The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
Translational Repression of C. elegans p53 by GLD-1 Regulates DNA Damage-Induced Apoptosis
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Cites
Vogelstein, B., Lane, D., & Levine, A. J. (2000) · Nature · doi:10.1038/35042675 · Cited by 6,607
In Vivo Activation of the p53 Pathway by Small-Molecule Antagonists of MDM2
Vassilev, L. T., Vu, B. T., Graves, B., Carvajal, D., Podlaski, F., Filipovic, Z., Kong, N., Kammlott, U., Lukacs, C., Klein, C., et al. (2004) · Science · doi:10.1126/science.1092472 · Cited by 4,661
p53- and Drug-Induced Apoptotic Responses Mediated by BH3-Only Proteins Puma and Noxa
Villunger, A., Michalak, E. M., Coultas, L., Müllauer, F., Böck, G., Ausserlechner, M. J., Adams, J. M., & Strasser, A. (2003) · Science · doi:10.1126/science.1090072 · Cited by 1,327
Inhibiting the p53–MDM2 interaction: an important target for cancer therapy
Chène, P. (2003) · Nature Reviews Cancer · doi:10.1038/nrc991 · Cited by 765
The INK4A/ARF locus and its two gene products.
Sharpless, N. E., & DePinho, R. A. (1999) · Current Opinion in Genetics & Development · doi:10.1016/s0959-437x(99)80004-5 · Cited by 533 · cited 2×
“Although human cancers commonly contain mutations in the p53 gene itself, many of the remaining tumors have defects in upstream signaling components of the p53 pathway such as inactivation of the positive regulators ARF or CHK2 (Sharpless and DePinho, 1999; Bartek and Lukas, 2003), or overexpression of the negative re…”
Background
Cited by
p53 in the DNA–Damage–Repair Process
Williams, A. B., & Schumacher, B. (2016) · Cold Spring Harbor Perspectives in Medicine · doi:10.1101/cshperspect.a026070 · Cited by 835
Self-citationThe Keap1-Nrf2 System: A Mediator between Oxidative Stress and Aging
Yu, C., & Xiao, J.-H. (2021) · Oxidative Medicine and Cellular Longevity · doi:10.1155/2021/6635460 · Cited by 537
DNA damage responses and p53 in the aging process
Ou, H.-L., & Schumacher, B. (2018) · Blood · doi:10.1182/blood-2017-07-746396 · Cited by 377
Self-citationMutations That Increase the Life Span of C. elegans Inhibit Tumor Growth
Pinkston, J. M., Garigan, D., Hansen, M., & Kenyon, C. (2006) · Science · doi:10.1126/science.1121908 · Cited by 242
Chen, J., & Kastan, M. B. (2010) · Genes & Development · doi:10.1101/gad.1968910 · Cited by 127
Metadata
- Authors
- Schumacher, B., Hanazawa, M., Lee, M.-H., Nayak, S., Volkmann, K., Hofmann, R., Hengartner, M., Schedl, T., & Gartner, A.
- Venue
- Cell
- Publisher
- Elsevier BV
- Published
- 2005-02
- Volume
- 120
- Issue
- 3
- Pages
- 357-368
- ISSN
- 0092-8674 (ISSN-L)
- OpenAlex
- W2048663475
- Cited by
- 214
- References
- 5
- FWCI
- 5.37
- Type
- journal article
- Language
- en
- Open access
- bronze
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- CoChat. (2026). Citation check: Translational Repression of C. elegans p53 by GLD-1 Regulates DNA Damage-Induced Apoptosis. https://app.cochat.ai/papers/10.1016/j.cell.2004.12.009
- The paper
- Schumacher, B., Hanazawa, M., Lee, M.-H., Nayak, S., Volkmann, K., Hofmann, R., Hengartner, M., Schedl, T., & Gartner, A. (2005). Translational Repression of C. elegans p53 by GLD-1 Regulates DNA Damage-Induced Apoptosis. Cell. https://doi.org/10.1016/j.cell.2004.12.009
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.