The DOI matches its Crossref record; 104 of the 104 references it cites were found in a registry or an index.
Epstein–Barr virus-associated lymphomas
Open accessAbstract
Epstein–Barr virus (EBV), originally discovered through its association with Burkitt lymphoma, is now aetiologically linked to a remarkably wide range of lymphoproliferative lesions and malignant lymphomas of B-, T- and NK-cell origin. Some occur as rare accidents of virus persistence in the B lymphoid system, while others arise as a result of viral entry into unnatural target cells. The early finding that EBV is a potent B-cell growth transforming agent hinted at a simple oncogenic mechanism by which this virus could promote lymphomagenesis. In reality, the pathogenesis of EBV-associated lymphomas involves a complex interplay between different patterns of viral gene expression and cellular genetic changes. Here we review recent developments in our understanding of EBV-associated lymphomagenesis in both the immunocompetent and immunocompromised host. This article is part of the themed issue ‘Human oncogenic viruses’.
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Cites
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Cited by
The Global Landscape of EBV-Associated Tumors
Shannon-Lowe, C., & Rickinson, A. (2019) · Frontiers in Oncology · doi:10.3389/fonc.2019.00713 · Cited by 459
Self-citationAllen, U. D., Preiksaitis, J. K., & Practice, T. A. I. D. C. O. (2019) · Clinical Transplantation · doi:10.1111/ctr.13652 · Cited by 375
Epstein–Barr virus reprograms human B lymphocytes immediately in the prelatent phase of infection
Mrozek-Gorska, P., Buschle, A., Pich, D., Schwarzmayr, T., Fechtner, R., Scialdone, A., & Hammerschmidt, W. (2019) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.1901314116 · Cited by 210
Epstein-Barr-Virus-Induced One-Carbon Metabolism Drives B Cell Transformation
Wang, L. W., Shen, H., Nobre, L., Ersing, I., Paulo, J. A., Trudeau, S., Wang, Z., Smith, N. A., Ma, Y., Reinstadler, B., et al. (2019) · Cell Metabolism · doi:10.1016/j.cmet.2019.06.003 · Cited by 190
Metadata
- Authors
- Shannon-Lowe, C., Rickinson, A. B., & Bell, A. I.
- Venue
- Philosophical Transactions of the Royal Society B: Biological Sciences
- Publisher
- The Royal Society
- Published
- 2017-09-11
- Volume
- 372
- Issue
- 1732
- Pages
- 20160271
- ISSN
- 0962-8436 (ISSN-L)
- 1471-2970
- OpenAlex
- W2755325205
- Cited by
- 428
- References
- 104
- FWCI
- 13.89
- Type
- journal article
- Language
- en
- Open access
- hybrid
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