The Hippo Signaling Pathway in Development and Disease

Zheng, Yonggang, Pan, Duojia·2019·Developmental Cell

DOI 10.1016/j.devcel.2019.06.003

Cited by (891)

Items 301–400 of 891

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Negative feedback couples Hippo pathway activation with Kibra degradation independent of Yorkie-mediated transcription
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The LKB1–TSSK1B axis controls YAP phosphorylation to regulate the Hippo–YAP pathway
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Keratinocyte integrin α3β1 induces expression of the macrophage stimulating factor, CSF-1, through a YAP/TEAD-dependent mechanism
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Hyaluronan Degradation Promotes Cancer via Hippo‐YAP Signaling: An Intervention Point for Cancer Therapy
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Modulation of Yorkie activity by alternative splicing is required for developmental stability
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miR‑497 inhibits proliferation and invasion in triple‑negative breast cancer cells via YAP1
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Prostaglandin E2 and its receptor EP2 trigger signaling that contributes to YAP‐mediated cell competition
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TEAD4 as a Prognostic Marker Promotes Cell Migration and Invasion of Urinary Bladder Cancer via EMT
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Hippo signaling regulates the nuclear behavior and DNA binding times of YAP and TEAD to control transcription
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Upregulated GPRC5A disrupting the Hippo pathway promotes the proliferation and migration of pancreatic cancer cells via the cAMP-CREB axis
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Nuclear receptor E75/NR1D2 promotes tumor malignant transformation by integrating Hippo and Notch pathways
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STK3 is a transcriptional target of YAP1 and a hub component in the crosstalk between Hippo and Wnt signaling pathways during gastric carcinogenesis
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Increasing kinase domain proximity promotes MST2 autophosphorylation during Hippo signaling
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Livin promotes tumor progression through YAP activation in ovarian cancer.
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Drug-Resistant Breast Cancer: Dwelling the Hippo Pathway to Manage the Treatment
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