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Non‐alcoholic steatohepatitis: another disease of affluence
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“Steatosis is believed to sensitize the liver to metabolic injury, leading to inflammation, necrosis, and fibrosis.2,[5][6][7]”
From Leptin reverses nonalcoholic steatohepatitis in patients with severe lipodystrophy† (2005) · cites this paper as [2]
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Metadata
- Authors
- James, *., & Day, C.
- Affiliations
- Newcastle University (GB)
- ORCID
- Day, Christopher
- Venue
- The Lancet
- Publisher
- Elsevier BV
- Published
- 1999-05
- Volume
- 353
- Issue
- 9165
- Pages
- 1634-1636
- ISSN
- 0099-5355 (ISSN-L)
- 0140-6736
- OpenAlex
- W2075783257
- Cited by
- 271
- References
- 15
- Citations by year
- 2026: 1 · 2024: 1 · 2022: 2 · 2021: 6 · 2020: 3 · 2019: 2 · 2018: 7 · 2017: 7 · 2016: 7 · 2015: 6 · 2014: 8 · 2013: 17 · 2012: 9
- FWCI
- 17.97
- Type
- journal article
- Language
- en
- Open access
- green
- doi.org
- Locations
- The Lancet (publishedVersion)
- PubMed (publishedVersion)
- Newcastle University ePrints (Newcastle Univesity)
- Topics
- Health Sciences › Medicine › Hepatology › Liver Disease Diagnosis and Treatment
- Health Sciences › Medicine › Pathology and Forensic Medicine › Alcohol Consumption and Health Effects
- Health Sciences › Medicine › Hepatology › Liver Diseases and Immunity
- Keywords
- nonalcoholic steatohepatitis, liver dysfunction, alcohol consumption, prevalence, health screening
- MeSH terms
- Diabetes Mellitus, Type 2, Fatty Liver, Hepatitis, Humans, Liver, Obesity, Lipid Peroxidation
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