The DOI matches its Crossref record; 318 of the 323 references it cites were found in a registry or an index.
Pain in an era of armed conflicts: Prevention and treatment for warfighters and civilian casualties
Abstract
Chronic pain is a common squealae of military- and terror-related injuries. While its pathophysiology has not yet been fully elucidated, it may be potentially related to premorbid neuropsychobiological status, as well as to the type of injury and to the neural alterations that it may evoke. Accordingly, optimized approaches for wounded individuals should integrate primary, secondary and tertiary prevention in the form of thorough evaluation of risk factors along with specific interventions to contravene and mitigate the ensuing chronicity. Thus, Premorbid Events phase may encompass assessments of psychological and neurobiological vulnerability factors in conjunction with fostering preparedness and resilience in both military and civilian populations at risk. Injuries per se phase calls for immediate treatment of acute pain in the field by pharmacological agents that spare and even enhance coping and adaptive capabilities. The key objective of the Post Injury Events is to prevent and/or reverse maladaptive peripheral- and central neural system's processes that mediate transformation of acute to chronic pain and to incorporate timely interventions for concomitant mental health problems including post-traumatic stress disorder and addiction We suggest that the proposed continuum of care may avert more disability and suffering than the currently employed less integrated strategies. While the requirements of the armed forces present a pressing need for this integrated continuum and a framework in which it can be most readily implemented, this approach may be also instrumental for the care of civilian casualties.
Overview
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
In the citing paper
“Patients with partners showing avoidant and anxious attachment styles are more likely to report increased pain and decreased well-being 103, 124–127.”
From Evaluating psychosocial contributions to chronic pain outcomes (2018) · cites this paper as [127]
Citations
Cites
Vos, T., Flaxman, A. D., Naghavi, M., Lozano, R., Michaud, C., Ezzati, M., Shibuya, K., Salomon, J. A., Abdalla, S., Aboyans, V., et al. (2012) · The Lancet · doi:10.1016/s0140-6736(12)61729-2 · Cited by 8,291
Physiology and neurobiology of stress and adaptation: central role of the brain
McEwen, B. S. (2007) · Physiological Reviews · doi:10.1152/physrev.00041.2006 · Cited by 5,429
Actual Causes of Death in the United States, 2000
Mokdad, A. H. (2004) · JAMA · doi:10.1001/jama.291.10.1238 · Cited by 5,211
Central sensitization: implications for the diagnosis and treatment of pain
Woolf, C. J. (2011) · Pain · doi:10.1016/j.pain.2010.09.030 · Cited by 4,771
Stress and the brain: from adaptation to disease
de Kloet, E. R., Joëls, M., & Holsboer, F. (2005) · Nature Reviews Neuroscience · doi:10.1038/nrn1683 · Cited by 4,621
Persistent postsurgical pain: risk factors and prevention
Kehlet, H., Jensen, T. S., & Woolf, C. J. (2006) · The Lancet · doi:10.1016/s0140-6736(06)68700-x · Cited by 4,086
Central sensitization: a generator of pain hypersensitivity by central neural plasticity
Latremoliere, A., & Woolf, C. J. (2009) · The Journal of Pain · doi:10.1016/j.jpain.2009.06.012 · Cited by 3,913
Millan, M. J. (2002) · Progress in Neurobiology · doi:10.1016/s0301-0082(02)00009-6 · Cited by 3,035
The self-medication hypothesis of addictive disorders: focus on heroin and cocaine dependence
(1985) · American Journal of Psychiatry · doi:10.1176/ajp.142.11.1259 · Cited by 2,538
Alexander, G. E., Crutcher, M. D., & DeLong, M. R. (1990) · PubMed · Cited by 2,422
Research works printed but not linked: 3
- Freemon, 2010, 95, 605
- Hierholzer, 2010, Pathological gambling in combat veterans, Fed. Pract., 27, 14
- Maihofner, 2010, Neuropathic pain and neuroplasticity in functional imaging studies, Schmerz, 24, 137
Cited by
Sorted by citations
Evaluating psychosocial contributions to chronic pain outcomes
Meints, S. M., & Edwards, R. R. (2018) · Progress in Neuro-Psychopharmacology and Biological Psychiatry · doi:10.1016/j.pnpbp.2018.01.017 · Cited by 719
When pain gets stuck: the evolution of pain chronification and treatment resistance
Borsook, D., Youssef, A. M., Simons, L., Elman, I., & Eccleston, C. (2018) · Pain · doi:10.1097/j.pain.0000000000001401 · Cited by 281
Stensland, S. Ø., Zwart, J., Wentzel‐Larsen, T., & Dyb, G. A. (2017) · Neurology · doi:10.1212/wnl.0000000000004805 · Cited by 34
“Model 3 also included a measure of level of current posttraumatic psychological distress in survivors and psychological distress in matched controls.(21,23) All tests were 2 tailed, with a significance level of p ≤ 0.”
MethodBugge, I., Dyb, G. A., Stensland, S. Ø., Ekeberg, Ø., Wentzel‐Larsen, T., & Diseth, T. H. (2017) · Journal of Traumatic Stress · doi:10.1002/jts.22191 · Cited by 29
Stensland, S. Ø., Thoresen, S., Jensen, T. K., Wentzel‐Larsen, T., & Dyb, G. A. (2020) · Journal of Traumatic Stress · doi:10.1002/jts.22562 · Cited by 29
“A heightened risk of adverse somatic health outcomes, such as chronic pain (George et al., 2016; Noel et al., 2016; Suri et al., 2017), somatic symptom disorder (Andreski et al., 1998; Kizilhan & Noll-Hussong, 2018), and gastrointestinal (Gradus et al., 2017) or cardiometabolic disease (Edmondson &…”
Background
Metadata
- Authors
- George, E., Elman, I., Becerra, L., Berg, S., & Borsook, D.
- Affiliations
- Harvard University (US)
- Corresponding author
- Borsook, D.
- Venue
- Progress in Neurobiology
- Publisher
- Elsevier BV
- Published
- 2016-06
- Volume
- 141
- Pages
- 25-44
- ISSN
- 0301-0082 (ISSN-L)
- OpenAlex
- W2341035808
- Cited by
- 18
- References
- 323
- Citations by year
- 2025: 2 · 2024: 1 · 2022: 1 · 2021: 3 · 2020: 2 · 2019: 1 · 2018: 5 · 2017: 3
- FWCI
- 0.56
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Progress in Neurobiology (publishedVersion)
- PubMed (publishedVersion)
- Funders
- National Institute of Neurological Disorders and Stroke (K24NS064050)
- Topics
- Health Sciences › Medicine › Anesthesiology and Pain Medicine › Pain Management and Treatment
- Health Sciences › Medicine › Physiology (medical) › Pain Mechanisms and Treatments
- Health Sciences › Medicine › Clinical Neurology › Traumatic Brain Injury Research
- Keywords
- chronic pain, war injuries, military personnel, civilian casualties, pain prevention, pain management, resilience, post-traumatic stress disorder, addiction, acute pain management, central sensitization, peripheral sensitization, continuum of care
- MeSH terms
- Armed Conflicts, Brain, Humans, Military Personnel, Neural Pathways, Pain, Risk Factors, Stress, Psychological, Warfare, Wounds and Injuries
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
