The DOI matches its Crossref record; 15 of the 15 references it cites were found in a registry or an index.
Non‐alcoholic steatohepatitis: another disease of affluence
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In the citing paper
“Steatosis is believed to sensitize the liver to metabolic injury, leading to inflammation, necrosis, and fibrosis.2,[5][6][7]”
From Leptin reverses nonalcoholic steatohepatitis in patients with severe lipodystrophy† (2005) · cites this paper as [2]
Citations
Cites
Steatohepatitis: A tale of two “hits”?
Day, C. P., & James, O. F. W. (1998) · Gastroenterology · doi:10.1016/s0016-5085(98)70599-2 · Cited by 4,285
Self-citationLudwig, J., Viggiano, T. R., McGill, D. B., & Ott, B. J. (1980) · Mayo Clinic Proceedings · doi:10.1016/s0025-6196(24)00530-5 · Cited by 2,903
Fatty liver hepatitis (steatohepatitis) and obesity: an autopsy study with analysis of risk factors.
Wanless, I. R., & Lentz, J. S. (1990) · Hepatology · doi:10.1002/hep.1840120505 · Cited by 1,191
Cryptogenic Cirrhosis: Clinical Characterization and Risk Factors for Underlying Disease
Caldwell, S. H., Oelsner, D. H., Iezzoni, J. C., Hespenheide, E. E., Battle, E. H., & Driscoll, C. J. (1999) · Hepatology · doi:10.1002/hep.510290347 · Cited by 1,138
Nonalcoholic steatohepatitis: An expanded clinical entity
Bacon, B. R., Farahvash, M. J., Janney, C. G., & Neuschwander‐Tetri, B. A. (1994) · Gastroenterology · doi:10.1016/0016-5085(94)90235-6 · Cited by 1,137
The natural history of nonalcoholic fatty liver: A follow-up study
Teli, M. R., James, O., Burt, A. D., Bennett, M. K., & Day, C. P. (1995) · Hepatology · doi:10.1002/hep.1840220616 · Cited by 913
Self-citationYang, S. Q., Lin, H. Z., Lane, M. D., Clemens, M. G., & Diehl, A. M. (1997) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.94.6.2557 · Cited by 818
Hepatic cytochrome P450 2E1 is increased in patients with nonalcoholic steatohepatitis
Weltman, M., Farrell, G. C., Hall, P. D. L. Μ., Ingelman‐Sundberg, M., & Liddle, C. (1998) · Hepatology · doi:10.1002/hep.510270121 · Cited by 613
Hepatic steatosis: Innocent bystander or guilty party?
Day, C. P., & James, O. (1998) · Hepatology · doi:10.1002/hep.510270601 · Cited by 395
Self-citationNon-alcoholic steatohepatitis (NASH): a disease of emerging identity and importance
James, O., & Day, C. P. (1998) · Journal of Hepatology · doi:10.1016/s0168-8278(98)80073-1 · Cited by 370
Self-citationArner, P. (1998) · The Lancet · doi:10.1016/s0140-6736(05)79052-8 · Cited by 182
RECURRENCE OF NONALCOHOLIC STEATOHEPATITIS FOLLOWING LIVER TRANSPLANTATION1
Kim, W. R., Poterucha, J. J., Porayko, M. K., Dickson, E. R., Steers, J. L., & Wiesner, R. H. (1996) · Transplantation · doi:10.1097/00007890-199612270-00021 · Cited by 167
Clinical Hepatology: Profile of An Urban, Hospital–Based Practic
Byron, D., & Minuk, G. Y. (1996) · Hepatology · doi:10.1002/hep.510240410 · Cited by 146
PROFILE OF AN URBAN HOSPITAL BASED PRACTICE
Byron, D., & Minuk, G. Y. (1996) · Cited by 25
Bass, N. M. (1999) · Hepatology · doi:10.1002/hep.510290247 · Cited by 13
Research works printed but not linked: 1
- Younossi, 1998, Patient characteristics predicting cirrhosis and death in non-alcoholic steatohepatitis, Hepatology, 28, 303A
Cited by
Sorted by citations
Donnelly, K. L., Smith, C. I., Schwarzenberg, S. J., Jessurun, J., Boldt, M. D., & Parks, E. J. (2005) · Journal of Clinical Investigation · doi:10.1172/jci23621 · Cited by 3,604
Triglyceride Metabolism in the Liver
Alves‐Bezerra, M., & Cohen, D. E. (2017) · Comprehensive physiology · doi:10.1002/cphy.c170012 · Cited by 1,324
Liver fibrosis in overweight patients
Ratziu, V., Giral, P., Charlotte, F., Bruckert, É., Thibault, V., Theodorou, I. D., Khalil, L., Turpin, G., Opolon, P., & Poynard, T. (2000) · Gastroenterology · doi:10.1016/s0016-5085(00)70364-7 · Cited by 969
Reddy, J. K., & Hashimoto, T. (2001) · Annual Review of Nutrition · doi:10.1146/annurev.nutr.21.1.193 · Cited by 991
Wigg, A. J., Roberts-Thomson, I. C., Dymock, R. B., McCarthy, P. J., Grose, R. H., & Cummins, A. G. (2001) · Gut · doi:10.1136/gut.48.2.206 · Cited by 836
Metadata
- Authors
- James, *., & Day, C.
- Affiliations
- Newcastle University (GB)
- ORCID
- Day, Christopher
- Venue
- The Lancet
- Publisher
- Elsevier BV
- Published
- 1999-05
- Volume
- 353
- Issue
- 9165
- Pages
- 1634-1636
- ISSN
- 0099-5355 (ISSN-L)
- 0140-6736
- OpenAlex
- W2075783257
- Cited by
- 271
- References
- 15
- Citations by year
- 2026: 1 · 2024: 1 · 2022: 2 · 2021: 6 · 2020: 3 · 2019: 2 · 2018: 7 · 2017: 7 · 2016: 7 · 2015: 6 · 2014: 8 · 2013: 17 · 2012: 9
- FWCI
- 17.97
- Type
- journal article
- Language
- en
- Open access
- green
- doi.org
- Locations
- The Lancet (publishedVersion)
- PubMed (publishedVersion)
- Newcastle University ePrints (Newcastle Univesity)
- Topics
- Health Sciences › Medicine › Hepatology › Liver Disease Diagnosis and Treatment
- Health Sciences › Medicine › Pathology and Forensic Medicine › Alcohol Consumption and Health Effects
- Health Sciences › Medicine › Hepatology › Liver Diseases and Immunity
- Keywords
- nonalcoholic steatohepatitis, liver dysfunction, alcohol consumption, prevalence, health screening
- MeSH terms
- Diabetes Mellitus, Type 2, Fatty Liver, Hepatitis, Humans, Liver, Obesity, Lipid Peroxidation
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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