The DOI matches its Crossref record; 80 of the 80 references it cites were found in a registry or an index.
Rheumatoid arthritis and cardiovascular disease
Open accessAbstract
Background Rheumatic disease and heart disease share common underpinnings involving inflammation. The high levels of inflammation that characterize rheumatic diseases provide a “natural experiment” to help elucidate the mechanisms by which inflammation accelerates heart disease. Rheumatoid arthritis (RA) is the most common of the rheumatic diseases and has the best studied relationships with heart disease. Methods Review of current literature on heart disease and rheumatoid arthritis Results Patients with RA have an increased risk of developing heart disease that is not fully explained by traditional cardiovascular risk factors. Therapies used to treat RA may also affect the development of heart disease; by suppressing inflammation, they may also reduce the risk of heart disease. However, their other effects, as in the case of steroids, may increase heart disease risk. Conclusions Investigations of the innate and adaptive immune responses occurring in RA may delineate novel mechanisms in the pathogenesis of heart disease, and help identify novel therapeutic targets for the prevention and treatment of heart disease.
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Cites
The Pathogenesis of Rheumatoid Arthritis
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Abatacept for Rheumatoid Arthritis Refractory to Tumor Necrosis Factor α Inhibition
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Cardiovascular death in rheumatoid arthritis: A population‐based study
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Self-citationMaradit‐Kremers, H., Crowson, C. S., Nicola, P. J., Ballman, K. V., Roger, V. L., Jacobsen, S. J., & Gabriel, S. E. (2005) · Arthritis & Rheumatism · doi:10.1002/art.20853 · Cited by 927
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Obesity in autoimmune diseases: Not a passive bystander
Versini, M., Jeandel, P.-Y., Rosenthal, E., & Shoenfeld, Y. (2014) · Autoimmunity Reviews · doi:10.1016/j.autrev.2014.07.001 · Cited by 694
Stoffer, M. A., Schoels, M. M., Smolen, J. S., Aletaha, D., Breedveld, F. C., Burmester, G., Bykerk, V., Dougados, M., Emery, P., Haraoui, B., et al. (2016) · Annals of the Rheumatic Diseases · doi:10.1136/annrheumdis-2015-207526 · Cited by 451
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Severity of periodontitis and metabolic syndrome: is there an association?
Gomes‐Filho, I. S., das Mercês, M. C., de Santana Passos‐Soares, J., Seixas da Cruz, S., Teixeira Ladeia, A. M., Trindade, S. C., de Moraes Marcílio Cerqueira, E., Freitas Coelho, J. M., Marques Monteiro, F. M., Barreto, M. L., et al. (2016) · Journal of Periodontology · doi:10.1902/jop.2015.150367 · Cited by 52
Ikdahl, E., Wibetoe, G., Rollefstad, S., Salberg, A., Bergsmark, K., Kvien, T. K., Olsen, I. C., Soldal, D. M., Bakland, G., Lexberg, Å., et al. (2019) · International Journal of Cardiology · doi:10.1016/j.ijcard.2018.06.111 · Cited by 35
Metadata
- Authors
- Crowson, C. S., Liao, K. P., Davis, J. M., Solomon, D. H., Matteson, E. L., Knutson, K. L., Hlatky, M. A., & Gabriel, S. E.
- Venue
- American Heart Journal
- Publisher
- Elsevier BV
- Published
- 2013-10
- Volume
- 166
- Issue
- 4
- Pages
- 622-628.e1
- ISSN
- 0002-8703 (ISSN-L)
- OpenAlex
- W2103691265
- Cited by
- 351
- References
- 80
- FWCI
- 14.29
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
