Mitochondrial abnormalities in temporal lobe of autistic brain

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Items 101–194 of 194

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  9. A molecular framework for autistic experiences: Mitochondrial allostatic load as a mediator between autism and psychopathology
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  10. In vivo magnetic resonance spectroscopy in the brain of Cdkl5 null mice reveals a metabolic profile indicative of mitochondrial dysfunctions
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  15. Chronic Ambient Ozone Exposure Aggravates Autism-Like Symptoms in a Susceptible Mouse Model
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  16. Association of NGF and Mitochondrial Respiration with Autism Spectrum Disorder
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  22. Taurine as a potential therapeutic agent interacting with multiple signaling pathways implicated in autism spectrum disorder (ASD): An in-silico analysis
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  23. Carnosine supplementation does not affect serum concentrations of advanced glycation and precursors of lipoxidation end products in autism: a randomized controlled clinical trial
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  28. Multivariate Analysis of Metabolomic and Nutritional Profiles among Children with Autism Spectrum Disorder
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  30. Positive Association Between Plasma Levels of Advanced Glycation and Precursor of Lipoxidation end Products with Gastrointestinal Problems in Children with Autism
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  31. Increased Vulnerability to Oxidative Stress and Mitochondrial Dysfunction in Autism
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  32. Autophagy in Neurodevelopmental Disorders
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  41. Mitochondrial Dysfunction in Autism Spectrum Disorders
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    Ylind Lila, Chieh-En Jane Tseng, Emma G. Johnston, Camila Canales, Christopher J. McDougle, Jacob M. Hooker, Nicole R. Zürcher · 2025 · Brain Behavior and Immunity
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  53. The Ketogenic Diet Metabolite β-Hydroxybutyrate Promotes Mitochondrial Elongation via Deacetylation in HeLa Cells and Improves Autism-like Behavior in Zebrafish
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  54. The Role of Mitochondrial DNA Copy Number in Neurodevelopmental Disorders: A Bidirectional Two-Sample Mendelian Randomization Study
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  60. Emerging Clues and Altered Metabolic Findings in Autism: Breakthroughs and Prospects from Omics Studies
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  70. Alteration in hippocampal mitochondria ultrastructure and cholesterol accumulation linked to mitochondrial dysfunction in the valproic acid rat model of autism spectrum disorders
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