The DOI matches its Crossref record; 84 of the 84 references it cites were found in a registry or an index.
Post-infarct remodelling: contribution of wound healing and inflammation
Open accessAbstract
In human and experimental myocardial infarction (MI), cessation of blood supply leads to rapid necrosis of cardiac myocytes in the ischaemic heart. Immediately after injury, various intra- and intercellular pathways contribute to healing the myocardial wound in order to achieve tissue integrity and function. MI and the consequent loss of myocardium are the major aetiology for heart failure. Despite aggressive primary therapy, prognosis remains poor in patients with large infarction and severe left ventricular dysfunction. Thus, it would be highly desirable to improve healing of the cardiac wound to maintain structure and function of the heart. Healing in the heart occurs in overlapping phases. Herein, we review the inflammatory phase as a trigger of tissue formation.
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Cites
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Cited by
Mesenchymal stem cells: environmentally responsive therapeutics for regenerative medicine
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Guidelines for experimental models of myocardial ischemia and infarction
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Role of Lymphocytes in Myocardial Injury, Healing, and Remodeling After Myocardial Infarction
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Metadata
- Authors
- Frantz, S., Bauersachs, J., & Ertl, G.
- Venue
- Cardiovascular Research
- Publisher
- Oxford University Press (OUP)
- Published
- 2008-10-29
- Volume
- 81
- Issue
- 3
- Pages
- 474-481
- ISSN
- 0008-6363 (ISSN-L)
- OpenAlex
- W2134012941
- Cited by
- 287
- References
- 84
- FWCI
- 13.20
- Type
- journal article
- Language
- en
- Open access
- bronze
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
