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SorLA Controls Neurotrophic Activity by Sorting of GDNF and Its Receptors GFRα1 and RET

Glerup, Simon, Lume, Maria, Olsen, Ditte, Nyengaard, Jens R., Vaegter, Christian B., Gustafsen, Camilla, Christensen, Erik I., Kjolby, Mads, Hay-Schmidt, Anders, Bender, Dirk, Madsen, Peder, Saarma, Mart, Nykjaer, Anders, Petersen, Claus M.·2013·Cell Reports
DOI 10.1016/j.celrep.2012.12.011
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  • SORLA has been previously shown to regulate and traffic neuronal components, including sorting/trafficking the GDNF/GFRa1 complex, regulating calpain-dependent degradation of synapsin, and trafficking glutamate receptors such as GLUA1.

    “Indeed, SORLA has been previously regulate and traffic neuronal components, including sorting/trafficking the GDNF/GFRa1 complex (Glerup, Lume et al. 2013), regulating calpain-dependent degradation of synapsin (Hartl, Nebrich et al. 2016), and trafficking glutamate receptors such as GLUA1 (Mishra, Knupp et al. 2022), which could consequently affect neurodegenerative outcome.”

    From SORLA up-regulation suppresses pathological effects in aged tauopathy mouse brain (2026) · cites this paper as Glerup, Lume et al. 2013

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Research works printed but not linked: 51

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Metadata

Authors
Glerup, S., Lume, M., Olsen, D., Nyengaard, J. R., Vaegter, C. B., Gustafsen, C., Christensen, E. I., Kjolby, M., Hay-Schmidt, A., Bender, D., Madsen, P., Saarma, M., Nykjaer, A., & Petersen, C. M.
Venue
Cell Reports
Publisher
Elsevier BV
Published
2013-01
Volume
3
Issue
1
Pages
186-199
ISSN
2211-1247
DOI
10.1016/j.celrep.2012.12.011
Cited by
68
References
3
Language
en
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