Cited by 661Obesity, Physical Activity, DietRegulation of Appetite and ObesityObesity and Health Practices

The DOI matches its Crossref record; 110 of the 112 references it cites were found in a registry or an index.

Putative contributors to the secular increase in obesity: exploring the roads less traveled

Abstract

Objective To investigate plausible contributors to the obesity epidemic beyond the two most commonly suggested factors, reduced physical activity and food marketing practices. Design A narrative review of data and published materials that provide evidence of the role of additional putative factors in contributing to the increasing prevalence of obesity. Data Information was drawn from ecological and epidemiological studies of humans, animal studies and studies addressing physiological mechanisms, when available. Results For at least 10 putative additional explanations for the increased prevalence of obesity over the recent decades, we found supportive (although not conclusive) evidence that in many cases is as compelling as the evidence for more commonly discussed putative explanations. Conclusion Undue attention has been devoted to reduced physical activity and food marketing practices as postulated causes for increases in the prevalence of obesity, leading to neglect of other plausible mechanisms and well-intentioned, but potentially ill-founded proposals for reducing obesity rates.

Overview

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Crossref · 10.1038/sj.ijo.0803326

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110 of 110 references verified0 citation errors · 0 not checkable

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Citations

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2 of the 112 references couldn’t be matched to a known paper.

Research works printed but not linked: 2

  1. Aronne LJ . Drug-induced weight gain: non-CNS medications. In: Aronne LJ (ed). A Practical Guide to Drug-induced Weight Gain. McGraw-Hill: Minneapolis, MN, 2002. pp 77–91.
  2. Hamilton BE, Martin JA, Sutton PD . Births: preliminary data for 2003. Natl Vital Stat Rep 2004; 53: 1–17.

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Metadata

Authors
Keith, S. W., Redden, D. T., Katzmarzyk, P. T., Boggiano, M. M., Hanlon, E. C., Benca, R. M., Ruden, D., Pietrobelli, A., Barger, J. L., Fontaine, K. R., Wang, C., Aronne, L. J., Wright, S. M., Baskin, M., Dhurandhar, N. V., Lijoi, M. C., Grilo, C. M., DeLuca, M., Westfall, A. O., & Allison, D. B.
Affiliations
  • University of Alabama at Birmingham (US)
  • Queen's University (CA)
  • University of Wisconsin–Madison (US)
  • University of Verona (IT)
  • Johns Hopkins University (US)
  • University of Louisville (US)
  • Cornell University (US)
  • Pennington Biomedical Research Center (US)
  • Wayne State University (US)
  • Yale University (US)
Venue
International Journal of Obesity
Published
2006-06-27
Volume
30
Issue
11
Pages
1585-1594
ISSN
  • 0307-0565 (ISSN-L)
  • 1476-5497
OpenAlex
W2138122607
Cited by
659
References
112
Citations by year
2026: 8 · 2025: 5 · 2024: 8 · 2023: 20 · 2022: 23 · 2021: 30 · 2020: 29 · 2019: 30 · 2018: 25 · 2017: 32 · 2016: 34 · 2015: 34 · 2014: 43 · 2013: 43 · 2012: 50
FWCI
7.25
Type
journal article
Language
en
Open access
closed
Locations
  • International Journal of Obesity (publishedVersion)
  • PubMed (publishedVersion)
Funders
  • National Institutes of Health
  • National Institute on Alcohol Abuse and Alcoholism (R01AA012276)
  • National Cancer Institute (R01CA105349)
  • National Institute of Diabetes and Digestive and Kidney Diseases (P30DK056336)
  • National Institute of Environmental Health Sciences (R01ES012933)
Topics
  • Health Sciences › Medicine › Public Health, Environmental and Occupational Health › Obesity, Physical Activity, Diet
  • Life Sciences › Neuroscience › Endocrine and Autonomic Systems › Regulation of Appetite and Obesity
  • Health Sciences › Medicine › Public Health, Environmental and Occupational Health › Obesity and Health Practices
Keywords
obesity epidemic, physical activity, food marketing, obesity prevention, animal models of obesity, obesity risk factors
MeSH terms
Age Factors, Disease Outbreaks, Endocrine System, Female, Humans, Maternal Age, Obesity, Selection, Genetic, Sleep, Smoking, Temperature, Body Mass Index, Prevalence, Epigenesis, Genetic, Drug-Related Side Effects and Adverse Reactions

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