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JNK is required for effector T-cell function but not for T-cell activation
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Citations
Cited by
Signal transduction by the JNK group of MAP kinases
Davis, R. J. (2000) · Cell · doi:10.1016/s0092-8674(00)00116-1 · Cited by 4,237
Self-citationPositive and negative selection of T cells
Starr, T. K., Jameson, S. C., & Hogquist, K. A. (2003) · Annual Review of Immunology · doi:10.1146/annurev.immunol.21.120601.141107 · Cited by 1,601
Mitogen-activated protein kinases in apoptosis regulation
Wada, T., & Penninger, J. M. (2004) · Oncogene · doi:10.1038/sj.onc.1207556 · Cited by 1,432
Phosphatase‐mediated crosstalk between MAPK signaling pathways in the regulation of cell survival
Junttila, M. R., Li, S., & Westermarck, J. (2008) · The FASEB Journal · doi:10.1096/fj.06-7859rev · Cited by 826
Targeting JNK for therapeutic benefit: from junk to gold?
Manning, A. M., & Davis, R. J. (2003) · Nature Reviews Drug Discovery · doi:10.1038/nrd1132 · Cited by 585
Self-citationMurine B7-H3 is a negative regulator of T cells
Prasad, D. V. R., Nguyen, T., Li, Z., Yang, Y., Duong, J., Wang, Y., & Dong, C. (2004) · The Journal of Immunology · doi:10.4049/jimmunol.173.4.2500 · Cited by 368
Alternative p38 activation pathway mediated by T cell receptor-proximal tyrosine kinases
Salvador, J. M., Mittelstadt, P. R., Guszczynski, T., Copeland, T. D., Yamaguchi, H., Appella, E., Fornace, A. J., & Ashwell, J. D. (2005) · Nature Immunology · doi:10.1038/ni1177 · Cited by 306
MAPK signaling in inflammation-associated cancer development
Huang, P., Han, J., & Hui, L. (2010) · Protein & Cell · doi:10.1007/s13238-010-0019-9 · Cited by 273
Activation of the E3 ubiquitin ligase Itch through a phosphorylation-induced conformational change
Gallagher, E., Gao, M., Liu, Y.-C., & Karin, M. (2006) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.0510664103 · Cited by 258
Requirement of the JIP1 scaffold protein for stress-induced JNK activation
Whitmarsh, A. J., Kuan, C.-Y., Kennedy, N. J., Kelkar, N., Haydar, T. F., Mordes, J. P., Appel, M., Rossini, A. A., Jones, S. N., Flavell, R. A., et al. (2001) · Genes & Development · doi:10.1101/gad.922801 · Cited by 238
Self-citationLu, B., Yu, H., Chow, C.-W., Li, B., Zheng, W.-P., Davis, R. J., & Flavell, R. A. (2001) · Immunity · doi:10.1016/s1074-7613(01)00141-8 · Cited by 134
Self-citationEmerging role of MAP kinase pathways as therapeutic targets in COPD
Mercer, B. A., & D'Armiento, J. M. (2006) · International Journal of COPD · doi:10.2147/copd.2006.1.2.137 · Cited by 118
c-Jun N-terminal kinase-dependent mechanisms in respiratory disease
Bennett, B. L. (2006) · European Respiratory Journal · doi:10.1183/09031936.06.00012106 · Cited by 63
Regulation of αβ/γδ T Cell Development by the Activator Protein 1 Transcription Factor c-Jun
Riera-Sans, L., & Behrens, A. (2007) · The Journal of Immunology · doi:10.4049/jimmunol.178.9.5690 · Cited by 47
Identification and functional analysis of specific MS risk miRNAs and their target genes
Luo, D., Wang, J., Zhang, X., Rang, X., Xu, C., & Fu, J. (2020) · Multiple Sclerosis and Related Disorders · doi:10.1016/j.msard.2020.102044 · Cited by 12
Metadata
- Authors
- Dong, C., Yang, D. D., Tournier, C., Whitmarsh, A. J., Xu, J., Davis, R. J., & Flavell, R. A.
- Venue
- Nature
- Published
- 2000-05
- Volume
- 405
- Issue
- 6782
- Pages
- 91-94
- ISSN
- 0028-0836 (ISSN-L)
- 1476-4687
- OpenAlex
- W2070330987
- Cited by
- 328
- References
- 0
- FWCI
- 13.16
- Type
- journal article
- Language
- en
- Open access
- closed
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
