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Cited by 362

The DOI matches its Crossref record; 133 of the 133 references it cites were found in a registry or an index.

Mast cells in atopic dermatitis

Open access
Kawakami, Toshiaki, Ando, Tomoaki, Kimura, Miho, Wilson, Bridget S, Kawakami, Yuko·2009·Current Opinion in Immunology

Abstract

Summary of Recent Advances Mast cells play as the major effector cells in immediate hypersensitivity through activation via the high-affinity IgE receptor, FcεRI, although many other functions have recently been discovered for this cell type. Given the broad array of proinflammatory mediators secreted from FcεRI-activated mast cells, as well as sensitization to allergens, IgE elevation, and increased mast cells in a majority of atopic dermatitis patients, mast cells are believed to be involved in the pathogenesis of atopic dermatitis. Numerous animal models have been used to study this epidemic disease. Here we review the recent progress to synthesize our current understanding of this disease and potential mechanisms for a mast cell's role in the disease.

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Crossref · 10.1016/j.coi.2009.09.006

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Metadata

Authors
Kawakami, T., Ando, T., Kimura, M., Wilson, B. S., & Kawakami, Y.
Venue
Current Opinion in Immunology
Publisher
Elsevier BV
Published
2009-12
Volume
21
Issue
6
Pages
666-678
ISSN
0952-7915 (ISSN-L)
OpenAlex
W2039603982
Cited by
362
References
133
FWCI
0.61
Type
journal article
Language
en
Open access
green

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CoChat. (2026). Citation check: Mast cells in atopic dermatitis. https://app.cochat.ai/papers/10.1016/j.coi.2009.09.006
The paper
Kawakami, T., Ando, T., Kimura, M., Wilson, B. S., & Kawakami, Y. (2009). Mast cells in atopic dermatitis. Current Opinion in Immunology. https://doi.org/10.1016/j.coi.2009.09.006

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.