s10555-010-9223-6

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.