The DOI matches its Crossref record; 4 of the 4 references it cites were found in a registry or an index.
HDAC inhibition promotes neuronal outgrowth and counteracts growth cone collapse through CBP/p300 and P/CAF-dependent p53 acetylation
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Citations
Cites
Stability and flexibility of epigenetic gene regulation in mammalian development
Reik, W. (2007) · Nature · doi:10.1038/nature05918 · Cited by 1,963 · cited 1×
“A permissive or relaxed chromatin conformation (euchromatin) correlates with a relative high level of acetylated histones and is a preferential chromatin state during early embryogenesis, nervous system development and synaptic plasticity.(4,5) The degree of acetylation is the result of a balance between histone acety…”
BackgroundHistone acetylation and transcriptional regulatory mechanisms
Struhl, K. (1998) · Genes & Development · doi:10.1101/gad.12.5.599 · Cited by 1,815 · cited 1×
“HATs modify core histone tails by acetylation of specific lysine residues, and induce a relaxed chromatin conformation, which along with acetylation of a set of TFs promotes DNA accessibility, promoter binding and gene expression.(8,9) In contrast, HDACs attenuate the transcription process at a particular site by deac…”
BackgroundHDAC2 negatively regulates memory formation and synaptic plasticity
Guan, J.-S., Haggarty, S. J., Giacometti, E., Dannenberg, J.-H., Joseph, N., Gao, J., Nieland, T. J. F., Zhou, Y., Wang, X., Mazitschek, R., et al. (2009) · Nature · doi:10.1038/nature07925 · Cited by 1,644
Acetylation and deacetylation of non-histone proteins
Glozak, M. A., Sengupta, N., Zhang, X., & Seto, E. (2005) · Gene · doi:10.1016/j.gene.2005.09.010 · Cited by 1,584
Cited by
Epigenetic regulation of gene expression in physiological and pathological brain processes
Gräff, J., Kim, D., Dobbin, M. M., & Tsai, L.-H. (2011) · Physiological Reviews · doi:10.1152/physrev.00012.2010 · Cited by 373
Shukla, S., & Tekwani, B. L. (2020) · Frontiers in Pharmacology · doi:10.3389/fphar.2020.00537 · Cited by 314
MicroRNA-138 and SIRT1 form a mutual negative feedback loop to regulate mammalian axon regeneration
Liu, C.-M., Wang, R.-Y., Saijilafu, Jiao, Z.-X., Zhang, B.-Y., & Zhou, F.-Q. (2013) · Genes & Development · doi:10.1101/gad.209619.112 · Cited by 148
Jia, Y., Li, L., Lin, Y.-H., Gopal, P., Shen, S., Zhou, K., Yu, X., Sharma, T., Zhang, Y., Siegwart, D. J., et al. (2022) · Cell Stem Cell · doi:10.1016/j.stem.2022.01.001 · Cited by 59
Chu, T., Zhou, H., Lu, L., Kong, X., Wang, T., Pan, B., & Feng, S. (2015) · Regenerative Medicine · doi:10.2217/rme.14.86 · Cited by 49
Metadata
- Authors
- Gaub, P., Tedeschi, A., Puttagunta, R., Nguyen, T., Schmandke, A., & Di Giovanni, S.
- Venue
- Cell Death & Differentiation
- Published
- 2010-01-22
- Volume
- 17
- Issue
- 9
- Pages
- 1392-1408
- ISSN
- 1350-9047 (ISSN-L)
- 1476-5403
- OpenAlex
- W1964392512
- Cited by
- 194
- References
- 4
- FWCI
- 5.03
- Type
- journal article
- Language
- en
- Open access
- green
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- CoChat. (2026). Citation check: HDAC inhibition promotes neuronal outgrowth and counteracts growth cone collapse through CBP/p300 and P/CAF-dependent p53 acetylation. https://app.cochat.ai/papers/10.1038/cdd.2009.216
- The paper
- Gaub, P., Tedeschi, A., Puttagunta, R., Nguyen, T., Schmandke, A., & Di Giovanni, S. (2010). HDAC inhibition promotes neuronal outgrowth and counteracts growth cone collapse through CBP/p300 and P/CAF-dependent p53 acetylation. Cell Death & Differentiation. https://doi.org/10.1038/cdd.2009.216
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
