The DOI matches its Crossref record; 64 of the 64 references it cites were found in a registry or an index.
Disruptive chemicals, senescence and immortality
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
In the citing paper
“However, there are also data indicating that acetaminophen can inhibit CDK4 and CDK2, thus imposing a cell cycle checkpoint at G1 and effectively blocking cellular proliferation [140].”
From Cell Immortality: In Vitro Effective Techniques to Achieve and Investigate Its Applications and Challenges (2024) · cites this paper as [140]
In the citing paper
“Physical carcinogens (such as ionizing radiation) are also powerful immortalization agents with different mechanisms and frequencies in rodent and human cells [140].”
From Cell Immortality: In Vitro Effective Techniques to Achieve and Investigate Its Applications and Challenges (2024) · cites this paper as [140]
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a
Serrano, M., Lin, A. W., McCurrach, M. E., Beach, D., & Lowe, S. W. (1997) · Cell · doi:10.1016/s0092-8674(00)81902-9 · Cited by 5,218
A DNA damage checkpoint response in telomere-initiated senescence
Fagagna, F. D. D., Reaper, P. M., Clay-Farrace, L., Fiegler, H., Carr, P., von Zglinicki, T., Saretzki, G., Carter, N. P., & Jackson, S. P. (2003) · Nature · doi:10.1038/nature02118 · Cited by 2,917
Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence
Narita, M., Nuñez, S., Heard, E., Narita, M., Lin, A. W., Hearn, S. A., Spector, D. L., Hannon, G. J., & Lowe, S. W. (2003) · Cell · doi:10.1016/s0092-8674(03)00401-x · Cited by 2,278
A complex secretory program orchestrated by the inflammasome controls paracrine senescence
Acosta, J. C., Banito, A., Wuestefeld, T., Georgilis, A., Janich, P., Morton, J. P., Athineos, D., Kang, T.-W., Lasitschka, F., Andrulis, M., et al. (2013) · Nature Cell Biology · doi:10.1038/ncb2784 · Cited by 2,278
Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis
Chen, Z., Trotman, L. C., Shaffer, D., Lin, H.-K., Dotan, Z. A., Niki, M., Koutcher, J. A., Scher, H. I., Ludwig, T., Gerald, W., et al. (2005) · Nature · doi:10.1038/nature03918 · Cited by 1,993
Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication
Di Micco, R., Fumagalli, M., Cicalese, A., Piccinin, S., Gasparini, P., Luise, C., Schurra, C., Garre’, M., Giovanni Nuciforo, P., Bensimon, A., et al. (2006) · Nature · doi:10.1038/nature05327 · Cited by 1,821
Chemokine signaling via the CXCR2 receptor reinforces senescence
Acosta, J. C., O'Loghlen, A., Banito, A., Guijarro, M. V., Augert, A., Raguz, S., Fumagalli, M., Da Costa, M., Brown, C., Popov, N., et al. (2008) · Cell · doi:10.1016/j.cell.2008.03.038 · Cited by 1,801
The RB and p53 pathways in cancer
Sherr, C. J., & McCormick, F. (2002) · Cancer Cell · doi:10.1016/s1535-6108(02)00102-2 · Cited by 1,690
Jacobs, J. J. L., Kieboom, K., Marino, S., DePinho, R. A., & van Lohuizen, M. (1999) · Nature · doi:10.1038/16476 · Cited by 1,580
Tumor Suppression at the Mouse INK4a Locus Mediated by the Alternative Reading Frame Product p19
Kamijo, T., Zindy, F., Roussel, M. F., Quelle, D. E., Downing, J. R., Ashmun, R. A., Grosveld, G., & Sherr, C. J. (1997) · Cell · doi:10.1016/s0092-8674(00)80452-3 · Cited by 1,566
Research works printed but not linked: 276
- 10.1038/299633a0 · DOI
- 10.1016/0014-4827(65)90211-9 · DOI · Paper page
- 10.1016/S0092-8674(00)81683-9 · DOI · Paper page
- Untergasser, 2002, Characterization of epithelial senescence by serial analysis of gene expression: identification of genes potentially involved in prostate cancer, Cancer Res, 62, 6255
- 10.1593/neo.05250 · DOI
- 10.1371/journal.pone.0003230 · DOI
- Correia-Melo, 2013, Robust multiparametric assessment of cellular senescence, Methods Mol. Biol, 965, 409, 10.1007/978-1-62703-239-1_27 · DOI
- 10.1016/j.lfs.2008.07.015 · DOI
- 10.1111/j.1469-185X.2010.00154.x · DOI
- 10.1016/0022-5193(73)90198-7 · DOI
- 10.1016/S0962-8924(00)89044-3 · DOI
- Kipling, 1999, Telomere-dependent senescence, Nat. Biotechnol, 17, 313, 10.1038/7827 · DOI
- Carnero, 2003, Cellular senescence and cancer, Res. Adv. Cancer, 3, 183
- Chandeck, 2010, Oncogene-induced cellular senescence, Adv. Anat. Pathol, 17, 42, 10.1097/PAP.0b013e3181c66f4e · DOI
- 10.4161/cc.10.16.16422 · DOI
- 10.1038/sj.onc.1210881 · DOI · Paper page
- 10.1016/j.mad.2008.03.009 · DOI
- 10.1093/nar/gkm1064 · DOI
- Wei, 1999, Expression of catalytically active telomerase does not prevent premature senescence caused by overexpression of oncogenic Ha-Ras in normal human fibroblasts, Cancer Res, 59, 1539
- 10.1016/S1535-6108(02)00159-9 · DOI
- 10.1101/gad.8.21.2540 · DOI · Paper page
- 10.1016/S0955-0674(00)00278-7 · DOI · Paper page
- 10.1172/JCI200420761 · DOI
- 10.1016/j.cell.2013.10.041 · DOI · Paper page
- 10.1073/pnas.95.25.14723 · DOI · Paper page
- 10.1038/22780 · DOI · Paper page
- 10.1101/gad.12.12.1769 · DOI
- 10.1158/0008-5472.CAN-03-4030 · DOI · Paper page
- 10.1007/s12094-010-0528-2 · DOI
- Malumbres, 2003, Cell cycle deregulation: a common motif in cancer, Prog. Cell Cycle Res, 5, 5
- 10.1038/sj.onc.1210704 · DOI
- 10.1371/journal.pone.0009305 · DOI
- 10.1038/sj.onc.1202261 · DOI
- 10.1006/excr.1993.1095 · DOI
- 10.1101/SQB.1994.059.01.046 · DOI
- 10.4061/2011/205378 · DOI
- Carnero, 2010, Epigenetic mechanisms in senescence, immortalisation and cancer, Biol. Rev. Camb. Philos. Soc
- 10.1128/MCB.22.10.3497-3508.2002 · DOI · Paper page
- 10.4161/cc.7.21.6919 · DOI
- 10.4161/cc.9.4.10766 · DOI
- 10.1093/carcin/bgm246 · DOI
- 10.1101/gad.12.19.3008 · DOI · Paper page
- 10.1073/pnas.091100298 · DOI
- 10.1038/35018127 · DOI
- 10.1002/(SICI)1096-9896(199610)180:2<118::AID-PATH673>3.0.CO;2-I · DOI
- Bond, 1996, Evidence that transcriptional activation by p53 plays a direct role in the induction of cellular senescence, Oncogene, 13, 2097
- 10.1038/sj.onc.1201880 · DOI
- 10.1038/35004020 · DOI
- 10.1126/science.277.5327.831 · DOI · Paper page
- 10.1038/sj.onc.1207839 · DOI
- 10.1128/MCB.25.17.7423-7431.2005 · DOI
- Serrano, 2003, Proliferation: the cell cycle, Adv. Exp. Med. Biol, 532, 13, 10.1007/978-1-4615-0081-0_2 · DOI
- 10.1073/pnas.190343497 · DOI
- Jarrard, 1999, p16/pRb pathway alterations are required for bypassing senescence in human prostate epithelial cells, Cancer Res, 59, 2957
- 10.1128/MCB.01592-06 · DOI
- 10.1016/S0168-9525(98)01470-X · DOI
- Carnero, 1998, The INK4 family of CDK inhibitors, Curr. Top. Microbiol. Immunol, 227, 43
- 10.1038/sj.onc.1201212 · DOI
- 10.1101/SQB.1994.059.01.008 · DOI
- 10.1038/onc.2012.45 · DOI · Paper page
- 10.1038/nrc1991 · DOI
- 10.1038/sj.onc.1208735 · DOI
- 10.1073/pnas.141235398 · DOI
- 10.1002/mc.20422 · DOI
- 10.1093/carcin/bgm246 · DOI
- 10.2174/138161210789941865 · DOI
- 10.1093/carcin/bgm052 · DOI
- Lacal, 1994, Regulation of ras proteins and their involvement in signal-transduction pathways (review), Oncol. Rep, 1, 677
- 10.1038/sj.onc.1205181 · DOI
- 10.1016/j.molcel.2010.09.026 · DOI
- 10.1074/jbc.M000759200 · DOI
- 10.1016/S0531-5565(02)00133-X · DOI
- 10.1038/84879 · DOI
- 10.1038/nature04809 · DOI
- 10.2174/156652409788488766 · DOI · Paper page
- 10.1158/0008-5472.CAN-08-1539 · DOI · Paper page
- 10.2174/156800910791054158 · DOI
- 10.1111/j.1748-1716.2007.01780.x · DOI
- 10.4049/jimmunol.176.5.2711 · DOI
- 10.1093/gerona/60.1.4 · DOI
- 10.1042/bst0320360 · DOI
- 10.1016/j.cell.2006.01.016 · DOI · Paper page
- 10.4161/cc.7.21.6965 · DOI · Paper page
- 10.2353/ajpath.2010.091050 · DOI
- 10.1016/j.gene.2007.10.022 · DOI
- 10.1093/carcin/15.1.11 · DOI
- 10.1016/0304-3835(96)04218-8 · DOI
- 10.1038/283596a0 · DOI · Paper page
- 10.1073/pnas.82.8.2394 · DOI
- 10.1093/carcin/14.5.833 · DOI
- 10.1186/bcr1275 · DOI · Paper page
- 10.1177/10454411010120010301 · DOI
- 10.1016/S0168-1702(02)00190-9 · DOI
- 10.1093/carcin/16.2.193 · DOI
- 10.1016/0304-3835(89)90060-8 · DOI
- 10.1080/01635589609514482 · DOI
- 10.1016/S0887-2333(00)00006-0 · DOI
- 10.1016/j.taap.2009.01.009 · DOI
- 10.1039/b903049b · DOI
- 10.3109/10408440903401511 · DOI
- 10.1016/S1734-1140(11)70707-1 · DOI · Paper page
- 10.3727/096368908786991542 · DOI · Paper page
- 10.1016/j.jhep.2005.05.036 · DOI · Paper page
- 10.1016/j.bbrc.2005.07.014 · DOI · Paper page
- Lee, 2012, Treatment with bisphenol A and methoxychlor results in the growth of human breast cancer cells and alteration of the expression of cell cycle-related genes, cyclin D1 and p21, via an estrogen receptor-dependent signaling pathway, Int. J. Mol. Med, 29, 883
- 10.1016/j.toxlet.2011.01.015 · DOI
- 10.1016/j.mrgentox.2007.08.006 · DOI
- 10.1016/S0890-6238(02)00005-9 · DOI
- 10.1080/009841097160573 · DOI
- Takahashi, 2004, Bisphenol A from dental polycarbonate crown upregulates the expression of hTERT, J. Biomed. Mater. Res. B. Appl. Biomater, 71, 214, 10.1002/jbm.b.30085 · DOI
- 10.1038/25870 · DOI
- 10.4049/jimmunol.181.10.7400 · DOI
- 10.1089/rej.2010.1085 · DOI
- Jacob, 2009, The effect of cotinine on telomerase activity in human vascular smooth muscle cells, J. Cardiovasc. Surg. (Torino), 50, 345
- 10.1093/carcin/bgm148 · DOI
- 10.1158/0008-5472.CAN-05-2494 · DOI
- 10.1097/FJC.0b013e31802ef519 · DOI
- 10.1038/sj.onc.1210886 · DOI
- 10.1038/bjp.2008.272 · DOI
- 10.1038/nature05354 · DOI · Paper page
- 10.1016/j.cell.2011.02.013 · DOI · Paper page
- 10.1097/COC.0b013e3181b0c4c2 · DOI
- Sun, 2003, Effects of allicin on both telomerase activity and apoptosis in gastric cancer SGC-7901 cells, World J. Gastroenterol, 9, 1930, 10.3748/wjg.v9.i9.1930 · DOI
- 10.1016/S0304-3835(02)00192-1 · DOI
- 10.1097/01.ju.0000121329.37206.1b · DOI
- 10.1371/journal.pone.0011457 · DOI
- 10.1358/dnp.2000.13.7.671717 · DOI
- 10.1016/j.canlet.2004.11.045 · DOI
- 10.2174/138955712802762220 · DOI
- Ambs, 1999, Cancer-prone oxyradical overload disease, IARC Sci. Publ, 295
- 10.3109/10715762.2011.571685 · DOI
- 10.1007/s00726-010-0600-9 · DOI · Paper page
- Alfonso, 2009, Does aspirin acetylate multiple cellular proteins? (Review), Mol. Med. Rep, 2, 533
- 10.1179/135100001101536085 · DOI
- Maehle, 1992, Altered p53 gene structure and expression in human epithelial cells after exposure to nickel, Cancer Res, 52, 218
- 10.1016/j.freeradbiomed.2011.04.008 · DOI
- 10.1016/j.mrrev.2007.11.008 · DOI
- 10.1023/B:MCBI.0000007262.26044.e8 · DOI · Paper page
- 10.1006/taap.2001.9157 · DOI
- 10.1016/S0898-6568(01)00175-9 · DOI · Paper page
- 10.2174/1568009053202072 · DOI
- 10.1016/j.niox.2010.06.001 · DOI
- Davis, 2000, Dietary selenium and arsenic affect DNA methylation in vitro in Caco-2 cells and in vivo in rat liver and colon, J. Nutr, 130, 2903, 10.1093/jn/130.12.2903 · DOI
- 10.1016/j.semcancer.2006.10.009 · DOI · Paper page
- 10.1038/nature01960 · DOI · Paper page
- 10.1177/0960327112440113 · DOI
- 10.1517/13543770902762893 · DOI · Paper page
- 10.1016/j.abb.2009.01.018 · DOI · Paper page
- Duntas, 2011, Resveratrol and its impact on aging and thyroid function, J. Endocrinol. Invest, 34, 788
- 10.4161/cc.7.8.5740 · DOI
- Maclaine, 2009, The regulation of p53 by phosphorylation: a model for how distinct signals integrate into the p53 pathway, Aging (Albany. NY), 1, 490, 10.18632/aging.100047 · DOI
- 10.1016/S0960-9822(99)80486-2 · DOI · Paper page
- 10.1258/ebm.2011.011107 · DOI
- 10.1007/s40265-013-0034-2 · DOI
- 10.1517/13543784.2012.726218 · DOI
- 10.1586/era.12.43 · DOI
- 10.1002/j.1875-9114.2012.01084.x · DOI
- 10.7150/ijbs.4499 · DOI
- 10.1016/j.niox.2010.06.005 · DOI
- Wang, 2012, Akt signaling-associated metabolic effects of dietary gold nanoparticles in Drosophila, Sci. Rep, 2, 563, 10.1038/srep00563 · DOI
- Jang, 2012, Silver nanoparticles modify VEGF signaling pathway and mucus hypersecretion in allergic airway inflammation, Int. J. Nanomed, 7, 1329
- 10.1016/j.colsurfb.2009.04.025 · DOI
- 10.1016/j.toxlet.2011.09.002 · DOI
- 10.1016/j.toxlet.2010.12.010 · DOI
- 10.2174/138945011798184155 · DOI
- 10.1002/ijc.24398 · DOI
- 10.1093/geronj/11.3.298 · DOI · Paper page
- Vergel, 2010, Cellular senescence as a target in cancer control, J. Aging Res, 2011, 725365
- 10.1186/1476-4598-8-3 · DOI
- 10.1016/S0047-6374(02)00091-X · DOI
- 10.1016/S0092-8674(00)81902-9 · DOI · Paper page
- 10.1128/MCB.23.1.389-401.2003 · DOI · Paper page
- 10.1038/534 · DOI
- 10.1126/science.8108730 · DOI
- 10.1074/jbc.274.12.7936 · DOI · Paper page
- 10.1093/pcp/pch110 · DOI
- 10.1126/science.1064653 · DOI
- 10.1046/j.1474-9728.2003.00065.x · DOI
- 10.1016/0891-5849(95)02111-6 · DOI · Paper page
- 10.1006/abbi.1996.0189 · DOI
- 10.1038/sj.onc.1202707 · DOI
- Kondoh, 2007, Protection from oxidative stress by enhanced glycolysis; a possible mechanism of cellular immortalization, Histol. Histopathol, 22, 85
- 10.1016/j.ccr.2012.09.020 · DOI
- 10.1007/BF01877355 · DOI
- Virchow, 1863, Aetiologie der neoplastischen Geshwültse/Pathogenie der neoplastishchen Geshwülste, Die Krankhaften Geschwülste, 57
- 10.1093/carcin/bgp127 · DOI
- 10.1016/j.ccr.2005.02.013 · DOI · Paper page
- 10.1023/B:APPT.0000018794.26438.22 · DOI
- 10.1016/S0092-8674(00)81988-1 · DOI
- 10.1016/j.semcancer.2004.06.006 · DOI · Paper page
- 10.1016/j.ccr.2007.01.012 · DOI · Paper page
- 10.1053/j.gastro.2011.05.041 · DOI
- Narlik-Grassow, (2014) Conditional transgenic expression of PIM1 kinase in prostate induces inflammation-dependent neoplasia, PLoS One, 8
- 10.1038/nature05529 · DOI · Paper page
- 10.1146/annurev-pathol-121808-102144 · DOI · Paper page
- 10.1073/pnas.211053698 · DOI · Paper page
- 10.1016/j.mad.2012.09.004 · DOI · Paper page
- 10.1186/1756-8722-5-13 · DOI · Paper page
- Chen, 2000, Replicative senescence and oxidant-induced premature senescence. Beyond the control of cell cycle checkpoints, Ann. N. Y. Acad. Sci, 908, 111, 10.1111/j.1749-6632.2000.tb06640.x · DOI
- Chang, 1999, A senescence-like phenotype distinguishes tumor cells that undergo terminal proliferation arrest after exposure to anticancer agents, Cancer Res, 59, 3761
200 of 276 shown
Cited by
Chalak, M., Hesaraki, M., Mirbahari, S. N., Yeganeh, M., Abdi, S., Rajabi, S., & Hemmatzadeh, F. (2024) · Life · doi:10.3390/life14030417 · Cited by 66
Metadata
- Authors
- Carnero, A., Blanco-Aparicio, C., Kondoh, H., Lleonart, M. E., Martinez-Leal, J. F., Mondello, C., Scovassi, A. I., Bisson, W. H., Amedei, A., Roy, R., Woodrick, J., Colacci, A., Vaccari, M., Raju, J., Al-Mulla, F., Al-Temaimi, R., Salem, H. K., Memeo, L., Forte, S., Singh, N., Hamid, R. A., Ryan, E. P., Brown, D. G., Wise, J. P., Wise, S. S., & Yasaei, H.
- Venue
- Carcinogenesis
- Publisher
- Oxford University Press (OUP)
- Published
- 2015-06
- Volume
- 36
- Issue
- Suppl 1
- Pages
- S19-S37
- ISSN
- 0143-3334
- 1460-2180
- Cited by
- 29
- References
- 64
- Language
- en
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review