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Cited by 995

The DOI matches its Crossref record; 9 of the 9 references it cites were found in a registry or an index.

Insulin Resistance, Hyperglycemia, and Atherosclerosis

Open access
Bornfeldt, Karin E., Tabas, Ira·2011·Cell Metabolism
DOI 10.1016/j.cmet.2011.07.015
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Abstract

Progress in preventing atherosclerotic coronary artery disease (CAD) has been stalled by the epidemic of type 2 diabetes. Further advances in this area demand a thorough understanding of how two major features of type 2 diabetes, insulin resistance and hyperglycemia, impact atherosclerosis. Insulin resistance is associated with systemic CAD risk factors, but increasing evidence suggests that defective insulin signaling in atherosclerotic lesional cells also plays an important role. The role of hyperglycemia in CAD associated with type 2 diabetes is less clear. Understanding the mechanisms whereby type 2 diabetes exacerbates CAD offers hope for new therapeutic strategies to prevent and treat atherosclerotic vascular disease.

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Registry

Crossref · 10.1016/j.cmet.2011.07.015

Cites

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Metadata

Authors
Bornfeldt, K. E., & Tabas, I.
Venue
Cell Metabolism
Publisher
Elsevier BV
Published
2011-11
Volume
14
Issue
5
Pages
575-585
ISSN
1550-4131 (ISSN-L)
DOI
10.1016/j.cmet.2011.07.015
OpenAlex
W2115872041
Cited by
995
References
9
FWCI
27.77
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Insulin Resistance, Hyperglycemia, and Atherosclerosis. https://app.cochat.ai/papers/10.1016/j.cmet.2011.07.015
The paper
Bornfeldt, K. E., & Tabas, I. (2011). Insulin Resistance, Hyperglycemia, and Atherosclerosis. Cell Metabolism. https://doi.org/10.1016/j.cmet.2011.07.015

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.