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Cited by 986

The DOI matches its Crossref record; 220 of the 220 references it cites were found in a registry or an index.

The MAPK cascades: Signaling components, nuclear roles and mechanisms of nuclear translocation

Open access
Plotnikov, Alexander, Zehorai, Eldar, Procaccia, Shiri, Seger, Rony·2011·Biochimica et Biophysica Acta (BBA) - Molecular Cell Research

Abstract

The MAPK cascades are central signaling pathways that regulate a wide variety of stimulated cellular processes, including proliferation, differentiation, apoptosis and stress response. Therefore, dysregulation, or improper functioning of these cascades, is involved in the induction and progression of diseases such as cancer, diabetes, autoimmune diseases, and developmental abnormalities. Many of these physiological, and pathological functions are mediated by MAPK-dependent transcription of various regulatory genes. In order to induce transcription and the consequent functions, the signals transmitted via the cascades need to enter the nucleus, where they may modulate the activity of transcription factors and chromatin remodeling enzymes. In this review, we briefly cover the composition of the MAPK cascades, as well as their physiological and pathological functions. We describe, in more detail, many of the important nuclear activities of the MAPK cascades, and we elaborate on the mechanisms of ERK1/2 translocation into the nucleus, including the identification of their nuclear translocation sequence (NTS) binding to the shuttling protein importin7. Overall, the nuclear translocation of signaling components may emerge as an important regulatory layer in the induction of cellular processes, and therefore, may serve as targets for therapeutic intervention in signaling-related diseases such as cancer and diabetes. This article is part of a Special Issue entitled: Regulation of Signaling and Cellular Fate through Modulation of Nuclear Protein Import.

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Show all 220 references

Research works printed but not linked: 6

  1. Bendetz-Nezer, 2005, Full molecular page of MEK1, AfCS/Nature Signaling Gateway
  2. Bendetz-Nezer, 2005, Full molecular page of MEK2, AfCS/Nature Signaling Gateway
  3. Yao, 2001, Non-regulated and stimulated mechanisms cooperate in the nuclear accumulation of MEK1, Oncogene, 20, 7588, 10.1038/sj.onc.1204963 · doi:10.1038/sj.onc.1204963
  4. Richard, 2007, Mechanical stretching stimulates smooth muscle cell growth, nuclear protein import, and nuclear pore expression through mitogen-activated protein kinase activation, J. Biol. Chem., 282, 23081, 10.1074/jbc.M703602200 · doi:10.1074/jbc.m703602200
  5. Yao, 2010, Calcium regulation of EGF-induced ERK5 activation: role of Lad1-MEKK2 interaction, PLoS ONE, 5, 10.1371/journal.pone.0012627 · doi:10.1371/journal.pone.0012627
  6. Dingwall, 1988, The nucleoplasmin nuclear location sequence is larger and more complex than that of SV-40 large T antigen, J. Cell Biol., 107, 841, 10.1083/jcb.107.3.841 · doi:10.1083/jcb.107.3.841

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Metadata

Authors
Plotnikov, A., Zehorai, E., Procaccia, S., & Seger, R.
Venue
Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
Publisher
Elsevier BV
Published
2011-09
Volume
1813
Issue
9
Pages
1619-1633
ISSN
0167-4889 (ISSN-L)
OpenAlex
W1986753065
Cited by
986
References
220
FWCI
10.52
Type
journal article
Language
en
Open access
bronze
Licence
Funders
  • Israeli Science Foundation
  • MINERVA

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Plotnikov, A., Zehorai, E., Procaccia, S., & Seger, R. (2011). The MAPK cascades: Signaling components, nuclear roles and mechanisms of nuclear translocation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. https://doi.org/10.1016/j.bbamcr.2010.12.012

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