The DOI matches its Crossref record; 7 of the 7 references it cites were found in a registry or an index.
Endothelial inflammation in insulin resistance
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“The anti-inflammatory activities of PPARs are partially effectuated by inhibition of NF-κB. PPARs are able to attenuate NF-κB function either by interfering with the transcription activating capacity of the NF-κB complex or by regulating genes that suppress the activation of NF-κB. By physical interaction with components of the NF-κB complex, PPARα impairs binding of NF-κB to the DNA and”
From Antioxidant and Anti-Inflammatory Activities of Essential Oils: A Short Review (2010) · cites this paper as [135]
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Antioxidant and Anti-Inflammatory Activities of Essential Oils: A Short Review
Miguel, M. G. (2010) · Molecules · doi:10.3390/molecules15129252 · Cited by 933
“Secondly, PPARα activation results in increased expression of IκB, the inhibitory protein that prevents the transfer of NF-κB to the nucleus [134,135].”
BackgroundMagnesium and the Inflammatory Response: Potential Physiopathological Implications
Mazur, A., Maier, J. A. M., Rock, E., Gueux, E., Nowacki, W., & Rayssiguier, Y. (2007) · Archives of Biochemistry and Biophysics · doi:10.1016/j.abb.2006.03.031 · Cited by 474
Wannamethee, S. G., Lowe, G. D. O., Rumley, A., Bruckdorfer, K. R., & Whincup, P. H. (2006) · American Journal of Clinical Nutrition · doi:10.1093/ajcn.83.3.567 · Cited by 317
PPARs, Obesity, and Inflammation
Stienstra, R., Duval, C., Müller, M., & Kersten, S. (2007) · PPAR Research · doi:10.1155/2007/95974 · Cited by 287
D’Aiuto, F., Sabbah, W., Netuveli, G., Donos, N., Hingorani, A. D., Deanfield, J. E., & Tsakos, G. (2008) · The Journal of Clinical Endocrinology & Metabolism · doi:10.1210/jc.2007-2522 · Cited by 289
Metadata
- Authors
- SJOHOLM, A., & NYSTROM, T.
- Venue
- The Lancet
- Publisher
- Elsevier BV
- Published
- 2005-02-18
- Volume
- 365
- Issue
- 9459
- Pages
- 610-612
- ISSN
- 0099-5355 (ISSN-L)
- 0140-6736
- OpenAlex
- W2161477191
- Cited by
- 278
- References
- 0
- Citations by year
- 2026: 5 · 2025: 5 · 2024: 7 · 2023: 9 · 2022: 7 · 2021: 7 · 2019: 6 · 2018: 5 · 2017: 4 · 2016: 6 · 2015: 7 · 2014: 8 · 2013: 8 · 2012: 15
- FWCI
- 5.60
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- The Lancet (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Adipokines, Inflammation, and Metabolic Diseases
- Health Sciences › Medicine › Physiology (medical) › Nitric Oxide and Endothelin Effects
- Life Sciences › Immunology and Microbiology › Immunology › Biomarkers in Disease Mechanisms
- Keywords
- endothelial inflammation, insulin resistance, type 2 diabetes, C-reactive protein, endothelial progenitor cells, endothelial nitric oxide synthase, rosiglitazone, atherosclerosis, atherothrombosis, metabolic syndrome, obesity, cardiovascular risk
- MeSH terms
- Arteriosclerosis, C-Reactive Protein, Diabetes Mellitus, Type 2, Endothelium, Vascular, Humans, Inflammation, Insulin Resistance, Biomarkers, Inflammation Mediators, Metabolic Syndrome
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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