The DOI matches its Crossref record; 180 of the 180 references it cites were found in a registry or an index.
Mitochondrial dynamics in adaptive and maladaptive cellular stress responses
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“During mitochondrial fission, cytosolic Drp1 is recruited to mitochondrial receptors (e.g., Fis1)3,4, allowing mitochondrial partitioning, turnover and reduction of network size under low energetic demand4,7.”
From Altered mitochondrial dynamics and function in APOE4-expressing astrocytes (2020) · cites this paper as [3]
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“Because mitochondrial dysfunction characterizes other cellular processes (Eisner et al., 2018), it is not a consistent biomarker of senescence.”
From Cellular Senescence: Defining a Path Forward (2019) · cites this paper as Eisner et al., 2018
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“Several proteins are required for mitochondrial fusion, including the mitofusins (Mfn1/Mfn2)3,4.”
From Altered mitochondrial dynamics and function in APOE4-expressing astrocytes (2020) · cites this paper as [3]
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“Mitochondrial dynamics include fusion, fission, and degradation events, linked to mitochondrial function3,4.”
From Altered mitochondrial dynamics and function in APOE4-expressing astrocytes (2020) · cites this paper as [3]
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“Mitochondrial fusion is mediated by mitofusins, which are regulated by transcription and protein modification, including ubiquitination3.”
From Altered mitochondrial dynamics and function in APOE4-expressing astrocytes (2020) · cites this paper as [3]
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Cites
Mitochondrial Fission, Fusion, and Stress
Youle, R. J., & van der Bliek, A. M. (2012) · Science · doi:10.1126/science.1219855 · Cited by 3,674
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Fission and selective fusion govern mitochondrial segregation and elimination by autophagy
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Holmström, K. M., & Finkel, T. (2014) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm3801 · Cited by 2,093
Mitochondria as sensors and regulators of calcium signalling
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During autophagy mitochondria elongate, are spared from degradation and sustain cell viability
Gomes, L. C., Benedetto, G. D., & Scorrano, L. (2011) · Nature Cell Biology · doi:10.1038/ncb2220 · Cited by 1,735
OPA1 Controls Apoptotic Cristae Remodeling Independently from Mitochondrial Fusion
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Mitochondrial Dynamics Controls T Cell Fate through Metabolic Programming
Buck, M. D., O’Sullivan, D., Geltink, R. I. K., Curtis, J. D., Chang, C., Sanin, D. E., Qiu, J., Kretz, O., Braas, D., van der Windt, G. J. W., et al. (2016) · Cell · doi:10.1016/j.cell.2016.05.035 · Cited by 1,491
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Differential effects of tropomyosin paralogs on mitochondrial dynamics in Saccharomyces cerevisiae
Cardozo, F., Paliwal, A., Bose, A., Adhikary, S., Laxman, S., D’Silva, P., & Palani, S. (2026) · Molecular Biology of the Cell · doi:10.1091/mbc.e25-09-0461
Shi, J., Zhao, Z., Wang, F., Zhao, Y., Zhao, Z., & Xu, D. (2026) · Theriogenology · doi:10.1016/j.theriogenology.2026.118054
Zeng, L., Huang, Y., Ke, J., Han, J., Xie, W., Ding, Z., & Zhou, F. (2026) · Frontiers in Pharmacology · doi:10.3389/fphar.2026.1851055
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Wang, Y., Dong, J., Xie, H., Cao, G., Pan, Z., Chen, N., Wong, W., He, Y., & Liu, X. (2026) · Spectrochimica Acta Part A Molecular and Biomolecular Spectroscopy · doi:10.1016/j.saa.2026.128643
Lv, Y., Kuang, L., Xiao, Z., Zhang, Y., Omindo, W. W., Zhan, X., Liu, X., Sun, Q., Wang, Y., Zhang, R., et al. (2026) · Cells · doi:10.3390/cells15171618
Circular RNAs at the crossroads of oxidative stress: novel targets for precision therapeutics
Singh, V., Sinha, R., Das, J., Islam, A., Sridhar, S. B., Panigrahy, U. P., Mantry, S., Shahin, H., Adhikary, K., Pal, R., et al. (2026) · Genome Instability & Disease · doi:10.1007/s42764-026-00190-2
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Zhang, J., Xie, H., Xu, H., Zhang, M., You, Y., Lv, J., Wang, L., & Xia, H. (2026) · Frontiers in Cell and Developmental Biology · doi:10.3389/fcell.2026.1937629
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Haider, I., Almazan, A. V. P., Gillespie, B., Yadav, S., Santhanakrishnan, R., Almazan, R. B. P., Biswas, K., Minassian, C., 鞠树林, & Zhong, Q. (2026) · Molecular Biology of the Cell · doi:10.1091/mbc.e25-11-0541
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Metadata
- Authors
- Eisner, V., Picard, M., & Hajnóczky, G.
- Affiliations
- Pontificia Universidad Católica de Chile (CL)
- Columbia University Irving Medical Center (US)
- Thomas Jefferson University (US)
- Corresponding author
- Hajnóczky, György
- Venue
- Nature Cell Biology
- Published
- 2018-06-27
- Volume
- 20
- Issue
- 7
- Pages
- 755-765
- ISSN
- 1465-7392 (ISSN-L)
- 1476-4679
- PMC
- PMC6716149
- OpenAlex
- W2809177340
- Cited by
- 622
- References
- 180
- Citations by year
- 2026: 63 · 2025: 78 · 2024: 83 · 2023: 86 · 2022: 81 · 2021: 93 · 2020: 87 · 2019: 44 · 2018: 5
- FWCI
- 5.62
- Type
- journal article
- Language
- en
- Open access
- green
- www.ncbi.nlm.nih.gov
- Licence
- www.springer.com/tdm (tdm)
- Locations
- Nature Cell Biology (publishedVersion)
- PubMed (publishedVersion)
- Europe PMC (PubMed Central) (acceptedVersion)
- PubMed Central (acceptedVersion)
- Americanae (AECID Library) (submittedVersion, cc-by-nc-nd)
- LA Referencia (Red Federada de Repositorios Institucionales de Publicaciones Científicas) (submittedVersion, cc-by-nc-nd)
- Funders
- National Institute on Alcohol Abuse and Alcoholism (U01AA021122)
- National Institute of Mental Health (R21MH113011)
- National Institute of General Medical Sciences (R35GM119793)
- National Institute of Diabetes and Digestive and Kidney Diseases (R01DK051526)
- National Institute of Environmental Health Sciences (R33ES025672)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Mitochondrial Function and Pathology
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Cell Biology › Autophagy in Disease and Therapy
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Cell death mechanisms and regulation
- Keywords
- mitochondrial dynamics, mitochondrial fusion, mitochondrial fission, mitochondrial motility, endoplasmic reticulum-mitochondria interaction, cellular stress response, adaptive stress response, maladaptive stress response, cell survival and death, energy production, mitochondrial signalling, mitochondrial disease
- MeSH terms
- Adaptation, Physiological, Animals, Cell Survival, Energy Metabolism, Humans, Mitochondria, Stress, Physiological, Signal Transduction, Cell Death, Mitochondrial Dynamics
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