The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
Immunopathogenesis of coronavirus infections: implications for SARS
Open accessAbstract
At the end of 2002, the first cases of severe acute respiratory syndrome (SARS) were reported, and in the following year, SARS resulted in considerable mortality and morbidity worldwide. SARS is caused by a novel species of coronavirus (SARS-CoV) and is the most severe coronavirus-mediated human disease that has been described so far. On the basis of similarities with other coronavirus infections, SARS might, in part, be immune mediated. As discussed in this Review, studies of animals that are infected with other coronaviruses indicate that excessive and sometimes dysregulated responses by macrophages and other pro-inflammatory cells might be particularly important in the pathogenesis of disease that is caused by infection with these viruses. It is hoped that lessons from such studies will help us to understand more about the pathogenesis of SARS in humans and to prevent or control outbreaks of SARS in the future.
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Cites
Angiotensin-converting enzyme 2 is a functional receptor for the SARS coronavirus
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The Acute Respiratory Distress Syndrome
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A crucial role of angiotensin converting enzyme 2 (ACE2) in SARS coronavirus–induced lung injury
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Angiotensin-converting enzyme 2 protects from severe acute lung failure
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CD4 and CD8 T Cells Have Redundant But Not Identical Roles in Virus-Induced Demyelination
Wu, G. F., Dandekar, A. A., Pewe, L., & Perlman, S. (2000) · The Journal of Immunology · doi:10.4049/jimmunol.165.4.2278 · Cited by 192
Self-citationEditorial Comment: The Host Response, Not the Virus, Causes the Symptoms of the Common Cold
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“Although it has not been proven for IBV-infected chickens, it is probable that this excessive production of mucus is mediated by several cytokines, such as IL-1β, IL-6 and CXCL8, which are secreted by infected epithelial cells in other respiratory-virus infections 94 .”
Background
Cited by
Single-cell landscape of bronchoalveolar immune cells in patients with COVID-19
Liao, M., Liu, Y., Yuan, J., Wen, Y., Xu, G., Zhao, J., Cheng, L., Li, J., Wang, X., Wang, F., et al. (2020) · Nature Medicine · doi:10.1038/s41591-020-0901-9 · Cited by 2,614
Coronaviruses post-SARS: update on replication and pathogenesis
Perlman, S., & Netland, J. (2009) · Nature Reviews Microbiology · doi:10.1038/nrmicro2147 · Cited by 1,764
Self-citationImmunology of COVID-19: Current State of the Science
Vabret, N., Britton, G. J., Gruber, C., Hegde, S., Kim, J., Kuksin, M., Levantovsky, R., Malle, L., Moreira, A., Park, M. D., et al. (2020) · Immunity · doi:10.1016/j.immuni.2020.05.002 · Cited by 1,623
Immune responses in COVID-19 and potential vaccines: Lessons learned from SARS and MERS epidemic
(2020) · Asian Pacific Journal of Allergy and Immunology · doi:10.12932/ap-200220-0772 · Cited by 1,537
Severe COVID-19 Is Marked by a Dysregulated Myeloid Cell Compartment
Schulte-Schrepping, J., Reusch, N., Paclik, D., Baßler, K., Schlickeiser, S., Zhang, B., Krämer, B., Krammer, T., Brumhard, S., Bonaguro, L., et al. (2020) · Cell · doi:10.1016/j.cell.2020.08.001 · Cited by 1,461
Metadata
- Authors
- Dandekar, A. A., & Perlman, S.
- Venue
- Nature Reviews Immunology
- Published
- 2005-12-01
- Volume
- 5
- Issue
- 12
- Pages
- 917-927
- ISSN
- 1474-1733 (ISSN-L)
- 1474-1741
- OpenAlex
- W2016029872
- Cited by
- 499
- References
- 6
- FWCI
- 1.90
- Type
- journal article
- Language
- en
- Open access
- bronze
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