The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
Role of keratinocyte‐derived factors involved in regulating the proliferation and differentiation of mammalian epidermal melanocytes
Open accessAbstract
Summary: Melanocytes characterized by the activities of tyrosinase, tyrosinase‐related protein (TRP)‐1 and TRP‐2 as well as by melanosomes and dendrites are located mainly in the epidermis, dermis and hair bulb of the mammalian skin. Melanocytes differentiate from melanoblasts, undifferentiated precursors, derived from embryonic neural crest cells. Because hair bulb melanocytes are derived from epidermal melanoblasts and melanocytes, the mechanism of the regulation of the proliferation and differentiation of epidermal melanocytes should be clarified. The regulation by the tissue environment, especially by keratinocytes is indispensable in addition to the regulation by genetic factors in melanocytes. Recent advances in the techniques of tissue culture and biochemistry have enabled us to clarify factors derived from keratinocytes. Alpha‐melanocyte‐stimulating hormone, adrenocorticotrophic hormone, basic fibroblast growth factor, nerve growth factor, endothelins, granulocyte‐macrophage colony‐stimulating factor, steel factor, leukemia inhibitory factor and hepatocyte growth factor have been suggested to be the keratinocyte‐derived factors and to regulate the proliferation and/or differentiation of mammalian epidermal melanocytes. Numerous factors may be produced in and released from keratinocytes and be involved in regulating the proliferation and differentiation of mammalian epidermal melanocytes through receptor‐mediated signaling pathways.
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The dominant-white spotting (W) locus of the mouse encodes the c-kit proto-oncogene
Geissler, E. N., Ryan, M. A., & Housman, D. E. (1988) · Cell · doi:10.1016/0092-8674(88)90020-7 · Cited by 1,287
Endothelins secreted from human keratinocytes are intrinsic mitogens for human melanocytes
Imokawa, G., Yada, Y., & Miyagishi, M. (1992) · Journal of Biological Chemistry · doi:10.1016/s0021-9258(18)35817-4 · Cited by 385
The protease-activated receptor 2 regulates pigmentation via keratinocyte-melanocyte interactions
Seiberg, M., Paine, C., Sharlow, E., Andrade-Gordon, P., Costanzo, M., Eisinger, M., & Shapiro, S. S. (2000) · Experimental Cell Research · doi:10.1006/excr.1999.4692 · Cited by 238 · cited 4×
“, 1990, 1993) or three-dimensional (skin equivalent model, Archambault et al., 1995; De Luca et al., 1988; Scott and Haake, 1991; Seiberg et al., 2000; Todd et al., 1993; Valyi-Nagy et al., 1990) co-culture systems.”
BackgroundInfluentialQuevedo, W. C., Szabo, G., & Virks, J. (1969) · Journal of Investigative Dermatology · doi:10.1038/jid.1969.45 · Cited by 102
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Hypopigmenting agents: an updated review on biological, chemical and clinical aspects
Solano, F., Briganti, S., Picardo, M., & Ghanem, G. (2006) · Pigment Cell Research · doi:10.1111/j.1600-0749.2006.00334.x · Cited by 696
Neuronal Control of Skin Function: The Skin as a Neuroimmunoendocrine Organ
Roosterman, D., Goerge, T., Schneider, S. W., Bunnett, N. W., & Steinhoff, M. (2006) · Physiological Reviews · doi:10.1152/physrev.00026.2005 · Cited by 665
Mitf regulation of Dia1 controls melanoma proliferation and invasiveness
Carreira, S., Goodall, J., Denat, L., Rodriguez, M., Nuciforo, P., Hoek, K. S., Testori, A., Larue, L., & Goding, C. R. (2006) · Genes & Development · doi:10.1101/gad.406406 · Cited by 608
Ribosomal mutations cause p53-mediated dark skin and pleiotropic effects
McGowan, K. A., Li, J. Z., Park, C. Y., Beaudry, V., Tabor, H. K., Sabnis, A. J., Zhang, W., Fuchs, H., de Angelis, M. H., Myers, R. M., et al. (2008) · Nature Genetics · doi:10.1038/ng.188 · Cited by 364
Metadata
- Authors
- Hirobe, T.
- Venue
- Pigment Cell Research
- Publisher
- Wiley
- Published
- 2005-01-12
- Volume
- 18
- Issue
- 1
- Pages
- 2-12
- ISSN
- 0893-5785 (ISSN-L)
- 1600-0749
- OpenAlex
- W2013102804
- Cited by
- 285
- References
- 5
- FWCI
- 5.57
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.