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Characterization of the plasma membrane localization and orientation of HPV16 E5 for cell-cell fusion
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“High-risk HPV-16 E5α is an 83 amino acid protein, localized mainly in the membranes of the endoplasmic reticulum, Golgi, nuclear membrane and endosomes and, also, in the plasma membrane [28,29]”
From The Not-So-Good, the Bad and the Ugly: HPV E5, E6 and E7 Oncoproteins in the Orchestration of Carcinogenesis (2021) · cites this paper as [28]
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Tight control of gene expression in mammalian cells by tetracycline-responsive promoters.
Gossen, M., & Bujard, H. (1992) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.89.12.5547 · Cited by 4,943 · cited 1×
“These adenoviruses can only be expressed in cells that express the tetracycline transactivator (tTA), a chimeric protein that constitutively binds and activates the tetracycline promoters, except in the presence of tetracycline (Gossen and Bujard, 1992).”
BackgroundNarisawa‐Saito, M., & Kiyono, T. (2007) · Cancer Science · doi:10.1111/j.1349-7006.2007.00546.x · Cited by 528
Research works printed but not linked: 42
- Bouvard, 1994, The human papillomavirus type 16 E5 gene cooperates with the E7 gene to stimulate proliferation of primary cells and increases viral gene expression, Virology, 203, 73, 10.1006/viro.1994.1456 · DOI · Paper page
- Bravo, 2005, The E5 protein of the human papillomavirus type 16 modulates composition and dynamics of membrane lipids in keratinocytes, Arch. Virol., 150, 231, 10.1007/s00705-004-0420-x · DOI
- Chen, 2005, Unveiling the mechanisms of cell–cell fusion, Science, 308, 369, 10.1126/science.1104799 · DOI
- Clifford, 2003, Human papillomavirus types in invasive cervical cancer worldwide: a meta-analysis, Brit. J. Cancer, 88, 63, 10.1038/sj.bjc.6600688 · DOI
- Cogliano, 2005, Carcinogenicity of human papillomaviruses, Lancet Oncol., 6, 204, 10.1016/S1470-2045(05)70086-3 · DOI
- Conrad, 1994, The E5 protein of HPV-6, but not HPV-16, associates efficiently with cellular growth factor receptors, Virology, 200, 796, 10.1006/viro.1994.1244 · DOI
- Dinneen, 2004, Constitutive activation of rab5 results in a ligand independent redistribution of the EGFR and attenuates its ability to signal, Traffic, 5, 606, 10.1111/j.1398-9219.2004.00204.x · DOI
- Disbrow, 2003, Codon optimization of HPV-16 E5 gene enhances protein expression, Virology, 311, 105, 10.1016/S0042-6822(03)00129-6 · DOI · Paper page
- Disbrow, 2005, Endoplasmic reticulum-localized human papillomavirus type 16 E5 protein alters endosomal pH but not trans-Golgi pH, J. Virol., 79, 5839, 10.1128/JVI.79.9.5839-5846.2005 · DOI · Paper page
- Duelli, 2007, Cell-to-cell fusion as a link between viruses and cancer, Nat. Rev. Cancer, 7, 968, 10.1038/nrc2272 · DOI
- Duelli, 2007, A virus causes cancer by inducing massive chromosomal instability through cell fusion, Curr. Biol., 17, 1, 10.1016/j.cub.2007.01.049 · DOI
- Gieswein, 2003, Oligomerization of the E5 protein of human papillomavirus type 16 occurs through multiple hydrophobic regions, Virology, 313, 415, 10.1016/S0042-6822(03)00296-4 · DOI · Paper page
- Gillison, 2000, Evidence for a causal association between human papillomavirus and a subset of head and neck cancers, J. Natl. Cancer Inst., 92, 709, 10.1093/jnci/92.9.709 · DOI
- Gillison, 2008, Distinct risk factor profiles for human papillomavirus type 16-positive and human papillomavirus type 16-negative head and neck cancers, J. Natl. Cancer Inst., 100, 407, 10.1093/jnci/djn025 · DOI
- Hu, 2008, A non-invasive technique for quantifying and isolating fused cells, Cytotechnology, 58, 113, 10.1007/s10616-009-9186-z · DOI
- Hu, 2009, Human papillomavirus 16 E5 induces polyploidy by cell fusion, Virology, 384, 125, 10.1016/j.virol.2008.10.011 · DOI
- Jones, 1997, The human papillomavirus E7 oncoprotein can uncouple cellular differentiation and proliferation in human keratinocytes by abrogating p21 Cip1-mediated inhibition of cdk2, Genes Dev., 111, 2101, 10.1101/gad.11.16.2101 · DOI · Paper page
- Kabsch, 2002, The human papillomavirus type 16 E5 protein impairs TRAIL- and FasL-mediated apoptosis in HaCaT cells by different mechanisms, J. Virol., 76, 12162, 10.1128/JVI.76.23.12162-12172.2002 · DOI
- Leechanachai, 1992, The E5 gene from human papillomavirus type 16 is an oncogene which enhances growth factor-mediated signal transduction to the nucleus, Oncogene, 7, 19
- Leptak, 1991, Tumorigenic transformation of murine keratinocytes by the E5 genes of bovine papillomavirus type 1 and human papillomavirus type 16, J. Virol., 65, 7078, 10.1128/jvi.65.12.7078-7083.1991 · DOI · Paper page
- Martin, 2000, Common properties of fusion peptides from diverse systems, Biosci. Rep., 20, 483, 10.1023/A:1010454803579 · DOI
- McCance, 2005, Human papillomaviruses and cell signaling, Sci. STKE, 29, 29
- McMurray, 2001, Biology of papillomaviruses, J. Exp. Path., 82, 15, 10.1046/j.1365-2613.2001.00177.x · DOI
- Mittal, 1990, Sensitivity and specificity of various morphological features of cervical condylomas, Arch. Pathol. Lab. Med., 114, 1038
- Oelze, 1995, Human papillomavirus type 16 E5 protein affects cell–cell communication in an epithelial cell line, J. Virol., 69, 4489, 10.1128/jvi.69.7.4489-4494.1995 · DOI
- Oetke, 2000, Human Papillomavirus type 16 E5 protein localizes to the Golgi apparatus but does not grossly affect cellular glycosylation, Arch. Virol., 145, 2183, 10.1007/s007050070048 · DOI
- Pim, 1992, Human Papillomavirus type 16 E5 gene stimulates the transforming activity of the epidermal growth factor receptor, Oncogene, 7, 27
- Prasad, 1993, The bi-nucleated squamous cell: histologic spectrum and relationship to low-grade squamous intraepithelial lesions, Mod. Pathol., 6, 313
- Riley, 2003, Dissection of human papillomavirus E6 and E7 function in transgenic mouse models of cervical carcinogenesis, Cancer Res., 63
- Schwartz, 1985, Structure and transcription of human papillomavirus sequences in cervical carcinoma cells, Nature, 314, 111, 10.1038/314111a0 · DOI
- Stoler, 1992, Human papillomavirus type 16 and 18 gene expression in cervical neoplasias, Hum. Pathol., 23, 117, 10.1016/0046-8177(92)90232-R · DOI
- Stoppler, 1996, The E5 gene of HPV-16 enhances keratinocyte immortalization by full-length DNA, Virology, 223, 251, 10.1006/viro.1996.0475 · DOI · Paper page
- Straight, 1993, The E5 oncoprotein of human papillomavirus type 16 transforms fibroblasts and effects the downregulation of the epidermal growth factor receptor in keratinocytes, J. Virol., 67, 4521, 10.1128/jvi.67.8.4521-4532.1993 · DOI · Paper page
- Straight, 1995, The E5 oncoprotein of human papillomavirus type 16 inhibits the acidification of endosomes in human keratinocytes, J. Virol., 69, 3185, 10.1128/jvi.69.5.3185-3192.1995 · DOI
- Suprynowicz, 2008, HPV-16 E5 oncoprotein upregulates lipid raft components caveolin-1 and ganglioside GM1 at the plasma membrane of cervical cells, Oncogene, 27, 1071, 10.1038/sj.onc.1210725 · DOI
- Thomsen, 2000, The HPV 16 E5 oncogene inhibits endocytic trafficking, Oncogene, 19, 6023, 10.1038/sj.onc.1204010 · DOI
- Tomakidi, 2000, Modulation of the epidermal growth factor receptor by the human papillomavirus type 16 E5 protein in raft cultures of human keratinocytes, Eur. J. Cell Biol., 407, 10.1078/0171-9335-00060 · DOI · Paper page
- Valle, 1995, The human papillomavirus (HPV)-6 and HPV-16 E5 proteins co-operate with HPV-16 E7 in the transformation of primary rodent cells, J. Gen. Virol., 76, 1239, 10.1099/0022-1317-76-5-1239 · DOI · Paper page
- Werness, 1990, Association of human papillomavirus types 16 and 18 E6 proteins with p53, Science, 248, 76, 10.1126/science.2157286 · DOI · Paper page
- Williams, 2005, Requirement of epidermal growth factor receptor for hyperplasia induced by E5, a high-risk human papillomavirus oncogene, Cancer Res., 65, 6534, 10.1158/0008-5472.CAN-05-0083 · DOI · Paper page
- Zhang, 2002, E5 protein of human papillomavirus type 16 protects human foreskin keratinocytes from UV B-irradiation–induced apoptosis, J. Virol., 76, 220, 10.1128/JVI.76.1.220-231.2002 · DOI · Paper page
- Zhang, 2003, The E5 protein of human papillomavirus type 16 perturbs MHC class II antigen maturation in human foreskin keratinocytes treated with interferon-gamma, Virology, 310, 100, 10.1016/S0042-6822(03)00103-X · DOI · Paper page
Cited by
Basukala, O., & Banks, L. (2021) · Viruses · doi:10.3390/v13101892 · Cited by 137
Metadata
- Authors
- Hu, L., & Ceresa, B. P.
- Venue
- Virology
- Publisher
- Elsevier BV
- Published
- 2009-10
- Volume
- 393
- Issue
- 1
- Pages
- 135-143
- ISSN
- 0042-6822
- PMC
- PMC2753727
- Cited by
- 28
- References
- 2
- Language
- en
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