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Cited by 1,085

The DOI matches its Crossref record; 28 of the 28 references it cites were found in a registry or an index.

Intrauterine Growth Retardation and Postnatal Growth Failure Associated with Deletion of the Insulin-Like Growth Factor I Gene

Open access
Woods, Katie A., Camacho-Hübner, Cecilia, Savage, Martin O., Clark, Adrian J.L.·1996·New England Journal of Medicine

Abstract

Insulin-like growth factor I (IGF-I) mediates the majority of the growth-promoting effects of growth hormone (GH) after birth.1 In the prenatal period, GH does not appear to have a major influence on fetal growth, whereas IGF-I does. Infants with congenital GH deficiency and defects in the GH-receptor gene have only mild retardation of growth at birth,2–4 whereas transgenic mice with a homozygous defect of the IGF-I gene (IGF-I knockout mice) have profound embryonic and postnatal growth retardation.5–7 Although there is no direct evidence that IGF-I has a prominent role in human fetal growth, fetal tissues express IGF-I from . . .

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Crossref · 10.1056/nejm199610313351805

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Metadata

Authors
Woods, K. A., Camacho-Hübner, C., Savage, M. O., & Clark, A. J. L.
Venue
New England Journal of Medicine
Published
1996-10-31
Volume
335
Issue
18
Pages
1363-1367
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2320296534
Cited by
1,085
References
28
FWCI
26.99
Type
journal article
Language
en
Open access
bronze

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The paper
Woods, K. A., Camacho-Hübner, C., Savage, M. O., & Clark, A. J. L. (1996). Intrauterine Growth Retardation and Postnatal Growth Failure Associated with Deletion of the Insulin-Like Growth Factor I Gene. New England Journal of Medicine. https://doi.org/10.1056/nejm199610313351805

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.